MCLR induces dysregulation of calcium homeostasis and endoplasmic reticulum stress resulting in apoptosis in Sertoli cells.


Journal

Chemosphere
ISSN: 1879-1298
Titre abrégé: Chemosphere
Pays: England
ID NLM: 0320657

Informations de publication

Date de publication:
Jan 2021
Historique:
received: 09 05 2020
revised: 20 07 2020
accepted: 27 07 2020
pubmed: 23 8 2020
medline: 22 12 2020
entrez: 23 8 2020
Statut: ppublish

Résumé

Microcystins-LR (MCLR) is a potent reproductive system toxin. We have previously shown that MCLR induced endoplasmic reticulum (ER) stress and apoptosis in testis. ER is the main calcium storage site in cells, and its calcium homeostasis plays an important role in the regulation of apoptosis. Hence, in the present study, we have investigated the role of calcium (Ca

Identifiants

pubmed: 32828052
pii: S0045-6535(20)32063-4
doi: 10.1016/j.chemosphere.2020.127868
pii:
doi:

Substances chimiques

Microcystins 0
Calcium SY7Q814VUP

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

127868

Informations de copyright

Copyright © 2020 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Sujuan Zhao (S)

School of Public Health, Anhui Medical University, Hefei, China.

Chunyang Yuan (C)

School of Public Health, Anhui Medical University, Hefei, China.

Xun Tuo (X)

College of Chemistry, Nanchang University, Nanchang, 330031, China.

Chengfan Zhou (C)

School of Public Health, Anhui Medical University, Hefei, China.

Qihong Zhao (Q)

School of Public Health, Anhui Medical University, Hefei, China.

Tong Shen (T)

School of Public Health, Anhui Medical University, Hefei, China. Electronic address: ahmusht@163.com.

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Classifications MeSH