Immunopathological features of air pollution and its impact on inflammatory airway diseases (IAD).
AR, Allergic rhinitis
Air pollution
Antioxidant
COPD, Chronic obstructive pulmonary disease
CRS, Chronic rhinosinusitis
DEP, Diesel exhaust particles
IAD, Inflammatory airway diseases
IL, Interleukin
ILC, Innate lymphoid cells
Inflammatory airway disease
NOx, Nitrogen oxides
Oxidative stress biomarkers
PAH, Polycyclic aromatic hydrocarbons
PM, Particulate matter
ROS, Reactive oxygen species
TBS, Tobacco smoke
TLR, Toll-like receptors
Tobacco smoke
Treg, Regulatory T cell
VOCs, Volatile organic compounds
Journal
The World Allergy Organization journal
ISSN: 1939-4551
Titre abrégé: World Allergy Organ J
Pays: United States
ID NLM: 101481283
Informations de publication
Date de publication:
Oct 2020
Oct 2020
Historique:
received:
11
05
2020
revised:
31
08
2020
accepted:
11
09
2020
entrez:
12
10
2020
pubmed:
13
10
2020
medline:
13
10
2020
Statut:
ppublish
Résumé
Air pollution causes significant morbidity and mortality in patients with inflammatory airway diseases (IAD) such as allergic rhinitis (AR), chronic rhinosinusitis (CRS), asthma, and chronic obstructive pulmonary disease (COPD). Oxidative stress in patients with IAD can induce eosinophilic inflammation in the airways, augment atopic allergic sensitization, and increase susceptibility to infection. We reviewed emerging data depicting the involvement of oxidative stress in IAD patients. We evaluated biomarkers, outcome measures and immunopathological alterations across the airway mucosal barrier following exposure, particularly when accentuated by an infectious insult.
Identifiants
pubmed: 33042360
doi: 10.1016/j.waojou.2020.100467
pii: S1939-4551(20)30370-7
pmc: PMC7534666
doi:
Types de publication
Journal Article
Review
Langues
eng
Pagination
100467Subventions
Organisme : NIEHS NIH HHS
ID : P30 ES010126
Pays : United States
Informations de copyright
© 2020 The Authors.
Déclaration de conflit d'intérêts
The authors have nothing to declare relative to this paper.
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