Functional interactions of adrenodoxin with several human mitochondrial cytochrome P450 enzymes.


Journal

Archives of biochemistry and biophysics
ISSN: 1096-0384
Titre abrégé: Arch Biochem Biophys
Pays: United States
ID NLM: 0372430

Informations de publication

Date de publication:
15 11 2020
Historique:
received: 06 07 2020
revised: 11 09 2020
accepted: 12 09 2020
pubmed: 28 9 2020
medline: 16 12 2020
entrez: 27 9 2020
Statut: ppublish

Résumé

Seven of the 57 human cytochrome P450 (P450) enzymes are mitochondrial and carry out important reactions with steroids and vitamins A and D. These seven P450s utilize an electron transport chain that includes NADPH, NADPH-adrenodoxin reductase (AdR), and adrenodoxin (Adx) instead of the diflavin NADPH-P450 reductase (POR) used by the other P450s in the endoplasmic reticulum. Although numerous studies have been published involving mitochondrial P450 systems, the experimental conditions vary considerably. We compared human Adx and bovine Adx, a commonly used component, and found very similar catalytic activities in reactions catalyzed by human P450s 11B2, 27A1, and 27C1. Binding constants of 6-200 nM were estimated for Adx binding to these P450s using microscale thermophoresis. All P450 catalytic reactions were saturated at 10 μM Adx, and higher concentrations were not inhibitory up to at least 50 μM. Collectively these studies demonstrate the tight binding of Adx (both human and bovine) to AdR and to several mitochondrial P450s and provide guidance for optimization of Adx-dependent P450 reactions.

Identifiants

pubmed: 32980349
pii: S0003-9861(20)30605-6
doi: 10.1016/j.abb.2020.108596
pmc: PMC7863566
mid: NIHMS1665187
pii:
doi:

Substances chimiques

Mitochondrial Proteins 0
Adrenodoxin 12687-22-8
Cytochrome P-450 Enzyme System 9035-51-2
Ferredoxin-NADP Reductase EC 1.18.1.2

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

108596

Subventions

Organisme : NIAMS NIH HHS
ID : F31 AR077386
Pays : United States
Organisme : NIGMS NIH HHS
ID : R01 GM118122
Pays : United States
Organisme : NIEHS NIH HHS
ID : T32 ES007028
Pays : United States

Informations de copyright

Copyright © 2020 Elsevier Inc. All rights reserved.

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Auteurs

Stella A Child (SA)

The Department of Biochemistry, Vanderbilt University School of Medicine, Nashville, TN, 37232-0146, United States.

Michael J Reddish (MJ)

The Department of Biochemistry, Vanderbilt University School of Medicine, Nashville, TN, 37232-0146, United States.

Sarah M Glass (SM)

The Department of Biochemistry, Vanderbilt University School of Medicine, Nashville, TN, 37232-0146, United States.

Margo H Goldfarb (MH)

The Department of Biochemistry, Vanderbilt University School of Medicine, Nashville, TN, 37232-0146, United States.

Ian R Barckhausen (IR)

The Department of Biochemistry, Vanderbilt University School of Medicine, Nashville, TN, 37232-0146, United States.

F Peter Guengerich (FP)

The Department of Biochemistry, Vanderbilt University School of Medicine, Nashville, TN, 37232-0146, United States. Electronic address: f.guengerich@vanderbilt.edu.

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Classifications MeSH