Psychopathological precursors of the onset of mood disorders in offspring of parents with and without mood disorders: results of a 13-year prospective cohort high-risk study.


Journal

Journal of child psychology and psychiatry, and allied disciplines
ISSN: 1469-7610
Titre abrégé: J Child Psychol Psychiatry
Pays: England
ID NLM: 0375361

Informations de publication

Date de publication:
04 2021
Historique:
revised: 07 07 2020
received: 18 11 2019
accepted: 08 07 2020
pubmed: 26 8 2020
medline: 16 10 2021
entrez: 26 8 2020
Statut: ppublish

Résumé

There is still limited evidence from prospective high-risk research on the evolution of specific disorders that may emerge early in the development of mood disorders. Moreover, few studies have examined the specificity of mood disorder subtypes among offspring of parents with both major subtypes of mood disorders and controls based on prospective tracking across the transition from childhood to adulthood. Our specific objectives were to (a) identify differences in patterns of psychopathological precursors among youth with (hypo)mania compared to MDD and (b) examine whether these patterns differ by subtypes of parental mood disorders. Our data stem from a prospective cohort study of 449 directly interviewed offspring (51% female, mean age 10.1 years at study intake) of 88 patients with BPD, 71 with MDD, 30 with substance use disorders and 60 medical controls. The mean duration of follow-up was 13.2 years with evaluations conducted every three years. Within the whole cohort of offspring, MDE (Hazard Ratio = 4.44; 95%CI: 2.19-9.02), CD (HR = 3.31;1.55-7.07) and DUD (HR = 2.54; 1.15-5.59) predicted the onset of (hypo)manic episodes, whereas MDD in offspring was predicted by SAD (HR = 1.53; 1.09-2.15), generalized anxiety (HR = 2.56; 1.05-6.24), and panic disorder (HR = 3.13; 1.06-9.23). The early predictors of (hypo)mania in the whole cohort were also significantly associated with the onset of (hypo)mania among the offspring of parents with BPD. The onset of mood disorders is frequently preceded by identifiable depressive episodes and nonmood disorders. These precursors differed by mood subtype in offspring. High-risk offspring with these precursors should be closely monitored to prevent the further development of MDD or conversion to BPD.

Sections du résumé

BACKGROUND
There is still limited evidence from prospective high-risk research on the evolution of specific disorders that may emerge early in the development of mood disorders. Moreover, few studies have examined the specificity of mood disorder subtypes among offspring of parents with both major subtypes of mood disorders and controls based on prospective tracking across the transition from childhood to adulthood. Our specific objectives were to (a) identify differences in patterns of psychopathological precursors among youth with (hypo)mania compared to MDD and (b) examine whether these patterns differ by subtypes of parental mood disorders.
METHODS
Our data stem from a prospective cohort study of 449 directly interviewed offspring (51% female, mean age 10.1 years at study intake) of 88 patients with BPD, 71 with MDD, 30 with substance use disorders and 60 medical controls. The mean duration of follow-up was 13.2 years with evaluations conducted every three years.
RESULTS
Within the whole cohort of offspring, MDE (Hazard Ratio = 4.44; 95%CI: 2.19-9.02), CD (HR = 3.31;1.55-7.07) and DUD (HR = 2.54; 1.15-5.59) predicted the onset of (hypo)manic episodes, whereas MDD in offspring was predicted by SAD (HR = 1.53; 1.09-2.15), generalized anxiety (HR = 2.56; 1.05-6.24), and panic disorder (HR = 3.13; 1.06-9.23). The early predictors of (hypo)mania in the whole cohort were also significantly associated with the onset of (hypo)mania among the offspring of parents with BPD.
CONCLUSIONS
The onset of mood disorders is frequently preceded by identifiable depressive episodes and nonmood disorders. These precursors differed by mood subtype in offspring. High-risk offspring with these precursors should be closely monitored to prevent the further development of MDD or conversion to BPD.

Identifiants

pubmed: 32841378
doi: 10.1111/jcpp.13307
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

404-413

Informations de copyright

© 2020 Association for Child and Adolescent Mental Health.

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Auteurs

Dominique Rudaz (D)

Department of Psychiatry, Lausanne University Hospital and University of Lausanne, Lausanne, Switzerland.

Caroline L Vandeleur (CL)

Department of Psychiatry, Lausanne University Hospital and University of Lausanne, Lausanne, Switzerland.

Mehdi Gholam (M)

Department of Psychiatry, Lausanne University Hospital and University of Lausanne, Lausanne, Switzerland.

Enrique Castelao (E)

Department of Psychiatry, Lausanne University Hospital and University of Lausanne, Lausanne, Switzerland.

Marie-Pierre F Strippoli (MF)

Department of Psychiatry, Lausanne University Hospital and University of Lausanne, Lausanne, Switzerland.

Pierre Marquet (P)

Department of Psychiatry, Lausanne University Hospital and University of Lausanne, Lausanne, Switzerland.
Institute of Mental Health, Laval University, Québec, QC, Canada.

Jean-Michel Aubry (JM)

Department of Psychiatry, University Hospital of Geneva, Geneva, Switzerland.

Kathleen R Merikangas (KR)

Genetic Epidemiology Research Branch, Intramural Research Program, National Institute of Mental Health, Bethesda, MD, USA.

Martin Preisig (M)

Department of Psychiatry, Lausanne University Hospital and University of Lausanne, Lausanne, Switzerland.

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