MerTK Expression and ERK Activation Are Essential for the Functional Maturation of Osteopontin-Producing Reparative Macrophages After Myocardial Infarction.
Animals
Disease Models, Animal
Flow Cytometry
MAP Kinase Signaling System
/ physiology
Macrophages
/ metabolism
Mice
Mice, Inbred C57BL
Myocardial Infarction
/ pathology
Osteopontin
/ metabolism
Real-Time Polymerase Chain Reaction
STAT3 Transcription Factor
/ metabolism
c-Mer Tyrosine Kinase
/ metabolism
MerTK
galectin‐3
macrophage
macrophage colony‐stimulating factor
myocardial infarction
osteopontin
Journal
Journal of the American Heart Association
ISSN: 2047-9980
Titre abrégé: J Am Heart Assoc
Pays: England
ID NLM: 101580524
Informations de publication
Date de publication:
15 09 2020
15 09 2020
Historique:
pubmed:
1
9
2020
medline:
12
3
2021
entrez:
1
9
2020
Statut:
ppublish
Résumé
Background We previously reported that osteopontin plays an essential role in accelerating both reparative fibrosis and clearance of dead cells (efferocytosis) during tissue repair after myocardial infarction (MI) and galectin-3
Identifiants
pubmed: 32865099
doi: 10.1161/JAHA.120.017071
pmc: PMC7726992
doi:
Substances chimiques
STAT3 Transcription Factor
0
Spp1 protein, mouse
0
Stat3 protein, mouse
0
Osteopontin
106441-73-0
Mertk protein, mouse
EC 2.7.10.1
c-Mer Tyrosine Kinase
EC 2.7.10.1
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
e017071Références
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