Cysteine-Altering


Journal

Stroke
ISSN: 1524-4628
Titre abrégé: Stroke
Pays: United States
ID NLM: 0235266

Informations de publication

Date de publication:
12 2020
Historique:
pubmed: 10 11 2020
medline: 18 2 2021
entrez: 9 11 2020
Statut: ppublish

Résumé

Cysteine altering A cross-sectional study using integrated clinical, neuroimaging, and whole-exome sequencing data of 92 456 participants from the Geisinger DiscovEHR initiative cohort. The case group consisted of individuals harboring a Of the 118 cases, 39.0% were men, mean age 58.1±16.9 years; 12.6% had a history of stroke, compared with 4.9% of controls. The risk of stroke was significantly increased after age 65 years (hazard ratio, 6.0 [95% CI, 1.4-26.3]). Dementia, mild cognitive impairment, migraine with aura and depression were equally prevalent in cases and controls. Twenty-nine cases (25%) and 45 controls (24%) had an available brain magnetic resonance imaging. After age 65 years, cases had a higher white matter lesion burden and more lacunes. A severe small vessel disease phenotype compatible with cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy was rarely seen. Cysteine altering

Sections du résumé

BACKGROUND AND PURPOSE
Cysteine altering
METHODS
A cross-sectional study using integrated clinical, neuroimaging, and whole-exome sequencing data of 92 456 participants from the Geisinger DiscovEHR initiative cohort. The case group consisted of individuals harboring a
RESULTS
Of the 118 cases, 39.0% were men, mean age 58.1±16.9 years; 12.6% had a history of stroke, compared with 4.9% of controls. The risk of stroke was significantly increased after age 65 years (hazard ratio, 6.0 [95% CI, 1.4-26.3]). Dementia, mild cognitive impairment, migraine with aura and depression were equally prevalent in cases and controls. Twenty-nine cases (25%) and 45 controls (24%) had an available brain magnetic resonance imaging. After age 65 years, cases had a higher white matter lesion burden and more lacunes. A severe small vessel disease phenotype compatible with cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy was rarely seen.
CONCLUSIONS
Cysteine altering

Identifiants

pubmed: 33161844
doi: 10.1161/STROKEAHA.120.030343
pmc: PMC7678653
doi:

Substances chimiques

NOTCH3 protein, human 0
Receptor, Notch3 0
Cysteine K848JZ4886

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3562-3569

Commentaires et corrections

Type : CommentIn
Type : ErratumIn

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Auteurs

Remco J Hack (RJ)

Department of Clinical Genetics, Leiden University Medical Center, the Netherlands (R.J.H., J.W.R., S.A.J.L.O.).

Julie W Rutten (JW)

Department of Clinical Genetics, Leiden University Medical Center, the Netherlands (R.J.H., J.W.R., S.A.J.L.O.).

Thomas N Person (TN)

Geisinger Genomic Medicine Institute, Danville, PA (T.N.P.).

Jiang Li (J)

Department of Molecular and Functional Genomics, Geisinger, Danville, PA (J.L., V.A.).

Ayesha Khan (A)

Neuroscience Institute, Geisinger, Danville, PA (A.K., C.J.G., R.Z.).

Christoph J Griessenauer (CJ)

Neuroscience Institute, Geisinger, Danville, PA (A.K., C.J.G., R.Z.).
Institute of Neurointervention, Paracelsus Medical University, Salzburg, Austria (C.J.G.). Regeneron Genetics Center, Tarrytown, New York.

Vida Abedi (V)

Department of Molecular and Functional Genomics, Geisinger, Danville, PA (J.L., V.A.).

Saskia A J Lesnik Oberstein (SAJ)

Department of Clinical Genetics, Leiden University Medical Center, the Netherlands (R.J.H., J.W.R., S.A.J.L.O.).

Ramin Zand (R)

Neuroscience Institute, Geisinger, Danville, PA (A.K., C.J.G., R.Z.).

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Classifications MeSH