Hepatitis C Clearance by Direct-Acting Antivirals Impacts Glucose and Lipid Homeostasis.

direct-acting antivirals hepatic fibrosis hepatitis C virus insulin resistance

Journal

Journal of clinical medicine
ISSN: 2077-0383
Titre abrégé: J Clin Med
Pays: Switzerland
ID NLM: 101606588

Informations de publication

Date de publication:
21 Aug 2020
Historique:
received: 13 07 2020
revised: 13 08 2020
accepted: 14 08 2020
entrez: 23 8 2020
pubmed: 23 8 2020
medline: 23 8 2020
Statut: epublish

Résumé

Chronic hepatitis C virus (HCV) infections are causally linked with metabolic comorbidities such as insulin resistance, hepatic steatosis, and dyslipidemia. However, the clinical impact of HCV eradication achieved by direct-acting antivirals (DAAs) on glucose and lipid homeostasis is still controversial. The study aimed to prospectively investigate whether antiviral therapy of HCV with DAAs alters glucose and lipid parameters. 50 patients with chronic HCV who were treated with DAAs were screened, and 49 were enrolled in the study. Biochemical and virological data, as well as noninvasive liver fibrosis parameters, were prospectively collected at baseline, at the end of treatment (EOT) and 12 and 24 weeks post-treatment. 45 of 46 patients achieved sustained virologic response (SVR). The prevalence of insulin resistance (HOMA-IR) after HCV clearance was significantly lower, compared to baseline (5.3 ± 6.1 to 2.5 ± 1.9, Viral eradication following DAA therapy may have beneficial effects on glucose homeostasis, whereas lipid profile seems to be worsened.

Sections du résumé

BACKGROUND BACKGROUND
Chronic hepatitis C virus (HCV) infections are causally linked with metabolic comorbidities such as insulin resistance, hepatic steatosis, and dyslipidemia. However, the clinical impact of HCV eradication achieved by direct-acting antivirals (DAAs) on glucose and lipid homeostasis is still controversial. The study aimed to prospectively investigate whether antiviral therapy of HCV with DAAs alters glucose and lipid parameters.
METHODS METHODS
50 patients with chronic HCV who were treated with DAAs were screened, and 49 were enrolled in the study. Biochemical and virological data, as well as noninvasive liver fibrosis parameters, were prospectively collected at baseline, at the end of treatment (EOT) and 12 and 24 weeks post-treatment.
RESULTS RESULTS
45 of 46 patients achieved sustained virologic response (SVR). The prevalence of insulin resistance (HOMA-IR) after HCV clearance was significantly lower, compared to baseline (5.3 ± 6.1 to 2.5 ± 1.9,
CONCLUSION CONCLUSIONS
Viral eradication following DAA therapy may have beneficial effects on glucose homeostasis, whereas lipid profile seems to be worsened.

Identifiants

pubmed: 32825571
pii: jcm9092702
doi: 10.3390/jcm9092702
pmc: PMC7564474
pii:
doi:

Types de publication

Journal Article

Langues

eng

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Auteurs

Christiana Graf (C)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Tania Welzel (T)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Dimitra Bogdanou (D)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Johannes Vermehren (J)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Anita Beckel (A)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Jörg Bojunga (J)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Mireen Friedrich-Rust (M)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Julia Dietz (J)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Alica Kubesch (A)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Antonia Mondorf (A)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Sarah Fischer (S)

Infektiologikum, Center for Infectious Diseases, 60596 Frankfurt, Germany.

Thomas Lutz (T)

Infektiologikum, Center for Infectious Diseases, 60596 Frankfurt, Germany.

Philipp Stoffers (P)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Eva Herrmann (E)

Institute of Biostatistics and Mathematical Modeling, Goethe University Frankfurt, 60596 Frankfurt, Germany.

Thierry Poynard (T)

BioPredictive, 75007 Paris, France.

Stefan Zeuzem (S)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Georg Dultz (G)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Ulrike Mihm (U)

Department of Internal Medicine, University Hospital Frankfurt, 60596 Frankfurt, Germany.

Classifications MeSH