Improvement of autophagy dysfunction as a potential mechanism for environmental enrichment to protect blood-brain barrier in rats with vascular cognitive impairment.


Journal

Neuroscience letters
ISSN: 1872-7972
Titre abrégé: Neurosci Lett
Pays: Ireland
ID NLM: 7600130

Informations de publication

Date de publication:
20 11 2020
Historique:
received: 06 10 2020
accepted: 15 10 2020
pubmed: 3 11 2020
medline: 1 5 2021
entrez: 2 11 2020
Statut: ppublish

Résumé

Vascular cognitive impairment (VCI) is the second most common cause of dementia after Alzheimer's disease, and the cognitive impairment is one of the common effects of VCI. Unfortunately, it lacks effective therapeutic treatments at present. In our previous study, environmental enrichment (EE), as an early intervention for lifestyle modification, can ameliorate cognitive impairment by attenuating hippocampal blood-brain barrier (BBB) injury in chronic cerebral hypoperfusion (CCH) rats. However, the underlying mechanism remains unclear. Here, we found CCH rats in the standard environment (SE) developed cognitive impairment and BBB damage, which were significantly alleviated with the EE intervention. Meantime, EE improved the autophagy dysfunction caused by CCH in the hippocampus of rats, suggesting that the effect of EE on cognitive function and BBB may be related to the improvement of autophagy pathway.

Identifiants

pubmed: 33132180
pii: S0304-3940(20)30707-2
doi: 10.1016/j.neulet.2020.135437
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

135437

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Auteurs

Linling Xu (L)

Department of Neurology, Zhongnan Hospital, Wuhan University, No. 169, Donghu Road, Wuhan 430071, Hubei, China.

Changhua Qu (C)

Department of Neurology, Zhongnan Hospital, Wuhan University, No. 169, Donghu Road, Wuhan 430071, Hubei, China.

Chujie Qu (C)

Department of Neurology, Zhongnan Hospital, Wuhan University, No. 169, Donghu Road, Wuhan 430071, Hubei, China.

Jun Shen (J)

Department of Neurology, Zhongnan Hospital, Wuhan University, No. 169, Donghu Road, Wuhan 430071, Hubei, China.

Hao Song (H)

Department of Neurology, Zhongnan Hospital, Wuhan University, No. 169, Donghu Road, Wuhan 430071, Hubei, China.

Yaqing Li (Y)

Department of Neurology, Zhongnan Hospital, Wuhan University, No. 169, Donghu Road, Wuhan 430071, Hubei, China.

Tian Li (T)

Department of Neurology, Zhongnan Hospital, Wuhan University, No. 169, Donghu Road, Wuhan 430071, Hubei, China.

Jiaxin Zheng (J)

Department of Neurology, Zhongnan Hospital, Wuhan University, No. 169, Donghu Road, Wuhan 430071, Hubei, China.

Junjian Zhang (J)

Department of Neurology, Zhongnan Hospital, Wuhan University, No. 169, Donghu Road, Wuhan 430071, Hubei, China. Electronic address: zhangjj@whu.edu.cn.

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Classifications MeSH