Endogenous production of glutamine and plasma glutamine concentration in critically ill patients.

Glutamine clearance Glutamine metabolism Protein breakdown Rate of appearance

Journal

Clinical nutrition ESPEN
ISSN: 2405-4577
Titre abrégé: Clin Nutr ESPEN
Pays: England
ID NLM: 101654592

Informations de publication

Date de publication:
12 2020
Historique:
received: 17 03 2020
revised: 08 09 2020
accepted: 14 09 2020
entrez: 13 11 2020
pubmed: 14 11 2020
medline: 25 9 2021
Statut: ppublish

Résumé

Glutamine plasma concentrations outside the normal range at intensive care unit (ICU) admission are associated with unfavorable outcomes. Based on the hypothesis that hypoglutaminemia in the ICU is the result of an increased utilization of glutamine which cannot be fully met by endogenous production, extra glutamine supplementation has been advocated to ICU patients with hypoglutaminemia. However, it is still unclear whether there is a causal relation between hypo- and hyperglutaminemia and outcomes. Present guidelines advise against supplementation, although there is no evidence available for patients with hypoglutaminemia. The pathophysiology of abnormal glutamine levels and whether glutamine production or glutamine utilization is compromised is largely unknown. Therefore, the aim of this study was to elucidate the relationship between plasma glutamine levels and the endogenous glutamine production in ICU patients. In this observational study, a technique using a small bolus of intravenous glutamine with an isotopic label was used to measure glutamine production. There was a statistically significant correlation between de novo endogenous production of glutamine (not emanating directly from protein breakdown) and plasma glutamine concentrations in the low and normal range in circulatory stabilized ICU patients (n = 19), R The predictive value of a low plasma glutamine concentration at ICU admission on outcomes may thus be related to a low endogenous production, which may need to be supplemented in the best interest of this cohort of patients.

Sections du résumé

BACKGROUND
Glutamine plasma concentrations outside the normal range at intensive care unit (ICU) admission are associated with unfavorable outcomes. Based on the hypothesis that hypoglutaminemia in the ICU is the result of an increased utilization of glutamine which cannot be fully met by endogenous production, extra glutamine supplementation has been advocated to ICU patients with hypoglutaminemia. However, it is still unclear whether there is a causal relation between hypo- and hyperglutaminemia and outcomes. Present guidelines advise against supplementation, although there is no evidence available for patients with hypoglutaminemia. The pathophysiology of abnormal glutamine levels and whether glutamine production or glutamine utilization is compromised is largely unknown. Therefore, the aim of this study was to elucidate the relationship between plasma glutamine levels and the endogenous glutamine production in ICU patients.
METHOD
In this observational study, a technique using a small bolus of intravenous glutamine with an isotopic label was used to measure glutamine production.
RESULTS
There was a statistically significant correlation between de novo endogenous production of glutamine (not emanating directly from protein breakdown) and plasma glutamine concentrations in the low and normal range in circulatory stabilized ICU patients (n = 19), R
CONCLUSION
The predictive value of a low plasma glutamine concentration at ICU admission on outcomes may thus be related to a low endogenous production, which may need to be supplemented in the best interest of this cohort of patients.

Identifiants

pubmed: 33183541
pii: S2405-4577(20)30202-3
doi: 10.1016/j.clnesp.2020.09.015
pii:
doi:

Substances chimiques

Glutamine 0RH81L854J

Types de publication

Journal Article Observational Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

226-230

Informations de copyright

Copyright © 2020 The Authors. Published by Elsevier Ltd.. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest None.

Auteurs

Marie Smedberg (M)

Department of Anesthesiology and Intensive Care, Karolinska Institutet and Perioperative Medicine and Intensive Care, Karolinska University Hospital Huddinge, B31 Perioperative Medicine and Intensive Care, 141 86, Stockholm, Sweden. Electronic address: marie.smedberg@sll.se.

Olav Rooyackers (O)

Department of Anesthesiology and Intensive Care, Karolinska Institutet and Perioperative Medicine and Intensive Care, Karolinska University Hospital Huddinge, B31 Perioperative Medicine and Intensive Care, 141 86, Stockholm, Sweden. Electronic address: olav.rooyackers@ki.se.

Åke Norberg (Å)

Department of Anesthesiology and Intensive Care, Karolinska Institutet and Perioperative Medicine and Intensive Care, Karolinska University Hospital Huddinge, B31 Perioperative Medicine and Intensive Care, 141 86, Stockholm, Sweden. Electronic address: ake.norberg@sll.se.

Inga Tjäder (I)

Department of Anesthesiology and Intensive Care, Karolinska Institutet and Perioperative Medicine and Intensive Care, Karolinska University Hospital Huddinge, B31 Perioperative Medicine and Intensive Care, 141 86, Stockholm, Sweden. Electronic address: inga.tjader@sll.se.

Jan Wernerman (J)

Department of Anesthesiology and Intensive Care, Karolinska Institutet and Perioperative Medicine and Intensive Care, Karolinska University Hospital Huddinge, B31 Perioperative Medicine and Intensive Care, 141 86, Stockholm, Sweden. Electronic address: jan.wernerman@sll.se.

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Classifications MeSH