Natural History of Cirrhosis: Changing Trends in Etiology Over the Years.


Journal

Digestive diseases (Basel, Switzerland)
ISSN: 1421-9875
Titre abrégé: Dig Dis
Pays: Switzerland
ID NLM: 8701186

Informations de publication

Date de publication:
2021
Historique:
received: 21 08 2020
accepted: 02 11 2020
pubmed: 4 11 2020
medline: 17 7 2021
entrez: 3 11 2020
Statut: ppublish

Résumé

The aims of the present study were to investigate the natural history of cirrhosis and to determine trends in the etiology of cirrhosis. Between January 2001 and January 2018, a total of 1,341 patients had been diagnosed with cirrhosis and were included. A total of 898 cirrhotic patients, who were followed up for at least 6 months were included into the analysis. The median age was 54 years. The median Child-Pugh and MELD scores were 7.5 and 11, respectively. Ascites (51%) was the most common causes of decompensation. Chronic viral hepatitis was the most frequent cause of cirrhosis (58%). Hepatitis B virus (HBV) infection was the main etiology (34%), followed by hepatitis C virus (HCV) infection (18%). Among 129 patients with cryptogenic cirrhosis (CC), 60 had metabolic abnormalities. If these 60 patients with CC were considered to have nonalcoholic fatty liver disease (NAFLD)-related cirrhosis, the proportion of NAFLD-related cirrhosis increased from 1.8 to 8.0%. At admission, 74 patients (8%) had been diagnosed with hepatocellular carcinoma (HCC). A new HCC developed in 80 patients during the follow-up period. The probability of developing HCC was 3.9% at 12 months. Logistic regression analysis showed that the development of HCC was significantly associated with older age (p < 0.001), male gender (p < 0.001), viral etiology (p = 0.026), and baseline high aspartate aminotransferase level (p = 0.01). Overall, 104 cirrhotic patients died. HBV and HCV remain the leading causes of etiology in cirrhosis and HCC. However, NAFLD-related cirrhosis is recognized as a growing burden.

Sections du résumé

BACKGROUND AND GOALS OBJECTIVE
The aims of the present study were to investigate the natural history of cirrhosis and to determine trends in the etiology of cirrhosis.
METHODS METHODS
Between January 2001 and January 2018, a total of 1,341 patients had been diagnosed with cirrhosis and were included.
RESULTS RESULTS
A total of 898 cirrhotic patients, who were followed up for at least 6 months were included into the analysis. The median age was 54 years. The median Child-Pugh and MELD scores were 7.5 and 11, respectively. Ascites (51%) was the most common causes of decompensation. Chronic viral hepatitis was the most frequent cause of cirrhosis (58%). Hepatitis B virus (HBV) infection was the main etiology (34%), followed by hepatitis C virus (HCV) infection (18%). Among 129 patients with cryptogenic cirrhosis (CC), 60 had metabolic abnormalities. If these 60 patients with CC were considered to have nonalcoholic fatty liver disease (NAFLD)-related cirrhosis, the proportion of NAFLD-related cirrhosis increased from 1.8 to 8.0%. At admission, 74 patients (8%) had been diagnosed with hepatocellular carcinoma (HCC). A new HCC developed in 80 patients during the follow-up period. The probability of developing HCC was 3.9% at 12 months. Logistic regression analysis showed that the development of HCC was significantly associated with older age (p < 0.001), male gender (p < 0.001), viral etiology (p = 0.026), and baseline high aspartate aminotransferase level (p = 0.01). Overall, 104 cirrhotic patients died.
CONCLUSION CONCLUSIONS
HBV and HCV remain the leading causes of etiology in cirrhosis and HCC. However, NAFLD-related cirrhosis is recognized as a growing burden.

Identifiants

pubmed: 33142287
pii: 000512746
doi: 10.1159/000512746
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

358-365

Informations de copyright

© 2020 S. Karger AG, Basel.

Auteurs

Ramazan Idilman (R)

Department of Gastroenterology, Ankara University School of Medicine, Ankara, Turkey, idilman@medicine.ankara.edu.tr.

Merve Aydogan (M)

Department of Internal Medicine, Ankara University School of Medicine, Ankara, Turkey.

Mehmet Berk Oruncu (MB)

Department of Internal Medicine, Ankara University School of Medicine, Ankara, Turkey.

Aysun Kartal (A)

Department of Gastroenterology, Ankara University School of Medicine, Ankara, Turkey.

Atilla Halil Elhan (AH)

Department of Biostatistics, Ankara University School of Medicine, Ankara, Turkey.

Zeynep Ellik (Z)

Department of Gastroenterology, Ankara University School of Medicine, Ankara, Turkey.

Mesut Gumussoy (M)

Department of Gastroenterology, Ankara University School of Medicine, Ankara, Turkey.

Ramazan Er (R)

Department of Gastroenterology, Ankara University School of Medicine, Ankara, Turkey.

Mubin Ozercan (M)

Department of Gastroenterology, Ankara University School of Medicine, Ankara, Turkey.

Serkan Duman (S)

Department of Gastroenterology, Ankara University School of Medicine, Ankara, Turkey.

Murat Toruner (M)

Department of Gastroenterology, Ankara University School of Medicine, Ankara, Turkey.

Kubilay Cinar (K)

Department of Gastroenterology, Ankara University School of Medicine, Ankara, Turkey.

Irfan Soykan (I)

Department of Gastroenterology, Ankara University School of Medicine, Ankara, Turkey.

Ali Resit Beyler (AR)

Department of Gastroenterology, Ankara University School of Medicine, Ankara, Turkey.

Hasan Ozkan (H)

Department of Gastroenterology, Ankara University School of Medicine, Ankara, Turkey.

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Classifications MeSH