Gynecological and reproductive factors and the risk of pancreatic cancer: A case-control study.


Journal

Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]
ISSN: 1424-3911
Titre abrégé: Pancreatology
Pays: Switzerland
ID NLM: 100966936

Informations de publication

Date de publication:
Sep 2020
Historique:
received: 20 03 2020
revised: 28 06 2020
accepted: 12 07 2020
pubmed: 18 8 2020
medline: 27 8 2021
entrez: 18 8 2020
Statut: ppublish

Résumé

/Objectives: Pancreatic ductal adenocarcinoma (PDAC) has a higher incidence in men compared to women, although the difference in known risk factors cannot explain this disparity completely. Reproductive and hormonal factors have been demonstrated in pre-clinical studies to influence pancreatic carcinogenesis, but the few published data on the topic are inconsistent. The aim was to investigate the role of reproductive and hormonal factors on PDAC occurrence in women. We conducted a unicenter case-control study; PDAC cases were matched to controls by age with a 1:2 ratio. Risk factors were screened through questionnaires about gynecologic and medical history. Comparisons were made using Chi-square and Fisher's exact tests where appropriate for categorical variables and Student's t-test for continuous variables. Logistic regression was used to calculate Odds Ratios (ORs) and their 95% confidence intervals (CI). Multivariable logistic regression models were adjusted for potential confounders. 253 PDAC and 506 matched controls were enrolled. At logistic regression multivariable analysis adjusted for confounding factors, older age at menopause (OR:0.95 per year; 95% CI:0.91-0.98; p = 0.007), use of Oral Contraceptives (OR:0.52; 95% CI:0.30-0.89; p = 0.018), use of Hormonal Replacement Therapy (OR:0.31; 95% CI:0.15-0.64; p = 0.001), and having had two children (OR:0.57; 95% CI:0.38-0.84; p = 0.005) were significant, independent protective factors for the onset of PDAC. These data confirm some previous findings on menopause age and number of births while, to our knowledge, this is the first study to show a protective effect of HRT and OC use. The results collectively support the hypothesis that exposure to estrogens plays a protective role towards PDAC.

Sections du résumé

BACKGROUND BACKGROUND
/Objectives: Pancreatic ductal adenocarcinoma (PDAC) has a higher incidence in men compared to women, although the difference in known risk factors cannot explain this disparity completely. Reproductive and hormonal factors have been demonstrated in pre-clinical studies to influence pancreatic carcinogenesis, but the few published data on the topic are inconsistent. The aim was to investigate the role of reproductive and hormonal factors on PDAC occurrence in women.
METHODS METHODS
We conducted a unicenter case-control study; PDAC cases were matched to controls by age with a 1:2 ratio. Risk factors were screened through questionnaires about gynecologic and medical history. Comparisons were made using Chi-square and Fisher's exact tests where appropriate for categorical variables and Student's t-test for continuous variables. Logistic regression was used to calculate Odds Ratios (ORs) and their 95% confidence intervals (CI). Multivariable logistic regression models were adjusted for potential confounders.
RESULTS RESULTS
253 PDAC and 506 matched controls were enrolled. At logistic regression multivariable analysis adjusted for confounding factors, older age at menopause (OR:0.95 per year; 95% CI:0.91-0.98; p = 0.007), use of Oral Contraceptives (OR:0.52; 95% CI:0.30-0.89; p = 0.018), use of Hormonal Replacement Therapy (OR:0.31; 95% CI:0.15-0.64; p = 0.001), and having had two children (OR:0.57; 95% CI:0.38-0.84; p = 0.005) were significant, independent protective factors for the onset of PDAC.
CONCLUSIONS CONCLUSIONS
These data confirm some previous findings on menopause age and number of births while, to our knowledge, this is the first study to show a protective effect of HRT and OC use. The results collectively support the hypothesis that exposure to estrogens plays a protective role towards PDAC.

Identifiants

pubmed: 32800645
pii: S1424-3903(20)30614-1
doi: 10.1016/j.pan.2020.07.398
pii:
doi:

Substances chimiques

Contraceptive Agents, Female 0
Gonadal Steroid Hormones 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1149-1154

Informations de copyright

Copyright © 2020 IAP and EPC. Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare no conflict of interest.

Auteurs

Livia Archibugi (L)

Digestive and Liver Disease Unit, Sant'Andrea Hospital, Faculty of Medicine and Psychology, Sapienza University of Rome, Milan, Italy; Pancreatobiliary Endoscopy and Endosonography Division, Pancreas Translational and Clinical Research Center, San Raffaele Hospital, San Raffaele Scientific Institute IRCCS, Milan, Italy.

Benedetta Graglia (B)

Digestive and Liver Disease Unit, Sant'Andrea Hospital, Faculty of Medicine and Psychology, Sapienza University of Rome, Milan, Italy.

Roberto Valente (R)

Digestive and Liver Disease Unit, Sant'Andrea Hospital, Faculty of Medicine and Psychology, Sapienza University of Rome, Milan, Italy; Department of Surgery, Umeå University Hospital, Umeå, Sweden; Department of Surgery, Anschutz Medical Campus, University of Colorado, Denver, USA.

Serena Stigliano (S)

Digestive and Liver Disease Unit, Sant'Andrea Hospital, Faculty of Medicine and Psychology, Sapienza University of Rome, Milan, Italy; Department of Internal Medicine and Medical Specialties, Gastroenterology Unit, Sapienza University of Rome, Milan, Italy.

Michela Roberto (M)

Oncology Unit, S. Andrea Hospital, Faculty of Medicine and Psychology, Sapienza University of Rome, Milan, Italy.

Carlo Capalbo (C)

Oncology Unit, S. Andrea Hospital, Faculty of Medicine and Psychology, Sapienza University of Rome, Milan, Italy.

Paolo Marchetti (P)

Oncology Unit, S. Andrea Hospital, Faculty of Medicine and Psychology, Sapienza University of Rome, Milan, Italy.

Giuseppe Nigri (G)

General Surgery and Hepato-pancreato-biliary Unit, Sant'Andrea Hospital, Faculty of Medicine and Psychology, Sapienza University of Rome, Milan, Italy.

Gabriele Capurso (G)

Digestive and Liver Disease Unit, Sant'Andrea Hospital, Faculty of Medicine and Psychology, Sapienza University of Rome, Milan, Italy; Pancreatobiliary Endoscopy and Endosonography Division, Pancreas Translational and Clinical Research Center, San Raffaele Hospital, San Raffaele Scientific Institute IRCCS, Milan, Italy. Electronic address: capurso.gabriele@hsr.it.

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Classifications MeSH