NLRP3 played a role in Trichinella spiralis-triggered Th2 and regulatory T cells response.


Journal

Veterinary research
ISSN: 1297-9716
Titre abrégé: Vet Res
Pays: England
ID NLM: 9309551

Informations de publication

Date de publication:
27 Aug 2020
Historique:
received: 09 07 2020
accepted: 10 08 2020
entrez: 29 8 2020
pubmed: 29 8 2020
medline: 26 3 2021
Statut: epublish

Résumé

Trichinella spiralis maintains chronic infections within its host. Muscle larvae excretory-secretory products (MLES) typically induce parasite-specific immune responses such as the Th2 response and regulatory T cells (Tregs) by modulating dendritic cell (DC) phenotype via the recognition of pattern recognition receptors (PRRs), such as Nod-like receptors (NLRs). We aimed to investigate the role of NLRP3 in T. spiralis-triggered immune response. We found that larvae burden was increased in NLRP3

Identifiants

pubmed: 32854770
doi: 10.1186/s13567-020-00829-2
pii: 10.1186/s13567-020-00829-2
pmc: PMC7457311
doi:

Substances chimiques

NLR Family, Pyrin Domain-Containing 3 Protein 0
Nlrp3 protein, mouse 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

107

Subventions

Organisme : National Natural Science Foundation of China
ID : 31520103916
Organisme : National Natural Science Foundation of China
ID : 31872467
Organisme : National Key Research and Development Program of China
ID : 2017YFD0501300
Organisme : Guangdong Innovative and Enterpreneurial Research Team Program
ID : 2014ZT05S123
Organisme : Department of Science and Technology of Jilin Province (CN)
ID : 20180520042JH

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Auteurs

Xuemin Jin (X)

Key Laboratory of Zoonosis Research, Ministry of Education, Institute of Zoonosis, College of Veterinary Medicine, Jilin University, Changchun, 130062, China.

Xue Bai (X)

Key Laboratory of Zoonosis Research, Ministry of Education, Institute of Zoonosis, College of Veterinary Medicine, Jilin University, Changchun, 130062, China.

Yong Yang (Y)

Key Laboratory of Zoonosis Research, Ministry of Education, Institute of Zoonosis, College of Veterinary Medicine, Jilin University, Changchun, 130062, China.

Jing Ding (J)

Key Laboratory of Zoonosis Research, Ministry of Education, Institute of Zoonosis, College of Veterinary Medicine, Jilin University, Changchun, 130062, China.

Haining Shi (H)

Mucosal Immunology and Biology Research Center, Massachusetts General Hospital, Charlestown, MA, USA.

Baoquan Fu (B)

Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou, China.

Pascal Boireau (P)

JRU BIPAR, ANSES, École Nationale Vétérinaire d'Alfort, INRA, Université Paris-Est, Animal Health Laboratory, Maisons-Alfort, France.

Mingyuan Liu (M)

Key Laboratory of Zoonosis Research, Ministry of Education, Institute of Zoonosis, College of Veterinary Medicine, Jilin University, Changchun, 130062, China. liumy36@163.com.
Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, Yangzhou, Jiangsu, People's Republic of China. liumy36@163.com.

Xiaolei Liu (X)

Key Laboratory of Zoonosis Research, Ministry of Education, Institute of Zoonosis, College of Veterinary Medicine, Jilin University, Changchun, 130062, China. liuxlei@163.com.

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Classifications MeSH