Bimekizumab, a Novel Humanized IgG1 Antibody That Neutralizes Both IL-17A and IL-17F.


Journal

Frontiers in immunology
ISSN: 1664-3224
Titre abrégé: Front Immunol
Pays: Switzerland
ID NLM: 101560960

Informations de publication

Date de publication:
2020
Historique:
received: 13 12 2019
accepted: 14 07 2020
entrez: 25 9 2020
pubmed: 26 9 2020
medline: 20 4 2021
Statut: epublish

Résumé

Interleukin (IL)-17A is a key driver of inflammation and the principal target of anti-IL-17 therapeutic monoclonal antibodies. IL-17A, and its structurally similar family member IL-17F, have been shown to be functionally dysregulated in certain human immune-mediated inflammatory diseases such as psoriasis, psoriatic arthritis, and axial spondyloarthritis. Given the overlapping biology of these two cytokines, we postulated that dual neutralization of IL-17A and IL-17F may provide a greater depth of clinical response in IL-17-mediated diseases than IL-17A inhibition alone. We identified 496.g1, a humanized antibody with strong affinity for IL-17A but poor affinity for IL-17F. Affinity maturation of 496.g1 to 496.g3 greatly enhanced the affinity of the Fab fragment for IL-17F while retaining strong binding to IL-17A. As an IgG1, the affinity for IL-17A and IL-17F was 3.2 pM and 23 pM, respectively. Comparison of 496.g3 IgG1 with the commercially available anti-IL-17A monoclonal antibodies ixekizumab and secukinumab, by surface plasmon resonance and in a human

Identifiants

pubmed: 32973785
doi: 10.3389/fimmu.2020.01894
pmc: PMC7473305
doi:

Substances chimiques

Anti-Inflammatory Agents 0
Antibodies, Monoclonal, Humanized 0
Antibodies, Neutralizing 0
IL17A protein, human 0
IL17F protein, human 0
Interleukin-17 0
bimekizumab 09495UIM6V
ixekizumab BTY153760O
secukinumab DLG4EML025

Types de publication

Comparative Study Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1894

Informations de copyright

Copyright © 2020 Adams, Maroof, Baker, Lawson, Oliver, Paveley, Rapecki, Shaw, Vajjah, West and Griffiths.

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Auteurs

Ralph Adams (R)

Discovery Science, New Modality Therapeutics, UCB Pharma, Slough, United Kingdom.

Asher Maroof (A)

Immuno-Bone Therapeutic Area, Immunology Research, UCB Pharma, Slough, United Kingdom.

Terry Baker (T)

Discovery Science, New Modality Therapeutics, UCB Pharma, Slough, United Kingdom.

Alastair D G Lawson (ADG)

Research Fellow, UCB Pharma, Slough, United Kingdom.

Ruth Oliver (R)

Development Science, QP/DMPK, UCB Pharma, Slough, United Kingdom.

Ross Paveley (R)

Immuno-Bone Therapeutic Area, Immuno-Bone Discovery, UCB Pharma, Slough, United Kingdom.

Steve Rapecki (S)

Discovery Science, New Modality Therapeutics, UCB Pharma, Slough, United Kingdom.

Stevan Shaw (S)

Immuno-Bone Therapeutic Area, Immunology Research, UCB Pharma, Slough, United Kingdom.

Pavan Vajjah (P)

Development Science, QP/DMPK, UCB Pharma, Slough, United Kingdom.

Shauna West (S)

Immuno-Bone Therapeutic Area, Immuno-Bone Discovery, UCB Pharma, Slough, United Kingdom.

Meryn Griffiths (M)

Translational Medicine, TM Immuno-Bone, UCB Pharma, Slough, United Kingdom.

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Classifications MeSH