Gastritis cystica profunda is associated with aberrant p53 and Epstein-Barr virus in gastric cancer: A clinicopathological, immunohistochemical and in situ hybridization study.


Journal

Pathology international
ISSN: 1440-1827
Titre abrégé: Pathol Int
Pays: Australia
ID NLM: 9431380

Informations de publication

Date de publication:
Jan 2021
Historique:
received: 03 08 2020
accepted: 02 10 2020
pubmed: 22 10 2020
medline: 15 10 2021
entrez: 21 10 2020
Statut: ppublish

Résumé

Gastritis cystica profunda (GCP) is a lesion characterized by cystic gastric glands within the submucosa. Some studies have reported that GCP is a precancerous lesion. Here, we investigated the association between GCP and gastric cancer. Gastric cancer specimens were taken from 1432 patients undergoing surgery or endoscopic submucosal resection and were classified as GCP or non-GCP. The clinicopathological features, immunohistochemistry and in situ hybridization expression of p53, Ki-67, KCNE2, Epstein-Barr virus (EBV) and programmed death ligand 1 (PD-L1) were compared between the two groups, as well as between GCPs and normal pyloric glands. One hundred and eighty patients (12.6%) had GCPs. In the GCP group, no cancerous lesions were found within the GCPs, but 13% were linked to GCPs and 60.2% were located above or near GCPs. Aberrant p53 expression, EBV-positive cancer cells and PD-L1 scores were significantly higher in the GCP group. The p53 score and Ki-67 labelling index were significantly higher and the KCNE2 score was significantly lower in GCPs than in pyloric glands. Although we suggest GCP is paracancerous, GCP has high proliferation activity and gastric cancer with GCP is associated with aberrant p53 and EBV. GCP is associated with aberrant p53 expression and EBV.

Identifiants

pubmed: 33084164
doi: 10.1111/pin.13039
doi:

Substances chimiques

B7-H1 Antigen 0
Tumor Suppressor Protein p53 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

42-50

Informations de copyright

© 2020 Japanese Society of Pathology and John Wiley & Sons Australia, Ltd.

Références

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Auteurs

Hiroe Itami (H)

Department of Diagnostic Pathology, Nara Medical University, Nara, Japan.

Kohei Morita (K)

Department of Diagnostic Pathology, Nara Medical University, Nara, Japan.

Tokiko Nakai (T)

Department of Diagnostic Pathology, Nara Medical University, Nara, Japan.

Tomoko Uchiyama (T)

Department of Diagnostic Pathology, Nara Medical University, Nara, Japan.

Sumire Sugimoto (S)

Department of Diagnostic Pathology, Nara Medical University, Nara, Japan.

Shoh Sasaki (S)

Department of Diagnostic Pathology, Nara Medical University, Nara, Japan.

Minami Matsuoka (M)

Department of Diagnostic Pathology, Nara Medical University, Nara, Japan.

Tomoya Myojin (T)

Department of Diagnostic Pathology, Nara Medical University, Nara, Japan.

Yuji Nitta (Y)

Department of Diagnostic Pathology, Nara Medical University, Nara, Japan.

Fumi Okabe (F)

Department of Diagnostic Pathology, Nara Medical University, Nara, Japan.

Tomomi Fujii (T)

Department of Diagnostic Pathology, Nara Medical University, Nara, Japan.

Kinta Hatakeyama (K)

Department of Diagnostic Pathology, Nara Medical University, Nara, Japan.

Akira Mitoro (A)

Department of Gastroenterology and Endocrinology, Nara Medical University, Nara, Japan.

Masayuki Sho (M)

Department of Surgery, Nara Medical University, Nara, Japan.

Chiho Ohbayashi (C)

Department of Diagnostic Pathology, Nara Medical University, Nara, Japan.

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