Dual CD4-based CAR T cells with distinct costimulatory domains mitigate HIV pathogenesis in vivo.


Journal

Nature medicine
ISSN: 1546-170X
Titre abrégé: Nat Med
Pays: United States
ID NLM: 9502015

Informations de publication

Date de publication:
11 2020
Historique:
received: 27 01 2020
accepted: 28 07 2020
pubmed: 2 9 2020
medline: 9 1 2021
entrez: 2 9 2020
Statut: ppublish

Résumé

An effective strategy to cure HIV will likely require a potent and sustained antiviral T cell response. Here we explored the utility of chimeric antigen receptor (CAR) T cells, expressing the CD4 ectodomain to confer specificity for the HIV envelope, to mitigate HIV-induced pathogenesis in bone marrow, liver, thymus (BLT) humanized mice. CAR T cells expressing the 4-1BB/CD3-ζ endodomain were insufficient to prevent viral rebound and CD4

Identifiants

pubmed: 32868878
doi: 10.1038/s41591-020-1039-5
pii: 10.1038/s41591-020-1039-5
pmc: PMC9422086
mid: NIHMS1827405
doi:

Substances chimiques

Antibodies, Monoclonal, Humanized 0
CD3 Complex 0
CD3 antigen, zeta chain 0
CD4 Antigens 0
CXCR4 protein, mouse 0
HIV Envelope Protein gp41 0
Peptide Fragments 0
Receptors, CXCR4 0
Receptors, Chimeric Antigen 0
Tumor Necrosis Factor Receptor Superfamily, Member 9 0
peptide C34 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1776-1787

Subventions

Organisme : NIAID NIH HHS
ID : UM1 AI126620
Pays : United States
Organisme : NIAID NIH HHS
ID : T32 AI007632
Pays : United States
Organisme : NIAID NIH HHS
ID : UM1 AI164570
Pays : United States
Organisme : NIAID NIH HHS
ID : U19 AI117950
Pays : United States
Organisme : NIAID NIH HHS
ID : P30 AI045008
Pays : United States
Organisme : NHLBI NIH HHS
ID : U19 HL129903
Pays : United States
Organisme : NIAID NIH HHS
ID : U19 AI149680
Pays : United States
Organisme : NIAID NIH HHS
ID : F32 AI136750
Pays : United States

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Auteurs

Colby R Maldini (CR)

Department of Microbiology, Center for Cellular Immunotherapies, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.

Daniel T Claiborne (DT)

Ragon Institute of MGH, MIT and Harvard, Cambridge, MA, USA.

Ken Okawa (K)

Ragon Institute of MGH, MIT and Harvard, Cambridge, MA, USA.

Tao Chen (T)

Ragon Institute of MGH, MIT and Harvard, Cambridge, MA, USA.

Derrick L Dopkin (DL)

Department of Pathology & Laboratory Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.

Xiaochuan Shan (X)

Department of Pathology & Laboratory Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.

Karen A Power (KA)

Ragon Institute of MGH, MIT and Harvard, Cambridge, MA, USA.

Radiana T Trifonova (RT)

Ragon Institute of MGH, MIT and Harvard, Cambridge, MA, USA.

Katharine Krupp (K)

Ragon Institute of MGH, MIT and Harvard, Cambridge, MA, USA.

Meredith Phelps (M)

Ragon Institute of MGH, MIT and Harvard, Cambridge, MA, USA.

Vladimir D Vrbanac (VD)

Ragon Institute of MGH, MIT and Harvard, Cambridge, MA, USA.
Human Immune System Mouse Program, Massachusetts General Hospital, Boston, MA, USA.

Serah Tanno (S)

Ragon Institute of MGH, MIT and Harvard, Cambridge, MA, USA.
Human Immune System Mouse Program, Massachusetts General Hospital, Boston, MA, USA.

Timothy Bateson (T)

Ragon Institute of MGH, MIT and Harvard, Cambridge, MA, USA.

George J Leslie (GJ)

Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.

James A Hoxie (JA)

Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.

Christian L Boutwell (CL)

Ragon Institute of MGH, MIT and Harvard, Cambridge, MA, USA.

James L Riley (JL)

Department of Microbiology, Center for Cellular Immunotherapies, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA. rileyj@upenn.edu.

Todd M Allen (TM)

Ragon Institute of MGH, MIT and Harvard, Cambridge, MA, USA. tallen2@mgh.harvard.edu.

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