DNA Methylation Age Is More Closely Associated With Infection Risk Than Chronological Age in Kidney Transplant Recipients.


Journal

Transplantation direct
ISSN: 2373-8731
Titre abrégé: Transplant Direct
Pays: United States
ID NLM: 101651609

Informations de publication

Date de publication:
Aug 2020
Historique:
received: 17 02 2020
revised: 03 04 2020
accepted: 07 04 2020
entrez: 2 11 2020
pubmed: 3 11 2020
medline: 3 11 2020
Statut: epublish

Résumé

Older kidney transplant recipients demonstrate increased rates of infection but decreased rates of rejection compared with younger recipients, suggesting that older transplant patients are functionally overimmunosuppressed. We hypothesized that this is a consequence of reduction in immunological activity due to biological aging and that an immune biological age, as determined by DNA methylation (DNAm), would be associated more strongly with incidence of infection than chronological age. DNAm analysis was performed on peripheral blood mononuclear cell collected from 60 kidney transplant recipients representing older (≥age 60 y) and younger (aged 30-59 y) patients 3 months after transplantation. DNAm age was calculated based on methylation status of a panel of CpG sites, which have been previously identified as indicative of biological age. Correlation was seen between chronological and DNAm age; however, there were many patients with significant differences (either acceleration or slowing) between DNAm age and chronological age. A statistically significant association was seen between increased DNAm age and incidence of infection in the first year after kidney transplantation, whereas no significant association was seen between chronological age and infection. Assessment of DNAm age holds promise as an approach for patient evaluation and individualization of immune suppression regimens. This analysis may provide insights into the immunological mechanism behind increased incidence of infection observed in older transplant patients. The ability to measure biological age would allow for patient risk stratification and individualization of immunosuppression, improving outcomes for the growing numbers of older patients undergoing kidney transplantation.

Identifiants

pubmed: 33134500
doi: 10.1097/TXD.0000000000001020
pmc: PMC7581059
doi:

Types de publication

Journal Article

Langues

eng

Pagination

e576

Subventions

Organisme : NIA NIH HHS
ID : R03 AG050946
Pays : United States
Organisme : NIA NIH HHS
ID : R21 AG055879
Pays : United States
Organisme : NIAID NIH HHS
ID : U19 AI128913
Pays : United States
Organisme : NCATS NIH HHS
ID : UL1 TR001881
Pays : United States

Informations de copyright

Copyright © 2020 The Author(s). Transplantation Direct. Published by Wolters Kluwer Health, Inc.

Déclaration de conflit d'intérêts

The authors declare no conflicts of interest.

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Auteurs

Joanna Schaenman (J)

Department of Medicine, Division of Infectious Diseases, David Geffen School of Medicine at UCLA, Los Angeles, CA.

Xinkai Zhou (X)

Division of General Internal Medicine and Health Services Research, David Geffen School of Medicine at UCLA, Los Angeles, CA.

Rong Guo (R)

Division of General Internal Medicine and Health Services Research, David Geffen School of Medicine at UCLA, Los Angeles, CA.

Maura Rossetti (M)

Department of Pathology and Laboratory Medicine, UCLA Immunogenetics Center, David Geffen School of Medicine at UCLA, Los Angeles, CA.

Emily C Liang (EC)

Department of Pathology and Laboratory Medicine, UCLA Immunogenetics Center, David Geffen School of Medicine at UCLA, Los Angeles, CA.

Erik Lum (E)

Department of Medicine, Division of Nephrology, David Geffen School of Medicine at UCLA, Los Angeles, CA.

Basmah Abdalla (B)

Department of Medicine, Division of Nephrology, David Geffen School of Medicine at UCLA, Los Angeles, CA.

Suphamai Bunnapradist (S)

Department of Medicine, Division of Nephrology, David Geffen School of Medicine at UCLA, Los Angeles, CA.

Phuong-Thu T Pham (PT)

Department of Medicine, Division of Nephrology, David Geffen School of Medicine at UCLA, Los Angeles, CA.

Gabriel Danovitch (G)

Department of Medicine, Division of Nephrology, David Geffen School of Medicine at UCLA, Los Angeles, CA.

Arun Karlamangla (A)

Department of Medicine, Division of Geriatrics, David Geffen School of Medicine at UCLA, Los Angeles, CA.

Elaine Reed (E)

Department of Pathology and Laboratory Medicine, UCLA Immunogenetics Center, David Geffen School of Medicine at UCLA, Los Angeles, CA.

Steve Horvath (S)

Department of Human Genetics, David Geffen School of Medicine at UCLA, Los Angeles, CA.

David Elashoff (D)

Division of General Internal Medicine and Health Services Research, David Geffen School of Medicine at UCLA, Los Angeles, CA.

Classifications MeSH