Central sleep apnea and atrial fibrillation: A review on pathophysiological mechanisms and therapeutic implications.
Atrial fibrillation
Central sleep apnea
Pathophysiological mechanisms
Phrenic nerve stimulation
Therapeutic options
Journal
International journal of cardiology. Heart & vasculature
ISSN: 2352-9067
Titre abrégé: Int J Cardiol Heart Vasc
Pays: Ireland
ID NLM: 101649525
Informations de publication
Date de publication:
Oct 2020
Oct 2020
Historique:
received:
24
10
2019
revised:
20
04
2020
accepted:
23
04
2020
entrez:
26
10
2020
pubmed:
27
10
2020
medline:
27
10
2020
Statut:
epublish
Résumé
Precipitating factors and chronic diseases associated with atrial fibrillation (AF) are detailed in the literature. Emerging evidence over the last several decades suggests a potential causal relationship between central sleep apnea (CSA) and AF. Mechanisms including apnea-induced hypoxia with intermittent arousal, fluctuating levels of carbon dioxide, enhanced sympathetic/neurohormonal activation and oxidative stress causing inflammation have been implicated as etiologic causes of AF within this subpopulation. CSA affects the efficacy of pharmacologic and catheter-based antiarrhythmic treatments, which is why treating CSA prior to these interventions may lead to lower rates of AF. Subsequently, a reduction in the AF burden with transvenous phrenic nerve stimulation (TPNS) has become a topic of interest. The present review describes the relationship between these conditions, pathophysiologic mechanisms implicating the role of CSA in development of AF, and emerging therapeutic interventions.
Identifiants
pubmed: 33102683
doi: 10.1016/j.ijcha.2020.100527
pii: S2352-9067(19)30277-5
pmc: PMC7573647
doi:
Types de publication
Journal Article
Review
Langues
eng
Pagination
100527Commentaires et corrections
Type : CommentIn
Informations de copyright
© 2020 The Authors.
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