Periodontal treatment and vascular inflammation in patients with advanced peripheral arterial disease: A randomized controlled trial.


Journal

Atherosclerosis
ISSN: 1879-1484
Titre abrégé: Atherosclerosis
Pays: Ireland
ID NLM: 0242543

Informations de publication

Date de publication:
11 2020
Historique:
received: 17 06 2020
revised: 16 09 2020
accepted: 18 09 2020
pubmed: 9 10 2020
medline: 24 6 2021
entrez: 8 10 2020
Statut: ppublish

Résumé

Observational studies support an association between periodontitis and cardiovascular diseases. The study objective was to assess vascular inflammation after periodontal treatment in patients with peripheral arterial disease. Ninety patients with peripheral arterial disease (PAD) and severe periodontitis were enrolled in a randomized, controlled trial. Thirty patients underwent non-surgical periodontal therapy and received additional systemic antibiotics (PT1 group), while 30 patients received the same therapy without antibiotics (PT2 group). The remaining thirty patients did not receive periodontal therapy (CG, control group). The primary outcome of this treatment was a reduction in vascular inflammation three months after periodontal treatment as determined by After three months of treatment, a significant improvement in periodontal health was observed in the treatment groups. However, no difference in the primary outcome in the aorta was observed in the three study groups (median target to background ratio follow-up/baseline, PT1 1.00; 95% CI 0.97-1.10, PT2 1.00; 95% CI 0.98-1.1, CG 1.1; 95% CI 0.99-1.1, p = 0.75). No significant differences were detected in most diseased segments and active segments. In addition, no differences were observed in Periodontal treatment was effective and safe but did not reduce vascular inflammation in patients with PAD.

Sections du résumé

BACKGROUND AND AIMS
Observational studies support an association between periodontitis and cardiovascular diseases. The study objective was to assess vascular inflammation after periodontal treatment in patients with peripheral arterial disease.
METHODS
Ninety patients with peripheral arterial disease (PAD) and severe periodontitis were enrolled in a randomized, controlled trial. Thirty patients underwent non-surgical periodontal therapy and received additional systemic antibiotics (PT1 group), while 30 patients received the same therapy without antibiotics (PT2 group). The remaining thirty patients did not receive periodontal therapy (CG, control group). The primary outcome of this treatment was a reduction in vascular inflammation three months after periodontal treatment as determined by
RESULTS
After three months of treatment, a significant improvement in periodontal health was observed in the treatment groups. However, no difference in the primary outcome in the aorta was observed in the three study groups (median target to background ratio follow-up/baseline, PT1 1.00; 95% CI 0.97-1.10, PT2 1.00; 95% CI 0.98-1.1, CG 1.1; 95% CI 0.99-1.1, p = 0.75). No significant differences were detected in most diseased segments and active segments. In addition, no differences were observed in
CONCLUSIONS
Periodontal treatment was effective and safe but did not reduce vascular inflammation in patients with PAD.

Identifiants

pubmed: 33032234
pii: S0021-9150(20)30538-4
doi: 10.1016/j.atherosclerosis.2020.09.019
pii:
doi:

Substances chimiques

Fluorodeoxyglucose F18 0Z5B2CJX4D

Types de publication

Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

60-69

Informations de copyright

Copyright © 2020 The Author(s). Published by Elsevier B.V. All rights reserved.

Auteurs

Gerald Seinost (G)

Division of Angiology, Department of Internal Medicine, Medical University of Graz, Austria. Electronic address: gerald.seinost@medunigraz.at.

Anja Horina (A)

Division of Angiology, Department of Internal Medicine, Medical University of Graz, Austria.

Behrouz Arefnia (B)

Division of Operative Dentistry, Periodontology and Prosthodontics, Department of Dental Medicine and Oral Health, Medical University of Graz, Austria.

Roman Kulnik (R)

Division of Nuclear Medicine Radiology, Department of Radiology, Medical University of Graz, Austria.

Stefan Kerschbaumer (S)

Division of Nuclear Medicine Radiology, Department of Radiology, Medical University of Graz, Austria.

Franz Quehenberger (F)

Institute for Medical Informatics, Statistics and Documentation, Medical University of Graz, Austria.

Viktoria Muster (V)

Division of Angiology, Department of Internal Medicine, Medical University of Graz, Austria.

Katharina Gütl (K)

Division of Angiology, Department of Internal Medicine, Medical University of Graz, Austria.

Sieglinde Zelzer (S)

Clinical Institute of Medical and Chemical Laboratory Diagnostics, Medical University of Graz, Austria.

Robert Gasser (R)

Division of Cardiology, Department of Internal Medicine, Medical University of Graz, Austria.

Harald Mangge (H)

Clinical Institute of Medical and Chemical Laboratory Diagnostics, Medical University of Graz, Austria.

Reingard Aigner (R)

Division of Nuclear Medicine Radiology, Department of Radiology, Medical University of Graz, Austria.

Marianne Brodmann (M)

Division of Angiology, Department of Internal Medicine, Medical University of Graz, Austria.

Gernot Wimmer (G)

Division of Operative Dentistry, Periodontology and Prosthodontics, Department of Dental Medicine and Oral Health, Medical University of Graz, Austria.

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