Human epididymis protein 4 is a new biomarker to predict the prognosis of progressive fibrosing interstitial lung disease.


Journal

Respiratory investigation
ISSN: 2212-5353
Titre abrégé: Respir Investig
Pays: Netherlands
ID NLM: 101581124

Informations de publication

Date de publication:
Jan 2021
Historique:
received: 13 06 2020
revised: 31 07 2020
accepted: 03 08 2020
pubmed: 14 9 2020
medline: 29 6 2021
entrez: 13 9 2020
Statut: ppublish

Résumé

The clinical course and prognosis of progressive fibrosing interstitial lung diseases (PF-ILDs) vary between individuals. Notably, predictive serum biomarkers for disease management are needed. Serum human epididymis protein 4 (HE4) is reportedly elevated in patients with idiopathic pulmonary fibrosis (IPF); however, its clinical utility remains unknown. We evaluated the potential of serum HE4 as a biomarker for patients with PF-ILD. Serum HE4 was measured in a retrospective study consisting of 34 patients with PF-ILD and 40 healthy volunteers. The relationship between serum HE4 levels and clinical parameters or prognosis was investigated. To validate the significance of results obtained, a prospective observational study was performed in 37 patients presenting PF-ILD and 40 control patients without PF-ILD. Serum HE4 levels were higher in patients with PF-ILD than in healthy volunteers (P < 0.01). Moreover, serum HE4 levels correlated with the extent of honeycombing on chest high-resolution computed tomography (r = 0.41, P = 0.015). In multivariate analysis using the Cox proportional hazard model, higher HE4 levels (>238 pmol/L) were associated with an elevated mortality risk; hazard ratio (HR) 7.27, 95% CI 1.56-34.0, P = 0.01 in the derivation cohort; HR 44.3, 95% CI 4.19-468, P < 0.01 in validation cohort. Serum HE4 levels may serve as a new diagnostic and prognostic biomarker for patients with PF-ILD.

Sections du résumé

BACKGROUND BACKGROUND
The clinical course and prognosis of progressive fibrosing interstitial lung diseases (PF-ILDs) vary between individuals. Notably, predictive serum biomarkers for disease management are needed. Serum human epididymis protein 4 (HE4) is reportedly elevated in patients with idiopathic pulmonary fibrosis (IPF); however, its clinical utility remains unknown. We evaluated the potential of serum HE4 as a biomarker for patients with PF-ILD.
METHODS METHODS
Serum HE4 was measured in a retrospective study consisting of 34 patients with PF-ILD and 40 healthy volunteers. The relationship between serum HE4 levels and clinical parameters or prognosis was investigated. To validate the significance of results obtained, a prospective observational study was performed in 37 patients presenting PF-ILD and 40 control patients without PF-ILD.
RESULTS RESULTS
Serum HE4 levels were higher in patients with PF-ILD than in healthy volunteers (P < 0.01). Moreover, serum HE4 levels correlated with the extent of honeycombing on chest high-resolution computed tomography (r = 0.41, P = 0.015). In multivariate analysis using the Cox proportional hazard model, higher HE4 levels (>238 pmol/L) were associated with an elevated mortality risk; hazard ratio (HR) 7.27, 95% CI 1.56-34.0, P = 0.01 in the derivation cohort; HR 44.3, 95% CI 4.19-468, P < 0.01 in validation cohort.
CONCLUSIONS CONCLUSIONS
Serum HE4 levels may serve as a new diagnostic and prognostic biomarker for patients with PF-ILD.

Identifiants

pubmed: 32919933
pii: S2212-5345(20)30118-0
doi: 10.1016/j.resinv.2020.08.002
pii:
doi:

Substances chimiques

Biomarkers 0
WAP Four-Disulfide Core Domain Protein 2 0
WFDC2 protein, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

90-98

Informations de copyright

Copyright © 2020 The Japanese Respiratory Society. Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Conflict of Interest Abbott Japan LLC covered the costs for serum HE4 measurement. K.M has an employment relationship with Abbott Japan, LLC.

Auteurs

Naoki Nishiyama (N)

Department of Respiratory Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo, 113-8519, Japan. Electronic address: nispulm@tmd.ac.jp.

Masahiro Masuo (M)

Department of Respiratory Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo, 113-8519, Japan. Electronic address: masupulm@tmd.ac.jp.

Yoshihisa Nukui (Y)

Department of Respiratory Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo, 113-8519, Japan. Electronic address: nukui.pulm@gmail.com.

Tomoya Tateishi (T)

Department of Respiratory Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo, 113-8519, Japan. Electronic address: tateishi.pulm@tmd.ac.jp.

Mitsuhiro Kishino (M)

Department of Diagnostic Radiology, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo, 113-8519, Japan. Electronic address: ksnmrad@tmd.ac.jp.

Ukihide Tateishi (U)

Department of Diagnostic Radiology, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo, 113-8519, Japan. Electronic address: ttisdrnm@tmd.ac.jp.

Kaori Morota (K)

Scientific Affairs, Diagnostics Division, Abbott Japan LLC, 3-5-27 Mita Minato-ku, Tokyo, 108-6305, Japan. Electronic address: kaori.morota@abbott.com.

Kazuyuki Ohbo (K)

Department of Histology and Cell Biology, Yokohama City University, 3-9 Fukuura, Kanazawa-ku, Yokohama, 236-0004, Japan. Electronic address: kohbo@yokohama-cu.ac.jp.

Yasunari Miyazaki (Y)

Department of Respiratory Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo, 113-8519, Japan. Electronic address: miyazaki.pilm@tmd.ac.jp.

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