The effect of conservative oxygen therapy on systemic biomarkers of oxidative stress in critically ill patients.


Journal

Free radical biology & medicine
ISSN: 1873-4596
Titre abrégé: Free Radic Biol Med
Pays: United States
ID NLM: 8709159

Informations de publication

Date de publication:
20 11 2020
Historique:
received: 27 04 2020
revised: 02 06 2020
accepted: 05 06 2020
pubmed: 10 8 2020
medline: 28 5 2021
entrez: 10 8 2020
Statut: ppublish

Résumé

Supplemental oxygen is delivered to critically ill patients who require mechanical ventilation. Oxidative stress is a potential complication of oxygen therapy, resulting in damage to essential biomolecules such as proteins, lipids, and nucleic acids. Whether plasma levels of oxidative stress biomarkers vary based on how liberally oxygen therapy is applied during mechanical ventilation is unknown. We carried out an oxidative stress substudy nested within a large multi-centre randomized controlled trial in which critically ill adults were randomized to receive either conservative oxygen therapy or standard oxygen therapy. Blood samples were collected at enrolment, and daily thereafter for up to three days. The antioxidant ascorbate (vitamin C) was assessed using HPLC with electrochemical detection and protein oxidation using a sensitive protein carbonyl ELISA. We also assessed whether critically ill patients with different disease states exhibited varying levels of oxidative stress biomarkers. A total of 125 patients were included. Mean ascorbate concentrations decreased over time (from 25 ± 9 μmol/L to 14 ± 2 μmol/L, p < 0.001), however, there was no significant difference between the conservative oxygen group and standard care (p = 0.2), despite a significantly lower partial pressure of oxygen (PaO Conservative oxygen therapy does not alter systemic markers of oxidative stress in critically ill ventilated patients compared with standard oxygen therapy. Patients with sepsis exhibited elevated protein carbonyls compared with the other critically ill patients implying increased oxidative stress in this patient subgroup.

Sections du résumé

BACKGROUND
Supplemental oxygen is delivered to critically ill patients who require mechanical ventilation. Oxidative stress is a potential complication of oxygen therapy, resulting in damage to essential biomolecules such as proteins, lipids, and nucleic acids. Whether plasma levels of oxidative stress biomarkers vary based on how liberally oxygen therapy is applied during mechanical ventilation is unknown.
METHODS
We carried out an oxidative stress substudy nested within a large multi-centre randomized controlled trial in which critically ill adults were randomized to receive either conservative oxygen therapy or standard oxygen therapy. Blood samples were collected at enrolment, and daily thereafter for up to three days. The antioxidant ascorbate (vitamin C) was assessed using HPLC with electrochemical detection and protein oxidation using a sensitive protein carbonyl ELISA. We also assessed whether critically ill patients with different disease states exhibited varying levels of oxidative stress biomarkers.
RESULTS
A total of 125 patients were included. Mean ascorbate concentrations decreased over time (from 25 ± 9 μmol/L to 14 ± 2 μmol/L, p < 0.001), however, there was no significant difference between the conservative oxygen group and standard care (p = 0.2), despite a significantly lower partial pressure of oxygen (PaO
CONCLUSIONS
Conservative oxygen therapy does not alter systemic markers of oxidative stress in critically ill ventilated patients compared with standard oxygen therapy. Patients with sepsis exhibited elevated protein carbonyls compared with the other critically ill patients implying increased oxidative stress in this patient subgroup.

Identifiants

pubmed: 32768569
pii: S0891-5849(20)31115-1
doi: 10.1016/j.freeradbiomed.2020.06.018
pii:
doi:

Substances chimiques

Biomarkers 0
Oxygen S88TT14065

Types de publication

Journal Article Multicenter Study Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

13-18

Commentaires et corrections

Type : ErratumIn

Informations de copyright

Copyright © 2020 Elsevier Inc. All rights reserved.

Auteurs

Anitra C Carr (AC)

Department of Pathology and Biomedical Science, University of Otago, Christchurch, New Zealand. Electronic address: anitra.carr@otago.ac.nz.

Emma Spencer (E)

Department of Pathology and Biomedical Science, University of Otago, Christchurch, New Zealand.

Diane Mackle (D)

Medical Research Institute of New Zealand, Wellington, New Zealand.

Anna Hunt (A)

Medical Research Institute of New Zealand, Wellington, New Zealand; Wellington Hospital Intensive Care Unit, Wellington, New Zealand.

Harriet Judd (H)

Wellington Hospital Intensive Care Unit, Wellington, New Zealand.

Jan Mehrtens (J)

Christchurch Hospital Intensive Care Unit, Christchurch, New Zealand.

Kim Parker (K)

Christchurch Hospital Intensive Care Unit, Christchurch, New Zealand.

Zoe Stockwell (Z)

Centre for Postgraduate Nursing Studies, University of Otago, Christchurch, New Zealand.

Caitlin Gale (C)

Centre for Postgraduate Nursing Studies, University of Otago, Christchurch, New Zealand.

Megan Beaumont (M)

Centre for Postgraduate Nursing Studies, University of Otago, Christchurch, New Zealand.

Simran Kaur (S)

Centre for Postgraduate Nursing Studies, University of Otago, Christchurch, New Zealand.

Shailesh Bihari (S)

Intensive and Critical Care Unit, Flinders Medical Centre, Adelaide, Australia; College of Medicine and Public Health, Flinders University, Adelaide, Australia.

Paul J Young (PJ)

Medical Research Institute of New Zealand, Wellington, New Zealand; Wellington Hospital Intensive Care Unit, Wellington, New Zealand.

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Classifications MeSH