MMP2 as an independent prognostic stratifier in oral cavity cancers.


Journal

Oncoimmunology
ISSN: 2162-4011
Titre abrégé: Oncoimmunology
Pays: United States
ID NLM: 101570526

Informations de publication

Date de publication:
13 05 2020
Historique:
entrez: 16 9 2020
pubmed: 17 9 2020
medline: 17 9 2020
Statut: epublish

Résumé

Around 25% of oral cavity squamous cell carcinoma (OCSCC) are not controlled by the standard of care, but there is currently no validated biomarker to identify those patients. Our objective was to determine a robust biomarker for severe OCSCC, using a biology-driven strategy. Tumor and juxtatumor secretome were analyzed in a prospective discovery cohort of 37 OCSCC treated by primary surgery. Independent biomarker validation was performed by RTqPCR in a retrospective cohort of 145 patients with similar clinical features. An 18-gene signature (18 G) predictive of the response to PD-1 blockade was evaluated in the same cohort. Among 29 deregulated molecules identified in a secretome analysis, including chemokines, cytokines, growth factors, and molecules related to tumor growth and tissue remodeling, only soluble MMP2 was a prognostic biomarker. In our validation cohort, high levels of High levels of MMP2 were an independent and validated prognostic biomarker, surpassing other molecules of a large panel of the tumor and immune-related processes, which may be used to select poor prognosis patients for intensified neoadjuvant or adjuvant regimens.

Sections du résumé

Background
Around 25% of oral cavity squamous cell carcinoma (OCSCC) are not controlled by the standard of care, but there is currently no validated biomarker to identify those patients. Our objective was to determine a robust biomarker for severe OCSCC, using a biology-driven strategy.
Patients and methods
Tumor and juxtatumor secretome were analyzed in a prospective discovery cohort of 37 OCSCC treated by primary surgery. Independent biomarker validation was performed by RTqPCR in a retrospective cohort of 145 patients with similar clinical features. An 18-gene signature (18 G) predictive of the response to PD-1 blockade was evaluated in the same cohort.
Results
Among 29 deregulated molecules identified in a secretome analysis, including chemokines, cytokines, growth factors, and molecules related to tumor growth and tissue remodeling, only soluble MMP2 was a prognostic biomarker. In our validation cohort, high levels of
Conclusion
High levels of MMP2 were an independent and validated prognostic biomarker, surpassing other molecules of a large panel of the tumor and immune-related processes, which may be used to select poor prognosis patients for intensified neoadjuvant or adjuvant regimens.

Identifiants

pubmed: 32934875
doi: 10.1080/2162402X.2020.1754094
pii: 1754094
pmc: PMC7466851
doi:

Substances chimiques

B7 Antigens 0
CD276 protein, human 0
FUT4 protein, human EC 2.4.1.-
Fucosyltransferases EC 2.4.1.-
MMP2 protein, human EC 3.4.24.24
Matrix Metalloproteinase 2 EC 3.4.24.24

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Pagination

1754094

Informations de copyright

© 2020 The Author(s). Published with license by Taylor & Francis Group, LLC.

Déclaration de conflit d'intérêts

The authors declare no potential conflicts of interest.

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Auteurs

Caroline Hoffmann (C)

Paris Sciences and Letters (PSL) University, Paris, France.
INSERM U932 Research Unit, Immunity and Cancer, Paris, France.
Department of Surgical Oncology, Institut Curie, Paris & Saint-Cloud, France.

Sophie Vacher (S)

Paris Sciences and Letters (PSL) University, Paris, France.
Department of Genetics, Institut Curie, Paris, France.

Philémon Sirven (P)

Paris Sciences and Letters (PSL) University, Paris, France.
INSERM U932 Research Unit, Immunity and Cancer, Paris, France.

Charlotte Lecerf (C)

Paris Sciences and Letters (PSL) University, Paris, France.
Department of Drug Development and Innovation (D3i), Institut Curie, Paris & Saint-Cloud, France.

Lucile Massenet (L)

Paris Sciences and Letters (PSL) University, Paris, France.
INSERM U932 Research Unit, Immunity and Cancer, Paris, France.

Aurélie Moreira (A)

Paris Sciences and Letters (PSL) University, Paris, France.
Department of Drug Development and Innovation (D3i), Institut Curie, Paris & Saint-Cloud, France.

Aurore Surun (A)

SIREDO Cancer Center (Care, Innovation and Research in Pediatric, Adolescents and Young Adults Oncology), Institut Curie, Paris, France.
Paris Descartes University, Paris, France.

Anne Schnitzler (A)

Paris Sciences and Letters (PSL) University, Paris, France.
Department of Genetics, Institut Curie, Paris, France.

Jerzy Klijanienko (J)

Paris Sciences and Letters (PSL) University, Paris, France.
Department of Pathology, Institut Curie, Paris, France.

Odette Mariani (O)

Paris Sciences and Letters (PSL) University, Paris, France.
Department of Pathology, Institut Curie, Paris, France.
Biological Resources Center, Institut Curie, Paris, France.

Emmanuelle Jeannot (E)

Paris Sciences and Letters (PSL) University, Paris, France.
Department of Pathology, Institut Curie, Paris, France.

Nathalie Badois (N)

Paris Sciences and Letters (PSL) University, Paris, France.
Department of Surgical Oncology, Institut Curie, Paris & Saint-Cloud, France.

Maria Lesnik (M)

Paris Sciences and Letters (PSL) University, Paris, France.
Department of Surgical Oncology, Institut Curie, Paris & Saint-Cloud, France.

Olivier Choussy (O)

Paris Sciences and Letters (PSL) University, Paris, France.
Department of Surgical Oncology, Institut Curie, Paris & Saint-Cloud, France.

Christophe Le Tourneau (C)

Paris Sciences and Letters (PSL) University, Paris, France.
Department of Drug Development and Innovation (D3i), Institut Curie, Paris & Saint-Cloud, France.
INSERM U900 Research Unit, Saint-Cloud, France.

Maude Guillot-Delost (M)

Paris Sciences and Letters (PSL) University, Paris, France.
INSERM U932 Research Unit, Immunity and Cancer, Paris, France.
Center of Clinical Investigation, CIC IGR-Curie, Paris, France.

Maud Kamal (M)

Paris Sciences and Letters (PSL) University, Paris, France.
Department of Drug Development and Innovation (D3i), Institut Curie, Paris & Saint-Cloud, France.

Ivan Bieche (I)

Paris Sciences and Letters (PSL) University, Paris, France.
Department of Genetics, Institut Curie, Paris, France.
Faculty of Pharmaceutical and Biological Sciences, INSERM U1016 Research Unit, Paris Descartes University, Paris, France.

Vassili Soumelis (V)

Paris Sciences and Letters (PSL) University, Paris, France.
INSERM U932 Research Unit, Immunity and Cancer, Paris, France.
Clinical Immunology Department, Institut Curie, Paris, France.

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