Measuring beta-galactose exposure on platelets: Standardization and healthy reference values.

N‐acetylneuraminic acid blood platelets galactose platelet count references values

Journal

Research and practice in thrombosis and haemostasis
ISSN: 2475-0379
Titre abrégé: Res Pract Thromb Haemost
Pays: United States
ID NLM: 101703775

Informations de publication

Date de publication:
Jul 2020
Historique:
received: 19 03 2020
revised: 28 04 2020
accepted: 30 04 2020
entrez: 2 11 2020
pubmed: 3 11 2020
medline: 3 11 2020
Statut: epublish

Résumé

Correct diagnosis of the cause of thrombocytopenia is crucial for the appropriate management of patients. Hyposialylation/desialylation (characterized by abnormally high β-galactose exposure) accelerates platelet clearance and can lead to thrombocytopenia. However, the reference range for β-galactose exposure in healthy individuals has not been defined previously. The objective of the present study was to develop a standardized assay of platelet β-galactose exposure for implementation in a clinical laboratory. β-Galactose exposure was measured in platelet-rich plasma by using flow cytometry and We determined an optimal RCA concentration of 12.5 μg/mL. The measure was stable for up to 4 hours (mean fluorescence intensity [MFI]-RCA: 1233 ± 329 at 0 hour and 1480 ± 410 at 4 hours). The platelet count did not induce a variation of RCA and the measure of RCA was stable when tested up to 24 hours after blood collection (MFI-RCA: 1252 ± 434 at day 0 and 1140 ± 297 24 hours after blood sampling). To take into account the platelet size, results should be expressed as RCA/forward scatter ratio. We used the assay to study variability in 120 healthy adults, and we found that the ratio is independent of sex and blood group. We defined a normal range in a healthy population and several preanalytical and analytical variables were evaluated, together with positive and negative controls. This assay may assist in the diagnosis of thrombocytopenic diseases linked to changes in β-galactose exposure.

Sections du résumé

BACKGROUND BACKGROUND
Correct diagnosis of the cause of thrombocytopenia is crucial for the appropriate management of patients. Hyposialylation/desialylation (characterized by abnormally high β-galactose exposure) accelerates platelet clearance and can lead to thrombocytopenia. However, the reference range for β-galactose exposure in healthy individuals has not been defined previously.
OBJECTIVE OBJECTIVE
The objective of the present study was to develop a standardized assay of platelet β-galactose exposure for implementation in a clinical laboratory.
METHODS METHODS
β-Galactose exposure was measured in platelet-rich plasma by using flow cytometry and
RESULTS RESULTS
We determined an optimal RCA concentration of 12.5 μg/mL. The measure was stable for up to 4 hours (mean fluorescence intensity [MFI]-RCA: 1233 ± 329 at 0 hour and 1480 ± 410 at 4 hours). The platelet count did not induce a variation of RCA and the measure of RCA was stable when tested up to 24 hours after blood collection (MFI-RCA: 1252 ± 434 at day 0 and 1140 ± 297 24 hours after blood sampling). To take into account the platelet size, results should be expressed as RCA/forward scatter ratio. We used the assay to study variability in 120 healthy adults, and we found that the ratio is independent of sex and blood group.
CONCLUSION CONCLUSIONS
We defined a normal range in a healthy population and several preanalytical and analytical variables were evaluated, together with positive and negative controls. This assay may assist in the diagnosis of thrombocytopenic diseases linked to changes in β-galactose exposure.

Identifiants

pubmed: 33134771
doi: 10.1002/rth2.12369
pii: S2475-0379(22)02039-8
pmc: PMC7586713
doi:

Types de publication

Journal Article

Langues

eng

Pagination

813-822

Informations de copyright

© 2020 The Authors. Research and Practice in Thrombosis and Haemostasis published by Wiley Periodicals LLC on behalf of International Society on Thrombosis and Haemostasis.

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Auteurs

Dominique Lasne (D)

Department of Biological Hematology Hôpital Necker AP-HP Paris France.
HITh UMR_S 1176 INSERM Univ. Paris-Saclay Le Kremlin-Bicêtre France.

Tiffany Pascreau (T)

Department of Biological Hematology Hôpital Necker AP-HP Paris France.
HITh UMR_S 1176 INSERM Univ. Paris-Saclay Le Kremlin-Bicêtre France.

Sadyo Darame (S)

Department of Biological Hematology Hôpital Necker AP-HP Paris France.

Marie-Charlotte Bourrienne (MC)

Department of Biological Hematology Hôpital Necker AP-HP Paris France.

Peggy Tournoux (P)

Department of Biological Hematology Hôpital Necker AP-HP Paris France.

Aurélien Philippe (A)

Department of Biological Hematology Hôpital Necker AP-HP Paris France.

Sara Ziachahabi (S)

Department of Biological Hematology Hôpital Necker AP-HP Paris France.

Felipe Suarez (F)

Department of Hematology Hôpital Necker AP-HP Paris France.

Ambroise Marcais (A)

Department of Hematology Hôpital Necker AP-HP Paris France.

Annabelle Dupont (A)

Department of Haemostasis and Transfusion CHU Lille Lille France.
Institut Pasteur de Lille U1011- EGID Inserm CHU Lille Univ. Lille Lille France.

Cécile V Denis (CV)

HITh UMR_S 1176 INSERM Univ. Paris-Saclay Le Kremlin-Bicêtre France.

Alexandre Kauskot (A)

HITh UMR_S 1176 INSERM Univ. Paris-Saclay Le Kremlin-Bicêtre France.

Delphine Borgel (D)

Department of Biological Hematology Hôpital Necker AP-HP Paris France.
HITh UMR_S 1176 INSERM Univ. Paris-Saclay Le Kremlin-Bicêtre France.

Classifications MeSH