Body Mass Index and Tumor-Infiltrating Lymphocytes in Triple-Negative Breast Cancer.


Journal

Journal of the National Cancer Institute
ISSN: 1460-2105
Titre abrégé: J Natl Cancer Inst
Pays: United States
ID NLM: 7503089

Informations de publication

Date de publication:
01 02 2021
Historique:
received: 11 02 2020
revised: 15 04 2020
accepted: 17 06 2020
pubmed: 6 11 2020
medline: 22 6 2021
entrez: 5 11 2020
Statut: ppublish

Résumé

High levels of stromal tumor-infiltrating lymphocytes (sTIL) are associated with increased pathological complete response (pCR) rate and longer survival after neoadjuvant chemotherapy in triple-negative breast cancer (TNBC) patients. Here, we evaluated the value of sTIL in predicting pCR and explored prognosis in TNBC patients treated with neoadjuvant chemotherapy according to body mass index (BMI). sTIL were scored centrally on pretreatment biopsies from 2 retrospective series of nonunderweight TNBC patients (n = 445). sTIL and BMI were considered as binary (sTIL: <30.0% vs ≥30.0%; BMI: lean vs overweight and obese) and continuous variables. Associations with pCR (ypT0/isN0) were assessed using logistic regression, and associations with event-free survival and overall survival were assessed using Cox regressions. 236 (53.0%) patients were lean and 209 (47.0%) overweight and obese. pCR was achieved in 181 of 445 (41.7%) patients. Median sTIL was 11.0%, and 99 of 445 (22.2%) tumors had high sTIL. A statistically significant interaction between sTIL and BMI, considered as categorical or continuous variables, for predicting pCR was observed in the multivariable analysis (Pinteraction = .03 and .04, respectively). High sTIL were statistically significantly associated with pCR in lean (odds ratio [OR] = 4.24, 95% confidence interval [CI] = 2.10 to 8.56; P < .001) but not in heavier patients (OR = 1.48, 95% CI = 0.75 to 2.91; P = .26) in the multivariable analysis. High sTIL were further associated with increased event-free survival in lean (hazard ratio [HR] = 0.22, 95% CI = 0.08 to 0.62; P = .004) but not in heavier patients (HR = 0.53, 95% CI = 0.26 to 1.08; P = .08). Similar results were obtained for overall survival. BMI is modifying the effect of sTIL on pCR and prognosis in TNBC patients treated with neoadjuvant chemotherapy.

Sections du résumé

BACKGROUND
High levels of stromal tumor-infiltrating lymphocytes (sTIL) are associated with increased pathological complete response (pCR) rate and longer survival after neoadjuvant chemotherapy in triple-negative breast cancer (TNBC) patients. Here, we evaluated the value of sTIL in predicting pCR and explored prognosis in TNBC patients treated with neoadjuvant chemotherapy according to body mass index (BMI).
METHODS
sTIL were scored centrally on pretreatment biopsies from 2 retrospective series of nonunderweight TNBC patients (n = 445). sTIL and BMI were considered as binary (sTIL: <30.0% vs ≥30.0%; BMI: lean vs overweight and obese) and continuous variables. Associations with pCR (ypT0/isN0) were assessed using logistic regression, and associations with event-free survival and overall survival were assessed using Cox regressions.
RESULTS
236 (53.0%) patients were lean and 209 (47.0%) overweight and obese. pCR was achieved in 181 of 445 (41.7%) patients. Median sTIL was 11.0%, and 99 of 445 (22.2%) tumors had high sTIL. A statistically significant interaction between sTIL and BMI, considered as categorical or continuous variables, for predicting pCR was observed in the multivariable analysis (Pinteraction = .03 and .04, respectively). High sTIL were statistically significantly associated with pCR in lean (odds ratio [OR] = 4.24, 95% confidence interval [CI] = 2.10 to 8.56; P < .001) but not in heavier patients (OR = 1.48, 95% CI = 0.75 to 2.91; P = .26) in the multivariable analysis. High sTIL were further associated with increased event-free survival in lean (hazard ratio [HR] = 0.22, 95% CI = 0.08 to 0.62; P = .004) but not in heavier patients (HR = 0.53, 95% CI = 0.26 to 1.08; P = .08). Similar results were obtained for overall survival.
CONCLUSION
BMI is modifying the effect of sTIL on pCR and prognosis in TNBC patients treated with neoadjuvant chemotherapy.

Identifiants

pubmed: 33152071
pii: 5867914
doi: 10.1093/jnci/djaa090
pmc: PMC7850533
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

146-153

Commentaires et corrections

Type : CommentIn

Informations de copyright

© The Author(s) 2020. Published by Oxford University Press. All rights reserved. For permissions, please email: journals.permissions@oup.com.

Auteurs

Giuseppe Floris (G)

Department of Imaging and Pathology, Laboratory of Translational Cell & Tissue Research and University Hospitals Leuven, KU Leuven, 3000 Leuven, Belgium.

François Richard (F)

Laboratory for Translational Breast Cancer Research, Department of Oncology, KU Leuven, 3000 Leuven, Belgium.

Anne-Sophie Hamy (AS)

Lab Residual Tumor and Response to Treatment, Université Paris-Descartes, Paris, France.

Lynn Jongen (L)

Department of Oncology, KU Leuven, 3000 Leuven, Belgium.

Hans Wildiers (H)

Department of Oncology, KU Leuven, 3000 Leuven, Belgium.
Department of General Medical Oncology, University Hospitals Leuven, KU Leuven, 3000 Leuven, Belgium.

Jan Ardui (J)

Department of Gynecology and Obstetrics, University Hospitals Leuven, KU Leuven, 3000 Leuven, Belgium.

Kevin Punie (K)

Department of Oncology, KU Leuven, 3000 Leuven, Belgium.
Department of General Medical Oncology, University Hospitals Leuven, KU Leuven, 3000 Leuven, Belgium.

Ann Smeets (A)

Department of Oncology, KU Leuven, 3000 Leuven, Belgium.
Department of Surgical Oncology, University Hospitals Leuven, KU Leuven, 3000 Leuven, Belgium.

Patrick Berteloot (P)

Department of Gynecology and Obstetrics, University Hospitals Leuven, KU Leuven, 3000 Leuven, Belgium.

Ignace Vergote (I)

Department of Oncology, KU Leuven, 3000 Leuven, Belgium.
Department of Gynecology and Obstetrics, University Hospitals Leuven, KU Leuven, 3000 Leuven, Belgium.

Diane De Croze (D)

Department of Pathology, Institut Curie, Paris, France.

Didier Meseure (D)

Department of Pathology, Institut Curie, Paris, France.

Anne Salomon (A)

Department of Pathology, Institut Curie, Paris, France.

Marick Laé (M)

Department of Pathology, Centre Henri Becquerel, INSERM U1245, UNIROUEN, University of Normandie, Rouen, France.

Fabien Reyal (F)

Lab Residual Tumor and Response to Treatment, Université Paris-Descartes, Paris, France.

Elia Biganzoli (E)

Unit of Medical Statistics, Biometry and Bioinformatics "Giulio A. Maccacaro," Department of Clinical Sciences and Community Health & Data Science Research Center (DSRC), University of Milan, Fondazione IRCCS Istituto Nazionale Tumori, Milan, Italy.

Patrick Neven (P)

Department of Oncology, KU Leuven, 3000 Leuven, Belgium.
Department of Gynecology and Obstetrics, University Hospitals Leuven, KU Leuven, 3000 Leuven, Belgium.

Christine Desmedt (C)

Laboratory for Translational Breast Cancer Research, Department of Oncology, KU Leuven, 3000 Leuven, Belgium.

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