CX3CR1 Deficiency Attenuates DNFB-Induced Contact Hypersensitivity Through Skewed Polarization Towards M2 Phenotype in Macrophages.
Animals
Biomarkers
CX3C Chemokine Receptor 1
/ deficiency
Dermatitis, Contact
/ etiology
Dinitrofluorobenzene
/ pharmacology
Disease Models, Animal
Disease Susceptibility
Immunohistochemistry
Macrophage Activation
/ drug effects
Macrophages
/ drug effects
Mice
Mice, Knockout
Neutrophil Infiltration
/ immunology
CX3CR1
arginase-1
contact hypersensitivity
interleukin-6
macrophage
tumor necrosis factor-α
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
07 Oct 2020
07 Oct 2020
Historique:
received:
28
09
2020
accepted:
05
10
2020
entrez:
10
10
2020
pubmed:
11
10
2020
medline:
2
3
2021
Statut:
epublish
Résumé
CX3CL1 can function as both an adhesion molecule and a chemokine for CX3CR1
Identifiants
pubmed: 33036460
pii: ijms21197401
doi: 10.3390/ijms21197401
pmc: PMC7582565
pii:
doi:
Substances chimiques
Biomarkers
0
CX3C Chemokine Receptor 1
0
Dinitrofluorobenzene
D241E059U6
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : the Ministry of Education, Culture, Sports, Science and Technology in Japan
ID : 26893052
Organisme : the Ministry of Education, Culture, Sports, Science and Technology in Japan
ID : 16K19709
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