Evolution from adherent to suspension: systems biology of HEK293 cell line development.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
04 11 2020
Historique:
received: 11 02 2020
accepted: 22 10 2020
entrez: 5 11 2020
pubmed: 6 11 2020
medline: 11 3 2021
Statut: epublish

Résumé

The need for new safe and efficacious therapies has led to an increased focus on biologics produced in mammalian cells. The human cell line HEK293 has bio-synthetic potential for human-like production attributes and is currently used for manufacturing of several therapeutic proteins and viral vectors. Despite the increased popularity of this strain we still have limited knowledge on the genetic composition of its derivatives. Here we present a genomic, transcriptomic and metabolic gene analysis of six of the most widely used HEK293 cell lines. Changes in gene copy and expression between industrial progeny cell lines and the original HEK293 were associated with cellular component organization, cell motility and cell adhesion. Changes in gene expression between adherent and suspension derivatives highlighted switching in cholesterol biosynthesis and expression of five key genes (RARG, ID1, ZIC1, LOX and DHRS3), a pattern validated in 63 human adherent or suspension cell lines of other origin.

Identifiants

pubmed: 33149219
doi: 10.1038/s41598-020-76137-8
pii: 10.1038/s41598-020-76137-8
pmc: PMC7642379
doi:

Substances chimiques

Cholesterol 97C5T2UQ7J

Types de publication

Comparative Study Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

18996

Commentaires et corrections

Type : ErratumIn

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Auteurs

Magdalena Malm (M)

KTH - School of Engineering Sciences in Chemistry, Biotechnology, and Health, Dept. of Protein Science, Royal Institute of Technology, 106 91, Stockholm, Sweden.

Rasool Saghaleyni (R)

Department of Biology and Biological Engineering, Chalmers University of Technology, 412 96, Gothenburg, Sweden.

Magnus Lundqvist (M)

KTH - School of Engineering Sciences in Chemistry, Biotechnology, and Health, Dept. of Protein Science, Royal Institute of Technology, 106 91, Stockholm, Sweden.

Marco Giudici (M)

KTH - School of Engineering Sciences in Chemistry, Biotechnology, and Health, Dept. of Protein Science, Royal Institute of Technology, 106 91, Stockholm, Sweden.

Veronique Chotteau (V)

KTH - School of Engineering Sciences in Chemistry, Biotechnology, and Health, Dept. of Protein Science, Royal Institute of Technology, 106 91, Stockholm, Sweden.

Ray Field (R)

Biopharmaceutical Development, BioPharmaceuticals R&D, AstraZeneca, Milstein Building, Granta Park, Cambridge, CB21 6GH, UK.
GammaDelta Therapeutics Ltd, White City Place, London, W12 7FQ, UK.

Paul G Varley (PG)

Biopharmaceutical Development, BioPharmaceuticals R&D, AstraZeneca, Milstein Building, Granta Park, Cambridge, CB21 6GH, UK.
Kymab, Babraham Research Campus, Cambridge, CB22 3AT, UK.

Diane Hatton (D)

Biopharmaceutical Development, BioPharmaceuticals R&D, AstraZeneca, Milstein Building, Granta Park, Cambridge, CB21 6GH, UK.

Luigi Grassi (L)

Biopharmaceutical Development, BioPharmaceuticals R&D, AstraZeneca, Milstein Building, Granta Park, Cambridge, CB21 6GH, UK.

Thomas Svensson (T)

Department of Biology and Biological Engineering, Chalmers University of Technology, 412 96, Gothenburg, Sweden.
NBIS - Bioinformatics Systems Biology Support, Chalmers University of Technology, 412 96, Gothenburg, Sweden.

Jens Nielsen (J)

Department of Biology and Biological Engineering, Chalmers University of Technology, 412 96, Gothenburg, Sweden. nielsenj@chalmers.se.
Novo Nordisk Foundation Center for Biosustainability, Technical University of Denmark, 2800, Kongens Lyngby, Denmark. nielsenj@chalmers.se.

Johan Rockberg (J)

KTH - School of Engineering Sciences in Chemistry, Biotechnology, and Health, Dept. of Protein Science, Royal Institute of Technology, 106 91, Stockholm, Sweden. johan.rockberg@biotech.kth.se.

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