Antibiotic use and risk of colorectal cancer: a systematic review and dose-response meta-analysis.


Journal

British journal of cancer
ISSN: 1532-1827
Titre abrégé: Br J Cancer
Pays: England
ID NLM: 0370635

Informations de publication

Date de publication:
12 2020
Historique:
received: 04 04 2020
accepted: 02 09 2020
revised: 24 08 2020
pubmed: 25 9 2020
medline: 7 4 2021
entrez: 24 9 2020
Statut: ppublish

Résumé

It is understudied whether the posed association of oral antibiotics with colorectal cancer (CRC) varies between antibiotic spectrums, colorectal continuum, and if a non-linear dose-dependent relationship is present. Three electronic databases and a trial platform were searched for all relevant studies, from inception until February 2020, without restrictions. Random-effects meta-analyses provided pooled effect-sizes (ES) with 95% confidence intervals (CI). Dose-response analyses modelling the relationship between number of days exposed to antibiotics and CRC risk were extended to non-linear multivariable random-effects models. Of 6483 identified publications ten were eligible, including 4.1 million individuals and over 73,550 CRC cases. The pooled CRC risk was increased among individuals who ever-used antibiotics (ES = 1.17, 95%CI 1.05-1.30), particularly for broad-spectrum antibiotics (ES = 1.70, 95%CI 1.26-2.30), but not for narrow-spectrum antibiotic (ES = 1.11, 95% 0.93-1.32). The dose-response analysis did not provide strong evidence of any particular dose-response association, and the risk patterns were rather similar for colon and rectal cancer. The antibiotic use associated CRC risk seemingly differs between broad- and narrow-spectrum antibiotics, and possibly within the colorectal continuum. It remains unclear whether this association is causal, requiring more mechanistic studies and further clarification of drug-microbiome interactions.

Sections du résumé

BACKGROUND
It is understudied whether the posed association of oral antibiotics with colorectal cancer (CRC) varies between antibiotic spectrums, colorectal continuum, and if a non-linear dose-dependent relationship is present.
DESIGN
Three electronic databases and a trial platform were searched for all relevant studies, from inception until February 2020, without restrictions. Random-effects meta-analyses provided pooled effect-sizes (ES) with 95% confidence intervals (CI). Dose-response analyses modelling the relationship between number of days exposed to antibiotics and CRC risk were extended to non-linear multivariable random-effects models.
RESULTS
Of 6483 identified publications ten were eligible, including 4.1 million individuals and over 73,550 CRC cases. The pooled CRC risk was increased among individuals who ever-used antibiotics (ES = 1.17, 95%CI 1.05-1.30), particularly for broad-spectrum antibiotics (ES = 1.70, 95%CI 1.26-2.30), but not for narrow-spectrum antibiotic (ES = 1.11, 95% 0.93-1.32). The dose-response analysis did not provide strong evidence of any particular dose-response association, and the risk patterns were rather similar for colon and rectal cancer.
DISCUSSION
The antibiotic use associated CRC risk seemingly differs between broad- and narrow-spectrum antibiotics, and possibly within the colorectal continuum. It remains unclear whether this association is causal, requiring more mechanistic studies and further clarification of drug-microbiome interactions.

Identifiants

pubmed: 32968205
doi: 10.1038/s41416-020-01082-2
pii: 10.1038/s41416-020-01082-2
pmc: PMC7722751
doi:

Substances chimiques

Anti-Bacterial Agents 0

Types de publication

Journal Article Meta-Analysis Systematic Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

1825-1832

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Auteurs

Johanna Simin (J)

Centre for Translational Microbiome Research (CTMR), Department of Microbiology, Tumor and Cell Biology, Karolinska Institutet, Biomedicum kvarter 8A. Solnavägen 9, SE-171 65, Stockholm, Sweden. Johanna.simin@ki.se.
Science for Life Laboratory (SciLifeLab), SE-171 21, Stockholm, Sweden. Johanna.simin@ki.se.

Romina Fornes (R)

Centre for Translational Microbiome Research (CTMR), Department of Microbiology, Tumor and Cell Biology, Karolinska Institutet, Biomedicum kvarter 8A. Solnavägen 9, SE-171 65, Stockholm, Sweden.
Science for Life Laboratory (SciLifeLab), SE-171 21, Stockholm, Sweden.

Qing Liu (Q)

Centre for Translational Microbiome Research (CTMR), Department of Microbiology, Tumor and Cell Biology, Karolinska Institutet, Biomedicum kvarter 8A. Solnavägen 9, SE-171 65, Stockholm, Sweden.
Science for Life Laboratory (SciLifeLab), SE-171 21, Stockholm, Sweden.

Renate Slind Olsen (RS)

Centre for Translational Microbiome Research (CTMR), Department of Microbiology, Tumor and Cell Biology, Karolinska Institutet, Biomedicum kvarter 8A. Solnavägen 9, SE-171 65, Stockholm, Sweden.
Department of Clinical Genetics, Karolinska University Hospital, SE-171 76, Stockholm, Sweden.

Steven Callens (S)

Department of Internal Medicine, Ghent University Hospital, Ghent, Belgium.

Lars Engstrand (L)

Centre for Translational Microbiome Research (CTMR), Department of Microbiology, Tumor and Cell Biology, Karolinska Institutet, Biomedicum kvarter 8A. Solnavägen 9, SE-171 65, Stockholm, Sweden.
Science for Life Laboratory (SciLifeLab), SE-171 21, Stockholm, Sweden.

Nele Brusselaers (N)

Centre for Translational Microbiome Research (CTMR), Department of Microbiology, Tumor and Cell Biology, Karolinska Institutet, Biomedicum kvarter 8A. Solnavägen 9, SE-171 65, Stockholm, Sweden.
Science for Life Laboratory (SciLifeLab), SE-171 21, Stockholm, Sweden.

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