Normalization of Fecal Calprotectin Within 12 Months of Diagnosis Is Associated With Reduced Risk of Disease Progression in Patients With Crohn's Disease.


Journal

Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association
ISSN: 1542-7714
Titre abrégé: Clin Gastroenterol Hepatol
Pays: United States
ID NLM: 101160775

Informations de publication

Date de publication:
09 2021
Historique:
received: 11 11 2019
revised: 08 07 2020
accepted: 07 08 2020
pubmed: 18 8 2020
medline: 10 9 2021
entrez: 18 8 2020
Statut: ppublish

Résumé

The level of fecal calprotectin (FC) correlates with endoscopic evidence of inflammation in Crohn's disease (CD). A treat-to-target algorithm for patients with CD, that incorporates FC, outperforms a treatment strategy based on symptoms alone in the induction of mucosal healing at 12 months. We investigated whether normalization of FC within 12 months of diagnosis of CD is associated with a reduction in disease progression. We performed a retrospective cohort study at a tertiary IBD centre in the United Kingdom. We identified all incident cases of CD diagnosed from 2005 through 2017. Patients with a FC measurement ≥250 μg/g at diagnosis who also had at least 1 follow-up FC measurement within the first 12 months of diagnosis and >12 months of follow up were included. The last FC measurement within 12 months of diagnosis was used to determine normalization (cut-off <250 μg/g). The primary endpoint was time to first disease progression (composite of progression in Montreal disease behavior B1 to B2/3, B2 to B3, or new perianal disease; CD-related surgery; or CD-related hospitalization). Cox proportional hazards regression analysis was used to determine independent factors associated with time to first disease progression. A total of 375 patients out of 1389 incident cases were included, with a median follow up of 5.3 years (interquartile range, 3.1-7.4 years). Normalization of FC within 12 months of diagnosis was confirmed in 43.5% of patients. Patients with normalized levels of FC had a significantly lower risk of composite disease progression (hazard ratio [HR], 0.36; 95% CI, 0.24-0.53; P < .001). They also had a lower risk of reaching any of the separate progression endpoints (progression in Montreal behavior or new perianal disease HR, 0.22; 95% CI, 0.11-0.45; P < .001; hospitalization HR, 0.33; 95% CI, 0.21-0.53; P <.001; surgery HR, 0.39; 95% CI, 0.19-0.78; P = .008) CONCLUSIONS: Normalization of FC within 12 months of diagnosis is associated with a reduced risk of progression of CD.

Sections du résumé

BACKGROUND & AIMS
The level of fecal calprotectin (FC) correlates with endoscopic evidence of inflammation in Crohn's disease (CD). A treat-to-target algorithm for patients with CD, that incorporates FC, outperforms a treatment strategy based on symptoms alone in the induction of mucosal healing at 12 months. We investigated whether normalization of FC within 12 months of diagnosis of CD is associated with a reduction in disease progression.
METHODS
We performed a retrospective cohort study at a tertiary IBD centre in the United Kingdom. We identified all incident cases of CD diagnosed from 2005 through 2017. Patients with a FC measurement ≥250 μg/g at diagnosis who also had at least 1 follow-up FC measurement within the first 12 months of diagnosis and >12 months of follow up were included. The last FC measurement within 12 months of diagnosis was used to determine normalization (cut-off <250 μg/g). The primary endpoint was time to first disease progression (composite of progression in Montreal disease behavior B1 to B2/3, B2 to B3, or new perianal disease; CD-related surgery; or CD-related hospitalization). Cox proportional hazards regression analysis was used to determine independent factors associated with time to first disease progression.
RESULTS
A total of 375 patients out of 1389 incident cases were included, with a median follow up of 5.3 years (interquartile range, 3.1-7.4 years). Normalization of FC within 12 months of diagnosis was confirmed in 43.5% of patients. Patients with normalized levels of FC had a significantly lower risk of composite disease progression (hazard ratio [HR], 0.36; 95% CI, 0.24-0.53; P < .001). They also had a lower risk of reaching any of the separate progression endpoints (progression in Montreal behavior or new perianal disease HR, 0.22; 95% CI, 0.11-0.45; P < .001; hospitalization HR, 0.33; 95% CI, 0.21-0.53; P <.001; surgery HR, 0.39; 95% CI, 0.19-0.78; P = .008) CONCLUSIONS: Normalization of FC within 12 months of diagnosis is associated with a reduced risk of progression of CD.

Identifiants

pubmed: 32798706
pii: S1542-3565(20)31131-9
doi: 10.1016/j.cgh.2020.08.022
pii:
doi:

Substances chimiques

Biomarkers 0
Leukocyte L1 Antigen Complex 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1835-1844.e6

Subventions

Organisme : Medical Research Council
ID : MR/S034919/1
Pays : United Kingdom

Informations de copyright

Copyright © 2021 AGA Institute. Published by Elsevier Inc. All rights reserved.

Auteurs

Nikolas Plevris (N)

Edinburgh IBD Unit, Western General Hospital, Edinburgh, United Kingdom. Electronic address: n.plevris12@gmail.com.

James Fulforth (J)

Edinburgh IBD Unit, Western General Hospital, Edinburgh, United Kingdom.

Mathew Lyons (M)

Edinburgh IBD Unit, Western General Hospital, Edinburgh, United Kingdom.

Spyros I Siakavellas (SI)

Edinburgh IBD Unit, Western General Hospital, Edinburgh, United Kingdom.

Philip W Jenkinson (PW)

Edinburgh IBD Unit, Western General Hospital, Edinburgh, United Kingdom.

Cher S Chuah (CS)

Edinburgh IBD Unit, Western General Hospital, Edinburgh, United Kingdom.

Laura Lucaciu (L)

Edinburgh IBD Unit, Western General Hospital, Edinburgh, United Kingdom.

Rebecca J Pattenden (RJ)

Department of Biochemistry, Western General Hospital, Edinburgh, United Kingdom.

Ian D Arnott (ID)

Edinburgh IBD Unit, Western General Hospital, Edinburgh, United Kingdom.

Gareth-Rhys Jones (GR)

Edinburgh IBD Unit, Western General Hospital, Edinburgh, United Kingdom.

Charlie W Lees (CW)

Edinburgh IBD Unit, Western General Hospital, Edinburgh, United Kingdom; Centre for Genomic and Experimental Medicine, MRC Institute of Genetics and Molecular Medicine, Western General Hospital Campus, University of Edinburgh, Edinburgh, United Kingdom.

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