Parallels between retinal and brain pathology and response to immunotherapy in old, late-stage Alzheimer's disease mouse models.
astrocytes reactivation
glutamine synthetase
myeloid cells
neurodegenerative disease
ocular proteins
retina
synaptic preservation
vascular amyloidosis
Journal
Aging cell
ISSN: 1474-9726
Titre abrégé: Aging Cell
Pays: England
ID NLM: 101130839
Informations de publication
Date de publication:
14 Oct 2020
14 Oct 2020
Historique:
received:
13
05
2020
revised:
14
08
2020
accepted:
09
09
2020
entrez:
22
10
2020
pubmed:
23
10
2020
medline:
23
10
2020
Statut:
aheadofprint
Résumé
Despite growing evidence for the characteristic signs of Alzheimer's disease (AD) in the neurosensory retina, our understanding of retina-brain relationships, especially at advanced disease stages and in response to therapy, is lacking. In transgenic models of AD (APP
Identifiants
pubmed: 33090673
doi: 10.1111/acel.13246
pmc: PMC7681044
doi:
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
e13246Subventions
Organisme : The Saban Family Foundation
Organisme : The Maurice Marciano Family Foundation
Organisme : Tom Gordon Foundation
Organisme : Macquarie University
Organisme : NIA NIH HHS
ID : R01AG055865
Pays : United States
Organisme : Australian Government's National Collaborative Research Infrastructure Scheme
Organisme : NIA NIH HHS
ID : R01AG056478
Pays : United States
Organisme : National Health and Medical Research Council
Organisme : Petersen Foundation
Organisme : NIA NIH HHS
ID : R01 AG056478
Pays : United States
Organisme : NIA NIH HHS
ID : R01 AG055865
Pays : United States
Informations de copyright
© 2020 The Authors. Aging Cell published by the Anatomical Society and John Wiley & Sons Ltd.
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