Genomic and Functional Regulation of TRIB1 Contributes to Prostate Cancer Pathogenesis.
TRIB1
cMYC
mouse models
prostate cancer
Journal
Cancers
ISSN: 2072-6694
Titre abrégé: Cancers (Basel)
Pays: Switzerland
ID NLM: 101526829
Informations de publication
Date de publication:
11 Sep 2020
11 Sep 2020
Historique:
received:
07
08
2020
revised:
26
08
2020
accepted:
04
09
2020
entrez:
16
9
2020
pubmed:
17
9
2020
medline:
17
9
2020
Statut:
epublish
Résumé
Prostate cancer is the most frequent malignancy in European men and the second worldwide. One of the major oncogenic events in this disease includes amplification of the transcription factor cMYC. Amplification of this oncogene in chromosome 8q24 occurs concomitantly with the copy number increase in a subset of neighboring genes and regulatory elements, but their contribution to disease pathogenesis is poorly understood. Here we show that
Identifiants
pubmed: 32932846
pii: cancers12092593
doi: 10.3390/cancers12092593
pmc: PMC7565426
pii:
doi:
Types de publication
Journal Article
Langues
eng
Subventions
Organisme : European Research Council
ID : 819242
Pays : International
Organisme : H2020 Marie Skłodowska-Curie Actions
ID : 721532
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