Incident Dementia in Elderly Patients with Nonalcoholic Fatty Liver Disease in Germany.


Journal

Digestive diseases and sciences
ISSN: 1573-2568
Titre abrégé: Dig Dis Sci
Pays: United States
ID NLM: 7902782

Informations de publication

Date de publication:
09 2021
Historique:
received: 01 07 2020
accepted: 28 09 2020
pubmed: 11 10 2020
medline: 30 9 2021
entrez: 10 10 2020
Statut: ppublish

Résumé

Dementia and NAFLD are two frequent conditions that share underlying risk factors mainly in the realm of metabolic disease. Additionally, an association between NAFLD and brain aging has been proposed. Therefore, we investigated the hypothesis if NAFLD is an independent risk factor for emerging dementia. In this population-based cohort study, elderly patients (≥ 65 years) with NAFLD diagnosed between 2000 and 2015 were matched 1:1 to a cohort without NAFLD based on ICD-10 coding in the Disease Analyzer database. Matching criteria were age, sex, physician, index year, and co-diagnoses associated with dementia. The primary outcomes of this study were all-cause dementia diagnoses, the incidence of vascular dementia, and antidementive drug prescription. A total of 22,317 patients with NAFLD were matched to 22,317 patients without NAFLD. Within 10 years of the index date, 16.0% of patients with NAFLD and 15.6% of the patients without NAFLD were diagnosed with dementia. On Cox regression analysis, there is no association between NAFLD and the incidence of all-cause dementia (HR 0.97, 95% CI 0.92-1.04), vascular dementia (HR 0.89, 95% CI 0.78-1.02), or the new prescription of antidementive therapy (HR 0.87, 95% CI 0.76-1.01). In sensitivity analyses, there was no association between NAFLD and dementia in different age-groups as well as men or women. In conclusion, in this database study of elderly patients coded with NAFLD no independent association with incident dementia was detected. Risk assessment regarding dementia in patients with NAFLD should be carried out in the same way as for metabolic burdened patients.

Identifiants

pubmed: 33037968
doi: 10.1007/s10620-020-06644-1
pii: 10.1007/s10620-020-06644-1
pmc: PMC8379104
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

3179-3185

Informations de copyright

© 2020. The Author(s).

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Auteurs

Christian Labenz (C)

Department of Internal Medicine I, University Medical Center of the Johannes Gutenberg-University, Mainz, Germany.
Cirrhosis Center Mainz (CCM), University Medical Center of the Johannes Gutenberg-University, Mainz, Germany.
Metabolic Liver Research Program, Department of Medicine I, University Medical Center of the Johannes Gutenberg-University, Langenbeckstrasse 1, 55131, Mainz, Germany.

Karel Kostev (K)

Epidemiology, IQVIA, Frankfurt am Main, Germany.

Leonard Kaps (L)

Department of Internal Medicine I, University Medical Center of the Johannes Gutenberg-University, Mainz, Germany.
Metabolic Liver Research Program, Department of Medicine I, University Medical Center of the Johannes Gutenberg-University, Langenbeckstrasse 1, 55131, Mainz, Germany.

Peter R Galle (PR)

Department of Internal Medicine I, University Medical Center of the Johannes Gutenberg-University, Mainz, Germany.
Cirrhosis Center Mainz (CCM), University Medical Center of the Johannes Gutenberg-University, Mainz, Germany.

Jörn M Schattenberg (JM)

Department of Internal Medicine I, University Medical Center of the Johannes Gutenberg-University, Mainz, Germany. joern.schattenberg@unimedizin-mainz.de.
Cirrhosis Center Mainz (CCM), University Medical Center of the Johannes Gutenberg-University, Mainz, Germany. joern.schattenberg@unimedizin-mainz.de.
Metabolic Liver Research Program, Department of Medicine I, University Medical Center of the Johannes Gutenberg-University, Langenbeckstrasse 1, 55131, Mainz, Germany. joern.schattenberg@unimedizin-mainz.de.

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