Prognostic significance of subtle coronary calcification in patients with zero coronary artery calcium score: From the CONFIRM registry.


Journal

Atherosclerosis
ISSN: 1879-1484
Titre abrégé: Atherosclerosis
Pays: Ireland
ID NLM: 0242543

Informations de publication

Date de publication:
09 2020
Historique:
received: 08 04 2020
revised: 18 06 2020
accepted: 15 07 2020
pubmed: 31 8 2020
medline: 24 6 2021
entrez: 31 8 2020
Statut: ppublish

Résumé

The Agatston coronary artery calcium score (CACS) may fail to identify small or less dense coronary calcification that can be detected on coronary CT angiography (CCTA). We investigated the prevalence and prognostic importance of subtle calcified plaques on CCTA among individuals with CACS 0. From the prospective multicenter CONFIRM registry, we evaluated patients without known CAD who underwent CAC scan and CCTA. CACS was categorized as 0, 1-10, 11-100, 101-400, and >400. Patients with CACS 0 were stratified according to the visual presence of coronary plaques on CCTA. Plaque composition was categorized as non-calcified (NCP), mixed (MP) and calcified (CP). The primary outcome was a major adverse cardiac event (MACE) which was defined as death and myocardial infarction. Of 4049 patients, 1741 (43%) had a CACS 0. NCP and plaques that contained calcium (MP or CP) were detected by CCTA in 110 patients (6% of CACS 0) and 64 patients (4% of CACS 0), respectively. During a 5.6 years median follow-up (IQR 5.1-6.2 years), 413 MACE events occurred (13%). Patients with CACS 0 and MP/CP detected by CCTA had similar MACE risk compared to patients with CACS 1-10 (p = 0.868). In patients with CACS 0, after adjustment for risk factors and symptom, MP/CP was associated with an increased MACE risk compared to those with entirely normal CCTA (HR 2.39, 95% CI [1.09-5.24], p = 0.030). A small but non-negligible proportion of patients with CACS 0 had identifiable coronary calcification, which was associated with increased MACE risk. Modifying CAC image acquisition and/or scoring methods could improve the detection of subtle coronary calcification.

Sections du résumé

BACKGROUND AND AIMS
The Agatston coronary artery calcium score (CACS) may fail to identify small or less dense coronary calcification that can be detected on coronary CT angiography (CCTA). We investigated the prevalence and prognostic importance of subtle calcified plaques on CCTA among individuals with CACS 0.
METHODS
From the prospective multicenter CONFIRM registry, we evaluated patients without known CAD who underwent CAC scan and CCTA. CACS was categorized as 0, 1-10, 11-100, 101-400, and >400. Patients with CACS 0 were stratified according to the visual presence of coronary plaques on CCTA. Plaque composition was categorized as non-calcified (NCP), mixed (MP) and calcified (CP). The primary outcome was a major adverse cardiac event (MACE) which was defined as death and myocardial infarction.
RESULTS
Of 4049 patients, 1741 (43%) had a CACS 0. NCP and plaques that contained calcium (MP or CP) were detected by CCTA in 110 patients (6% of CACS 0) and 64 patients (4% of CACS 0), respectively. During a 5.6 years median follow-up (IQR 5.1-6.2 years), 413 MACE events occurred (13%). Patients with CACS 0 and MP/CP detected by CCTA had similar MACE risk compared to patients with CACS 1-10 (p = 0.868). In patients with CACS 0, after adjustment for risk factors and symptom, MP/CP was associated with an increased MACE risk compared to those with entirely normal CCTA (HR 2.39, 95% CI [1.09-5.24], p = 0.030).
CONCLUSIONS
A small but non-negligible proportion of patients with CACS 0 had identifiable coronary calcification, which was associated with increased MACE risk. Modifying CAC image acquisition and/or scoring methods could improve the detection of subtle coronary calcification.

Identifiants

pubmed: 32862086
pii: S0021-9150(20)30378-6
doi: 10.1016/j.atherosclerosis.2020.07.011
pii:
doi:

Substances chimiques

Calcium SY7Q814VUP

Types de publication

Journal Article Multicenter Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

33-38

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Auteurs

Donghee Han (D)

Department of Imaging, Cedars Sinai Medical Center, Los Angeles, CA, USA.

Eyal Klein (E)

Department of Imaging, Cedars Sinai Medical Center, Los Angeles, CA, USA.

John Friedman (J)

Department of Imaging, Cedars Sinai Medical Center, Los Angeles, CA, USA.

Heidi Gransar (H)

Department of Imaging, Cedars Sinai Medical Center, Los Angeles, CA, USA.

Stephan Achenbach (S)

Department of Medicine, University of Erlangen, Erlangen, Germany.

Mouaz H Al-Mallah (MH)

Department of Medicine, Wayne State University, Henry Ford Hospital, Detroit, MI, USA.

Matthew J Budoff (MJ)

Department of Medicine, Harbor UCLA Medical Center, Los Angeles, CA, USA.

Filippo Cademartiri (F)

Department of Radiology/Centre de Recherche, Montreal Heart Institute/Unniversitè de Montreal, Montreal, Quebec, Canada.

Erica Maffei (E)

Department of Radiology/Centre de Recherche, Montreal Heart Institute/Unniversitè de Montreal, Montreal, Quebec, Canada.

Tracy Q Callister (TQ)

Tennessee Heart and Vascular Institute, Hendersonville, TN, USA.

Kavitha Chinnaiyan (K)

William Beaumont Hospital, Royal Oaks, MI, USA.

Benjamin J W Chow (BJW)

Department of Medicine and Radiology, University of Ottawa, ON, Canada.

Augustin DeLago (A)

Capitol Cardiology Associate, Albany, NY, USA.

Martin Hadamitzky (M)

Deutsches Herzzentrum Munchen, Munich, Germany.

Joerg Hausleiter (J)

Medizinische Klinik I der Ludwig-Maximilians-Universität München, Munich, Germany.

Philipp A Kaufmann (PA)

University Hospital, Zurich, Switzerland.

Todd C Villines (TC)

Department of Medicine, Walter Reed Medical Center, Washington, DC, USA.

Yong-Jin Kim (YJ)

Seoul National University Hospital, Seoul, South Korea.

Jonathon Leipsic (J)

Department of Medicine and Radiology, University of British Columbia, Vancouver, BC, Canada.

Gudrun Feuchtner (G)

Department of Radiology, Medical University of Innsbruck, Innsbruck, Austria.

Ricardo C Cury (RC)

Baptist Cardiac and Vascular Institute, Miami, FL, USA.

Gianluca Pontone (G)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Daniele Andreini (D)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Hugo Marques (H)

Department of Surgery, Curry Cabral Hospital, Lisbon, Portugal.

Ronen Rubinshtein (R)

Department of Cardiology at the Lady Davis Carmel Medical Center, The Ruth and Bruce Rappaport School of Medicine, Technion-Israel Institute of Technology, Haifa, Israel.

Hyuk-Jae Chang (HJ)

Division of Cardiology, Severance Cardiovascular Hospital and Severance Biomedical Science Institute, Yonsei University College of Medicine, Yonsei University Health System, Seoul, South Korea.

Fay Y Lin (FY)

Dalio Institute of Cardiovascular Imaging, Department of Radiology, New York-Presbyterian Hospital and the Weill Cornell Medicine, New York, NY, USA.

Leslee J Shaw (LJ)

Dalio Institute of Cardiovascular Imaging, Department of Radiology, New York-Presbyterian Hospital and the Weill Cornell Medicine, New York, NY, USA.

James K Min (JK)

Dalio Institute of Cardiovascular Imaging, Department of Radiology, New York-Presbyterian Hospital and the Weill Cornell Medicine, New York, NY, USA.

Daniel S Berman (DS)

Department of Imaging, Cedars Sinai Medical Center, Los Angeles, CA, USA. Electronic address: Daniel.Berman@cshs.org.

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