High pre-operative fasting blood glucose levels predict a poor prognosis in patients with pancreatic neuroendocrine tumour.


Journal

Endocrine
ISSN: 1559-0100
Titre abrégé: Endocrine
Pays: United States
ID NLM: 9434444

Informations de publication

Date de publication:
02 2021
Historique:
received: 07 05 2020
accepted: 19 08 2020
pubmed: 31 8 2020
medline: 9 7 2021
entrez: 31 8 2020
Statut: ppublish

Résumé

Hyperglycaemia has been indicated as a pro-tumoural factor; however, the prognostic role of diabetes mellitus (DM) in pancreatic neuroendocrine tumours (panNETs) remains ambiguous, partly due to the effects of anti-diabetic drugs. We hypothesise that the blood sugar level per se affects the outcome of panNETs, and thus, we investigated the prognostic significance of the fasting blood glucose (FBG) level in resected panNET patients with no pre-existing DM. A retrospective cohort study comprising 201 patients with radically resected non-functional panNETs was conducted. A total of 164 patients without pre-existing DM were further studied. An FBG level greater than 5.6 mmol/L was defined as high (otherwise, normal). Survival was evaluated using Kaplan-Meier methods and log-rank tests. Multivariate analyses for survival were performed using the Cox regression model. High FBG levels were significantly associated with poor overall survival (OS; p = 0.019) and recurrence-free survival (RFS; p = 0.011) in resected patients with panNET who had no pre-existing DM. The multivariable-adjusted hazard ratios (HRs) for mortality and recurrence comparing patients with high and normal FBG levels were 12.19 (95% confidence interval (CI) = 1.15-128.78, p = 0.038) and 2.43 (95% CI = 1.03-5.72, p = 0.042), respectively. A pre-operative FBG level greater than 5.6 mmol/L is associated with poor OS and RFS metastasis for patients with panNET who undergo radical surgical resection.

Sections du résumé

BACKGROUND
Hyperglycaemia has been indicated as a pro-tumoural factor; however, the prognostic role of diabetes mellitus (DM) in pancreatic neuroendocrine tumours (panNETs) remains ambiguous, partly due to the effects of anti-diabetic drugs. We hypothesise that the blood sugar level per se affects the outcome of panNETs, and thus, we investigated the prognostic significance of the fasting blood glucose (FBG) level in resected panNET patients with no pre-existing DM.
METHODS
A retrospective cohort study comprising 201 patients with radically resected non-functional panNETs was conducted. A total of 164 patients without pre-existing DM were further studied. An FBG level greater than 5.6 mmol/L was defined as high (otherwise, normal). Survival was evaluated using Kaplan-Meier methods and log-rank tests. Multivariate analyses for survival were performed using the Cox regression model.
RESULTS
High FBG levels were significantly associated with poor overall survival (OS; p = 0.019) and recurrence-free survival (RFS; p = 0.011) in resected patients with panNET who had no pre-existing DM. The multivariable-adjusted hazard ratios (HRs) for mortality and recurrence comparing patients with high and normal FBG levels were 12.19 (95% confidence interval (CI) = 1.15-128.78, p = 0.038) and 2.43 (95% CI = 1.03-5.72, p = 0.042), respectively.
CONCLUSION
A pre-operative FBG level greater than 5.6 mmol/L is associated with poor OS and RFS metastasis for patients with panNET who undergo radical surgical resection.

Identifiants

pubmed: 32862321
doi: 10.1007/s12020-020-02469-0
pii: 10.1007/s12020-020-02469-0
doi:

Substances chimiques

Blood Glucose 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

494-501

Subventions

Organisme : National Natural Science Foundation of China (CN)
ID : 81625016, 81871940, 81902417

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Auteurs

Yitao Gong (Y)

Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, 200032, Shanghai, China.
Department of Oncology, Shanghai Medical College, Fudan University, 200032, Shanghai, China.
Shanghai Pancreatic Cancer Institute, 200032, Shanghai, China.
Pancreatic Cancer Institute, Fudan University, 200032, Shanghai, China.

Zhiyao Fan (Z)

Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, 200032, Shanghai, China.
Department of Oncology, Shanghai Medical College, Fudan University, 200032, Shanghai, China.
Shanghai Pancreatic Cancer Institute, 200032, Shanghai, China.
Pancreatic Cancer Institute, Fudan University, 200032, Shanghai, China.

Pin Zhang (P)

Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, 200032, Shanghai, China.
Department of Oncology, Shanghai Medical College, Fudan University, 200032, Shanghai, China.
Shanghai Pancreatic Cancer Institute, 200032, Shanghai, China.
Pancreatic Cancer Institute, Fudan University, 200032, Shanghai, China.

Yunzhen Qian (Y)

Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, 200032, Shanghai, China.
Department of Oncology, Shanghai Medical College, Fudan University, 200032, Shanghai, China.
Shanghai Pancreatic Cancer Institute, 200032, Shanghai, China.
Pancreatic Cancer Institute, Fudan University, 200032, Shanghai, China.

Qiuyi Huang (Q)

Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, 200032, Shanghai, China.
Department of Oncology, Shanghai Medical College, Fudan University, 200032, Shanghai, China.
Shanghai Pancreatic Cancer Institute, 200032, Shanghai, China.
Pancreatic Cancer Institute, Fudan University, 200032, Shanghai, China.

Shengming Deng (S)

Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, 200032, Shanghai, China.
Department of Oncology, Shanghai Medical College, Fudan University, 200032, Shanghai, China.
Shanghai Pancreatic Cancer Institute, 200032, Shanghai, China.
Pancreatic Cancer Institute, Fudan University, 200032, Shanghai, China.

Guopei Luo (G)

Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, 200032, Shanghai, China.
Department of Oncology, Shanghai Medical College, Fudan University, 200032, Shanghai, China.
Shanghai Pancreatic Cancer Institute, 200032, Shanghai, China.
Pancreatic Cancer Institute, Fudan University, 200032, Shanghai, China.

He Cheng (H)

Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, 200032, Shanghai, China.
Department of Oncology, Shanghai Medical College, Fudan University, 200032, Shanghai, China.
Shanghai Pancreatic Cancer Institute, 200032, Shanghai, China.
Pancreatic Cancer Institute, Fudan University, 200032, Shanghai, China.

Kaizhou Jin (K)

Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, 200032, Shanghai, China.
Department of Oncology, Shanghai Medical College, Fudan University, 200032, Shanghai, China.
Shanghai Pancreatic Cancer Institute, 200032, Shanghai, China.
Pancreatic Cancer Institute, Fudan University, 200032, Shanghai, China.

Quanxing Ni (Q)

Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, 200032, Shanghai, China.
Department of Oncology, Shanghai Medical College, Fudan University, 200032, Shanghai, China.
Shanghai Pancreatic Cancer Institute, 200032, Shanghai, China.
Pancreatic Cancer Institute, Fudan University, 200032, Shanghai, China.

Xianjun Yu (X)

Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, 200032, Shanghai, China. yuxianjun@fudanpci.org.
Department of Oncology, Shanghai Medical College, Fudan University, 200032, Shanghai, China. yuxianjun@fudanpci.org.
Shanghai Pancreatic Cancer Institute, 200032, Shanghai, China. yuxianjun@fudanpci.org.
Pancreatic Cancer Institute, Fudan University, 200032, Shanghai, China. yuxianjun@fudanpci.org.

Chen Liu (C)

Department of Pancreatic Surgery, Fudan University Shanghai Cancer Center, 200032, Shanghai, China. liuchen@fudanpci.org.
Department of Oncology, Shanghai Medical College, Fudan University, 200032, Shanghai, China. liuchen@fudanpci.org.
Shanghai Pancreatic Cancer Institute, 200032, Shanghai, China. liuchen@fudanpci.org.
Pancreatic Cancer Institute, Fudan University, 200032, Shanghai, China. liuchen@fudanpci.org.

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