Cumulative Damage in Juvenile Idiopathic Arthritis: A Multicenter Study From the Pediatric Rheumatology Arab Group.


Journal

Arthritis care & research
ISSN: 2151-4658
Titre abrégé: Arthritis Care Res (Hoboken)
Pays: United States
ID NLM: 101518086

Informations de publication

Date de publication:
04 2021
Historique:
received: 20 04 2020
accepted: 27 08 2020
pubmed: 5 9 2020
medline: 27 4 2021
entrez: 4 9 2020
Statut: ppublish

Résumé

To report the cumulative articular and extraarticular damage in Arab children with juvenile idiopathic arthritis (JIA) and to identify variables that correlate with disease damage. We conducted a multicenter, cross-sectional study among 14 pediatric rheumatology centers from 7 Arab countries. JIA patients who met the International League of Associations for Rheumatology classification criteria and had a disease duration of >1 year were enrolled. Disease activity status was assessed using the Juvenile Arthritis Multidimensional Assessment Report. Disease damage was assessed by the Juvenile Arthritis Damage Index, articular (JADI-A) and extraarticular (JADI-E). A total of 702 (471 female) JIA patients with a median age of 11.3 years (interquartile range [IQR] 8.0-14.0 years) were studied. Median age at disease onset was 5 years (IQR 2.0-9.0 years) and the median disease duration was 4 years (IQR 2.0-7.0 years). The most frequent JIA categories were oligoarticular JIA (34.9%), polyarticular JIA (29.5%), and systemic JIA (24.5%). Clinical remission was achieved in 73.9% of patients. At the last clinic visit, 193 patients experienced joint damage, with a mean ± SD JADI-A score of 1.7 ± 4.5, while 156 patients had extraarticular damage, with a mean ± SD JADI-E score of 0.5 ± 1.1. Patients with enthesitis-related arthritis had the highest JADI-A score. JADI-A correlated significantly with the presence of a family history of JIA. JADI-A and JADI-E had a significant correlation with long disease duration. Cumulative damage was common in this Arab JIA cohort, and consanguinity and JIA in a sibling were frequent findings and were associated with a greater cumulative damage.

Identifiants

pubmed: 32886859
doi: 10.1002/acr.24436
doi:

Substances chimiques

Antirheumatic Agents 0

Types de publication

Journal Article Multicenter Study Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

586-592

Informations de copyright

© 2020, American College of Rheumatology.

Références

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Auteurs

Sulaiman M Al-Mayouf (SM)

King Faisal Specialist Hospital and Research Center, Riyadh, Saudi Arabia.

Soad Hashad (S)

Tripoli Childrens Hospital, Tripoli, Libya.

Khulood Khawaja (K)

Al-Mafraq Hospital, Abu Dhabi, United Arab Emirates.

Abeer Alrasheedi (A)

King Faisal Specialist Hospital and Research Center, Riyadh, Saudi Arabia.

Reem Abdwani (R)

Sultan Qaboos University Hospital, Muscat, Oman.

Awatif Abushhaiwia (A)

Tripoli Childrens Hospital, Tripoli, Libya.

Muatasem AlSuwaiti (M)

Queen Rania Childrens Hospital, Amman, Jordan.

Raed Alzyoud (R)

Queen Rania Childrens Hospital, Amman, Jordan.

Safiya Al Abrawi (S)

Royal Hospital, Muscat, Oman.

Abdulrahman Asiri (A)

Prince Sultan Military Medical City, Riyadh, Saudi Arabia.

Manal Alshaikh (M)

King Faisal Specialist Hospital and Research Center, Riyadh, Saudi Arabia.

Elsadeg Sharif (E)

Al Ain Hospital, Al Ain, United Arab Emirates.

Mohammed Muzaffer (M)

King Abdulaziz University, Jeddah, Saudi Arabia.

Wafa Alsewairi (W)

King Abdulaziz Medical City, Riyadh, Saudi Arabia.

Mabruka Zlenti (M)

Tripoli Childrens Hospital, Tripoli, Libya.

Ebtisam Kawaja (E)

Tripoli Childrens Hospital, Tripoli, Libya.

Muna Almutairi (M)

Adan Hospital, Hadiya, Kuwait.

Mahmoud Majeed (M)

King Fahad Armed Forces Hospital, Jeddah, Saudi Arabia.

Hala Lotfy (H)

Medical School, Cairo University, Cairo, Egypt.

Munira AlMarri (M)

Prince Sultan Military Medical City, Riyadh, Saudi Arabia.

Nora Almutairi (N)

AlSabah Hospital, Kuwait City, Kuwait.

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