Bystander IFN-γ activity promotes widespread and sustained cytokine signaling altering the tumor microenvironment.


Journal

Nature cancer
ISSN: 2662-1347
Titre abrégé: Nat Cancer
Pays: England
ID NLM: 101761119

Informations de publication

Date de publication:
03 2020
Historique:
entrez: 18 8 2020
pubmed: 18 8 2020
medline: 18 8 2020
Statut: ppublish

Résumé

The cytokine IFN-γ produced by tumor-reactive T cells is a key effector molecule with pleiotropic effects during anti-tumor immune responses. While IFN-γ production is targeted at the immunological synapse, its spatiotemporal activity within the tumor remains elusive. Here, we report that while IFN-γ secretion requires local antigen recognition, IFN-γ diffuses extensively to alter the tumor microenvironment in distant areas. Using intravital imaging and a reporter for STAT1 translocation, we provide evidence that T cells mediate sustained IFN-γ signaling in remote tumor cells. Furthermore, tumor phenotypic alterations required several hours of exposure to IFN-γ, a feature that disfavored local IFN-γ activity over diffusion and bystander activity. Finally, single-cell RNA-seq data from melanoma patients also suggested bystander IFN-γ activity in human tumors. Thus, tumor-reactive T cells act collectively to create large cytokine fields that profoundly modify the tumor microenvironment.

Identifiants

pubmed: 32803171
doi: 10.1038/s43018-020-0038-2
pmc: PMC7115926
mid: EMS88525
pii: 10.1038/s43018-020-0038-2
doi:

Substances chimiques

Cytokines 0
Interferon-gamma 82115-62-6

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Pagination

302-314

Subventions

Organisme : European Research Council
ID : 741167
Pays : International

Commentaires et corrections

Type : CommentIn

Déclaration de conflit d'intérêts

Competing interests statement The authors declare no competing interests.

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Auteurs

Ronan Thibaut (R)

Dynamics of Immune Responses Unit, Equipe Labellisée Ligue Contre le Cancer, Institut Pasteur, INSERM U1223, Paris, France.
University Paris Diderot, Sorbonne Paris Cité, Cellule Pasteur, Paris, France.

Pierre Bost (P)

Department of Immunology, Weizmann Institute of Science, Rehovot, Israel.
Systems Biology Group, Center for Bioinformatics, Biostatistics, and Integrative Biology and USR 3756, Institut Pasteur CNRS, Paris, France.
Sorbonne Université, Paris, France.

Idan Milo (I)

Dynamics of Immune Responses Unit, Equipe Labellisée Ligue Contre le Cancer, Institut Pasteur, INSERM U1223, Paris, France.

Marine Cazaux (M)

Dynamics of Immune Responses Unit, Equipe Labellisée Ligue Contre le Cancer, Institut Pasteur, INSERM U1223, Paris, France.
University Paris Diderot, Sorbonne Paris Cité, Cellule Pasteur, Paris, France.

Fabrice Lemaître (F)

Dynamics of Immune Responses Unit, Equipe Labellisée Ligue Contre le Cancer, Institut Pasteur, INSERM U1223, Paris, France.

Zacarias Garcia (Z)

Dynamics of Immune Responses Unit, Equipe Labellisée Ligue Contre le Cancer, Institut Pasteur, INSERM U1223, Paris, France.

Ido Amit (I)

Department of Immunology, Weizmann Institute of Science, Rehovot, Israel.

Béatrice Breart (B)

Dynamics of Immune Responses Unit, Equipe Labellisée Ligue Contre le Cancer, Institut Pasteur, INSERM U1223, Paris, France.

Clémence Cornuot (C)

Dynamics of Immune Responses Unit, Equipe Labellisée Ligue Contre le Cancer, Institut Pasteur, INSERM U1223, Paris, France.

Benno Schwikowski (B)

Systems Biology Group, Center for Bioinformatics, Biostatistics, and Integrative Biology and USR 3756, Institut Pasteur CNRS, Paris, France.

Philippe Bousso (P)

Dynamics of Immune Responses Unit, Equipe Labellisée Ligue Contre le Cancer, Institut Pasteur, INSERM U1223, Paris, France. philippe.bousso@pasteur.fr.

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