Tumor burden of persistent disease in patients with differentiated thyroid cancer: correlation with postoperative risk-stratification and impact on outcome.


Journal

BMC cancer
ISSN: 1471-2407
Titre abrégé: BMC Cancer
Pays: England
ID NLM: 100967800

Informations de publication

Date de publication:
14 Aug 2020
Historique:
received: 13 02 2020
accepted: 06 08 2020
entrez: 18 8 2020
pubmed: 18 8 2020
medline: 15 4 2021
Statut: epublish

Résumé

In patients with differentiated thyroid cancer (DTC), tumor burden of persistent disease (PD) is a variable that could affect therapy efficiency. Our aim was to assess its correlation with the 2015 American Thyroid Association (ATA) risk-stratification system, and its impact on response to initial therapy and outcome. This retrospective cohort study included 618 consecutive DTC patients referred for postoperative radioiodine (RAI) treatment. Patients were risk-stratified using the 2015 ATA guidelines according to postoperative data, before RAI treatment. Tumor burden of PD was classified into three categories, i.e. very small-, small- and large-volume PD. Very small-volume PD was defined by the presence of abnormal foci on post-RAI scintigraphy with SPECT/CT or PD was evidenced in 107 patients (17%). Mean follow-up for patients with PD was 7 ± 3 years. The percentage of large-volume PD increased with the ATA risk (18, 56 and 89% in low-, intermediate- and high-risk patients, respectively, p < 0.0001). There was a significant trend for a decrease in excellent response rate from the very small-, small- to large-volume PD groups at 9-12 months after initial therapy (71, 20 and 7%, respectively; p = 0.01) and at last follow-up visit (75, 28 and 16%, respectively; p = 0.04). On multivariate analysis, age ≥ 45 years, distant and/or thyroid bed disease, small-volume or large-volume tumor burden and The tumor burden of PD correlates with the ATA risk-stratification, affects the response to initial therapy and is an independent predictor of residual disease after a mean 7-yr follow-up. This variable might be taken into account in addition to the postoperative ATA risk-stratification to refine outcome prognostication after initial treatment.

Sections du résumé

BACKGROUND BACKGROUND
In patients with differentiated thyroid cancer (DTC), tumor burden of persistent disease (PD) is a variable that could affect therapy efficiency. Our aim was to assess its correlation with the 2015 American Thyroid Association (ATA) risk-stratification system, and its impact on response to initial therapy and outcome.
METHODS METHODS
This retrospective cohort study included 618 consecutive DTC patients referred for postoperative radioiodine (RAI) treatment. Patients were risk-stratified using the 2015 ATA guidelines according to postoperative data, before RAI treatment. Tumor burden of PD was classified into three categories, i.e. very small-, small- and large-volume PD. Very small-volume PD was defined by the presence of abnormal foci on post-RAI scintigraphy with SPECT/CT or
RESULTS RESULTS
PD was evidenced in 107 patients (17%). Mean follow-up for patients with PD was 7 ± 3 years. The percentage of large-volume PD increased with the ATA risk (18, 56 and 89% in low-, intermediate- and high-risk patients, respectively, p < 0.0001). There was a significant trend for a decrease in excellent response rate from the very small-, small- to large-volume PD groups at 9-12 months after initial therapy (71, 20 and 7%, respectively; p = 0.01) and at last follow-up visit (75, 28 and 16%, respectively; p = 0.04). On multivariate analysis, age ≥ 45 years, distant and/or thyroid bed disease, small-volume or large-volume tumor burden and
CONCLUSIONS CONCLUSIONS
The tumor burden of PD correlates with the ATA risk-stratification, affects the response to initial therapy and is an independent predictor of residual disease after a mean 7-yr follow-up. This variable might be taken into account in addition to the postoperative ATA risk-stratification to refine outcome prognostication after initial treatment.

Identifiants

pubmed: 32799836
doi: 10.1186/s12885-020-07269-3
pii: 10.1186/s12885-020-07269-3
pmc: PMC7429727
doi:

Substances chimiques

Iodine Radioisotopes 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

765

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Auteurs

Renaud Ciappuccini (R)

Department of Nuclear Medicine and Thyroid Unit, François Baclesse Cancer Centre, 3 Avenue Général Harris, F-14000, Caen, France. r.ciappuccini@baclesse.unicancer.fr.
INSERM 1086 ANTICIPE, Caen University, Caen, France. r.ciappuccini@baclesse.unicancer.fr.

Natacha Heutte (N)

CETAPS EA 3832, Rouen University, Rouen, France.

Audrey Lasne-Cardon (A)

Department of Head and Neck Surgery, François Baclesse Cancer Centre, Caen, France.

Virginie Saguet-Rysanek (V)

Department of Pathology, François Baclesse Cancer Centre, Caen, France.

Camille Leroy (C)

Department of Oncology, François Baclesse Cancer Centre, Caen, France.

Véronique Le Hénaff (V)

Department of Nuclear Medicine and Thyroid Unit, François Baclesse Cancer Centre, 3 Avenue Général Harris, F-14000, Caen, France.

Dominique Vaur (D)

Department of Cancer Biology and Genetics, François Baclesse Cancer Centre, Caen, France.

Emmanuel Babin (E)

INSERM 1086 ANTICIPE, Caen University, Caen, France.
Department of Head and Neck Surgery, François Baclesse Cancer Centre, Caen, France.
Department of Head and Neck Surgery, University Hospital, Caen, France.

Stéphane Bardet (S)

Department of Nuclear Medicine and Thyroid Unit, François Baclesse Cancer Centre, 3 Avenue Général Harris, F-14000, Caen, France.

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