Update of a colorimetric method for quantitative determination of galactose in blood samples: A simple and rapid method for the early detection of inherited metabolic diseases.


Journal

Carbohydrate research
ISSN: 1873-426X
Titre abrégé: Carbohydr Res
Pays: Netherlands
ID NLM: 0043535

Informations de publication

Date de publication:
Dec 2020
Historique:
received: 14 08 2020
revised: 08 10 2020
accepted: 13 10 2020
pubmed: 3 11 2020
medline: 24 8 2021
entrez: 2 11 2020
Statut: ppublish

Résumé

A colorimetric microassay for the quantitative determination of galactose in the blood was taken and updated. This method helps in diagnosis and follow-up of several inherited metabolic diseases connected to galactose metabolism deficiency such as galactosemia, glycogenosis, glycosylation, tyrosinemia and citrin deficiency. Galactose assay in the blood presents difficulties due to interference with glucose. In this study, we update a method to get around these difficulties. This procedure was based on the incubation of whole blood with orcinol in a strongly acidic solution to form a galactose and glucose complexes able to absorb at two different wavelengths. The standard curve analysis for the individual solutions of these two sugars showed a wide range of linearity from 0 to 200 mg / l. Under optimal experimental conditions, the stirring time of the orcinol is 3 minutes, the heating time of the reaction is 20 minutes at 56 ° C, and the duration of the incubation in the dark is 40 minutes. The analysis is carried out on fresh blood. The maximum absorbance of galactose and glucose is respectively 569 nm and 421 nm. An adapted diagnosis algorithm was developed based on our results. this method could help in screening and identifying patients with hypergalactosemia that need further investigations. It could represent a promising method for neonatal screening in countries with limited resources.

Sections du résumé

BACKGROUND BACKGROUND
A colorimetric microassay for the quantitative determination of galactose in the blood was taken and updated. This method helps in diagnosis and follow-up of several inherited metabolic diseases connected to galactose metabolism deficiency such as galactosemia, glycogenosis, glycosylation, tyrosinemia and citrin deficiency. Galactose assay in the blood presents difficulties due to interference with glucose. In this study, we update a method to get around these difficulties.
METHOD METHODS
This procedure was based on the incubation of whole blood with orcinol in a strongly acidic solution to form a galactose and glucose complexes able to absorb at two different wavelengths.
RESULTS RESULTS
The standard curve analysis for the individual solutions of these two sugars showed a wide range of linearity from 0 to 200 mg / l. Under optimal experimental conditions, the stirring time of the orcinol is 3 minutes, the heating time of the reaction is 20 minutes at 56 ° C, and the duration of the incubation in the dark is 40 minutes. The analysis is carried out on fresh blood. The maximum absorbance of galactose and glucose is respectively 569 nm and 421 nm. An adapted diagnosis algorithm was developed based on our results.
CONCLUSION CONCLUSIONS
this method could help in screening and identifying patients with hypergalactosemia that need further investigations. It could represent a promising method for neonatal screening in countries with limited resources.

Identifiants

pubmed: 33137585
pii: S0008-6215(20)30550-4
doi: 10.1016/j.carres.2020.108179
pii:
doi:

Substances chimiques

Galactose X2RN3Q8DNE

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

108179

Informations de copyright

Copyright © 2020. Published by Elsevier Ltd.

Auteurs

Karima Lafhal (K)

Metabolic Platform, Biochemistry Laboratory, Team for Childhood, Health and Development Faculty of Medicine, Cadi Ayad University, Marrakech, Morocco.

Es-Said Sabir (ES)

Metabolic Platform, Biochemistry Laboratory, Team for Childhood, Health and Development Faculty of Medicine, Cadi Ayad University, Marrakech, Morocco.

Mouna Cheggour (M)

Biochemistry Laboratory, Faculty of Medicine, Cadi Ayad University, Marrakesh, Morocco.

Fatimazahra Mouad (F)

Pediatric Department, Mohammed VI Hospital University, Marrakesh, Morocco.

Miloud Hammoud (M)

Metabolic Platform, Biochemistry Laboratory, Team for Childhood, Health and Development Faculty of Medicine, Cadi Ayad University, Marrakech, Morocco.

Abdessamad Lalaoui (A)

Pediatric Department, Mohammed VI Hospital University, Marrakesh, Morocco.

Aicha Ezoubeiri (A)

Clinical Laboratory, Ibn Tofail Hospital, Mohammed VI Hospital University, Marrakesh, Morocco.

Salwa Baki (S)

Endocrinology Department, Provincial Hospital of Taroudant, Morocco.

Laila Chabaa (L)

Biochemistry Laboratory, Faculty of Medicine, Cadi Ayad University, Marrakesh, Morocco.

Fadl Mrabih Rabou Maoulainine (FMR)

Neonatal Intensive Care Department, Team for Childhood, Health and Development, Marrakesh Faculty of Medicine, Mohammed VI University Hospital and Research, Cadi Ayad University, Marrakech, Morocco.

Noureddine Rada (N)

Pediatric Department, Mohammed VI Hospital University, Marrakesh, Morocco.

Mohammed Bouskraoui (M)

Pediatric Department, Mohammed VI Hospital University, Marrakesh, Morocco.

Imane Ait Saab (I)

Pediatric Department, Mohammed VI Hospital University, Marrakesh, Morocco.

Aicha Bourrahouat (A)

Pediatric Department, Mohammed VI Hospital University, Marrakesh, Morocco.

Naima Fdil (N)

Metabolic Platform, Biochemistry Laboratory, Team for Childhood, Health and Development Faculty of Medicine, Cadi Ayad University, Marrakech, Morocco. Electronic address: nfdil@yahoo.fr.

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Classifications MeSH