Association Between Thrombus Perviousness Assessed on Computed Tomography and Stroke Cause.


Journal

Stroke
ISSN: 1524-4628
Titre abrégé: Stroke
Pays: United States
ID NLM: 0235266

Informations de publication

Date de publication:
12 2020
Historique:
pubmed: 2 10 2020
medline: 18 2 2021
entrez: 1 10 2020
Statut: ppublish

Résumé

A recent study proposed that thrombus perviousness (TP)-the degree to which contrast agents penetrate the thrombus in an occluded vessel measured on noncontrast computed tomography (NCCT) and CT angiography-may be associated with cardioembolic stroke cause with high specificity. Our aim was to investigate which clinical and laboratory parameters affect measures of TP and to validate its diagnostic accuracy in an independent cohort of patients with acute ischemic stroke. Seventy-five patients from a prospectively maintained database with proximal occlusions of the middle cerebral artery (M1) were retrospectively analyzed. Thrombi were segmented on coregistered noncontrast computed tomography and CT angiography to determine the thrombus attenuation increase and void fraction (attenuation increase relative to contralateral side). TP measures were significantly higher in patients with cardioembolic stroke compared to patients with stroke attributed to large artery atherosclerosis (median thrombus attenuation increase [interquartile range], 2.79 [-3.54 to 8.85] versus -5.11 [-11.23 to -1.47]; The assessment of TP on baseline noncontrast computed tomography/CT angiography in patients with M1 occlusion may aid in determining cardioembolic stroke cause and guide secondary prevention. Selected clinical and laboratory parameters other than stroke cause did not affect TP measures.

Sections du résumé

BACKGROUND AND PURPOSE
A recent study proposed that thrombus perviousness (TP)-the degree to which contrast agents penetrate the thrombus in an occluded vessel measured on noncontrast computed tomography (NCCT) and CT angiography-may be associated with cardioembolic stroke cause with high specificity. Our aim was to investigate which clinical and laboratory parameters affect measures of TP and to validate its diagnostic accuracy in an independent cohort of patients with acute ischemic stroke.
METHODS
Seventy-five patients from a prospectively maintained database with proximal occlusions of the middle cerebral artery (M1) were retrospectively analyzed. Thrombi were segmented on coregistered noncontrast computed tomography and CT angiography to determine the thrombus attenuation increase and void fraction (attenuation increase relative to contralateral side).
RESULTS
TP measures were significantly higher in patients with cardioembolic stroke compared to patients with stroke attributed to large artery atherosclerosis (median thrombus attenuation increase [interquartile range], 2.79 [-3.54 to 8.85] versus -5.11 [-11.23 to -1.47];
CONCLUSIONS
The assessment of TP on baseline noncontrast computed tomography/CT angiography in patients with M1 occlusion may aid in determining cardioembolic stroke cause and guide secondary prevention. Selected clinical and laboratory parameters other than stroke cause did not affect TP measures.

Identifiants

pubmed: 32998650
doi: 10.1161/STROKEAHA.120.031148
doi:

Substances chimiques

Contrast Media 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3613-3622

Auteurs

Anna Kufner (A)

Klinik und Hochschulambulanz für Neurologie (A.K., H.E., M.E., C.H.N., L.S.).
Center for Stroke Research Berlin (CSB), Charité - Universitätsmedizin Berlin, Germany (A.K., H.E., M.E., C.H.N., L.S.).
Berlin Institute of Health (BIH), Germany (A.K., M.E., C.H.N., L.S.).

Hebun Erdur (H)

Klinik und Hochschulambulanz für Neurologie (A.K., H.E., M.E., C.H.N., L.S.).
Center for Stroke Research Berlin (CSB), Charité - Universitätsmedizin Berlin, Germany (A.K., H.E., M.E., C.H.N., L.S.).

Matthias Endres (M)

Klinik und Hochschulambulanz für Neurologie (A.K., H.E., M.E., C.H.N., L.S.).
Center for Stroke Research Berlin (CSB), Charité - Universitätsmedizin Berlin, Germany (A.K., H.E., M.E., C.H.N., L.S.).
Berlin Institute of Health (BIH), Germany (A.K., M.E., C.H.N., L.S.).
DZHK (German Center for Cardiovascular Research) (M.E., C.H.N.), Partner Site Berlin.
DZNE (German Center for Neurodegenerative Diseases) (M.E., C.H.N.), Partner Site Berlin.

Christian H Nolte (CH)

Klinik und Hochschulambulanz für Neurologie (A.K., H.E., M.E., C.H.N., L.S.).
Center for Stroke Research Berlin (CSB), Charité - Universitätsmedizin Berlin, Germany (A.K., H.E., M.E., C.H.N., L.S.).
Berlin Institute of Health (BIH), Germany (A.K., M.E., C.H.N., L.S.).
DZHK (German Center for Cardiovascular Research) (M.E., C.H.N.), Partner Site Berlin.
DZNE (German Center for Neurodegenerative Diseases) (M.E., C.H.N.), Partner Site Berlin.

Michael Scheel (M)

Department of Neuroradiology (M.S.), Charité - Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health (BIH), Germany.

Ludwig Schlemm (L)

Klinik und Hochschulambulanz für Neurologie (A.K., H.E., M.E., C.H.N., L.S.).
Center for Stroke Research Berlin (CSB), Charité - Universitätsmedizin Berlin, Germany (A.K., H.E., M.E., C.H.N., L.S.).
Berlin Institute of Health (BIH), Germany (A.K., M.E., C.H.N., L.S.).

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