A transcriptomic signature to predict adjuvant gemcitabine sensitivity in pancreatic adenocarcinoma.


Journal

Annals of oncology : official journal of the European Society for Medical Oncology
ISSN: 1569-8041
Titre abrégé: Ann Oncol
Pays: England
ID NLM: 9007735

Informations de publication

Date de publication:
02 2021
Historique:
received: 29 07 2020
revised: 29 10 2020
accepted: 29 10 2020
pubmed: 15 11 2020
medline: 9 2 2021
entrez: 14 11 2020
Statut: ppublish

Résumé

Chemotherapy is the only systemic treatment approved for pancreatic ductal adenocarcinoma (PDAC), with a selection of regimens based on patients' performance status and expected efficacy. The establishment of a potent stratification associated with chemotherapeutic efficacy could potentially improve prognosis by tailoring treatments. Concomitant chemosensitivity and genome-wide RNA profiles were carried out on preclinical models (primary cell cultures and patient-derived xenografts) derived from patients with PDAC included in the PaCaOmics program (NCT01692873). The RNA-based stratification was tested in a monocentric cohort and validated in a multicentric cohort, both retrospectively collected from resected PDAC samples (67 and 368 patients, respectively). Forty-three (65%) and 203 (55%) patients received adjuvant gemcitabine in the monocentric and the multicentric cohorts, respectively. The relationships between predicted gemcitabine sensitivity and patients' overall survival (OS) and disease-free survival were investigated. The GemPred RNA signature was derived from preclinical models, defining gemcitabine sensitive PDAC as GemPred+. Among the patients who received gemcitabine in the test and validation cohorts, the GemPred+ patients had a higher OS than GemPred- (P = 0.046 and P = 0.00216). In both cohorts, the GemPred stratification was not associated with OS among patients who did not receive gemcitabine. Among gemcitabine-treated patients, GemPred+ patients had significantly higher OS than the GemPred-: 91.3 months [95% confidence interval (CI): 61.2-not reached] versus 33 months (95% CI: 24-35.2); hazard ratio 0.403 (95% CI: 0.221-0.735, P = 0.00216). The interaction test for gemcitabine and GemPred+ stratification was significant (P = 0.0245). Multivariate analysis in the gemcitabine-treated population retained an independent predictive value. The RNA-based GemPred stratification predicts the benefit of adjuvant gemcitabine in PDAC patients.

Sections du résumé

BACKGROUND
Chemotherapy is the only systemic treatment approved for pancreatic ductal adenocarcinoma (PDAC), with a selection of regimens based on patients' performance status and expected efficacy. The establishment of a potent stratification associated with chemotherapeutic efficacy could potentially improve prognosis by tailoring treatments.
PATIENTS AND METHODS
Concomitant chemosensitivity and genome-wide RNA profiles were carried out on preclinical models (primary cell cultures and patient-derived xenografts) derived from patients with PDAC included in the PaCaOmics program (NCT01692873). The RNA-based stratification was tested in a monocentric cohort and validated in a multicentric cohort, both retrospectively collected from resected PDAC samples (67 and 368 patients, respectively). Forty-three (65%) and 203 (55%) patients received adjuvant gemcitabine in the monocentric and the multicentric cohorts, respectively. The relationships between predicted gemcitabine sensitivity and patients' overall survival (OS) and disease-free survival were investigated.
RESULTS
The GemPred RNA signature was derived from preclinical models, defining gemcitabine sensitive PDAC as GemPred+. Among the patients who received gemcitabine in the test and validation cohorts, the GemPred+ patients had a higher OS than GemPred- (P = 0.046 and P = 0.00216). In both cohorts, the GemPred stratification was not associated with OS among patients who did not receive gemcitabine. Among gemcitabine-treated patients, GemPred+ patients had significantly higher OS than the GemPred-: 91.3 months [95% confidence interval (CI): 61.2-not reached] versus 33 months (95% CI: 24-35.2); hazard ratio 0.403 (95% CI: 0.221-0.735, P = 0.00216). The interaction test for gemcitabine and GemPred+ stratification was significant (P = 0.0245). Multivariate analysis in the gemcitabine-treated population retained an independent predictive value.
CONCLUSION
The RNA-based GemPred stratification predicts the benefit of adjuvant gemcitabine in PDAC patients.

Identifiants

pubmed: 33188873
pii: S0923-7534(20)43131-X
doi: 10.1016/j.annonc.2020.10.601
pii:
doi:

Substances chimiques

Deoxycytidine 0W860991D6
Gemcitabine 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

250-260

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2020 The Authors. Published by Elsevier Ltd.. All rights reserved.

Déclaration de conflit d'intérêts

Disclosure RN, YB, MG, JI and NJD have a pending patent entitled ‘Evaluation of the efficiency of an anticancer compound for a PDAC patient’ filed 23 January 2020 (European patent application number EP20305052.1). All other authors have declared no conflicts of interest.

Auteurs

R Nicolle (R)

Tumour Identity Card Program (CIT), French League Against Cancer, Paris, France.

O Gayet (O)

Cancer Research Center of Marseille, CRCM, Inserm, CNRS, Paoli-Calmettes Institut, Aix-Marseille University, Marseille, France.

P Duconseil (P)

Cancer Research Center of Marseille, CRCM, Inserm, CNRS, Paoli-Calmettes Institut, Aix-Marseille University, Marseille, France; Nord Hospital, Marseille, France.

C Vanbrugghe (C)

Cancer Research Center of Marseille, CRCM, Inserm, CNRS, Paoli-Calmettes Institut, Aix-Marseille University, Marseille, France; Nord Hospital, Marseille, France.

J Roques (J)

Cancer Research Center of Marseille, CRCM, Inserm, CNRS, Paoli-Calmettes Institut, Aix-Marseille University, Marseille, France.

M Bigonnet (M)

Cancer Research Center of Marseille, CRCM, Inserm, CNRS, Paoli-Calmettes Institut, Aix-Marseille University, Marseille, France.

Y Blum (Y)

Tumour Identity Card Program (CIT), French League Against Cancer, Paris, France.

N Elarouci (N)

Tumour Identity Card Program (CIT), French League Against Cancer, Paris, France.

L Armenoult (L)

Tumour Identity Card Program (CIT), French League Against Cancer, Paris, France.

M Ayadi (M)

Tumour Identity Card Program (CIT), French League Against Cancer, Paris, France.

A de Reyniès (A)

Tumour Identity Card Program (CIT), French League Against Cancer, Paris, France.

F Puleo (F)

Laboratory of Experimental Gastroenterology (Université Libre de Bruxelles), Brussels, Belgium; Department of Gastroenterology and Digestive Oncology, Delta Hospital, Center Hospitalier Interregional Edith Cavell, Brussels, Belgium.

J Augustin (J)

Sorbonne University, UPMC University, Department of Gastroenterology, Pitié-Salpetriére Hospital, Paris, France.

J F Emile (JF)

Ambroise Paré Hospital, Boulogne, AP-HP, Boulogne-Billancourt, France.

M Svrcek (M)

Sorbonne University, UPMC University, Department of Gastroenterology, Pitié-Salpetriére Hospital, Paris, France.

T Arsenijevic (T)

Laboratory of Experimental Gastroenterology (Université Libre de Bruxelles), Brussels, Belgium; Department of Gastroenterology and Digestive Oncology, Delta Hospital, Center Hospitalier Interregional Edith Cavell, Brussels, Belgium.

P Hammel (P)

Department of Digestive Oncology, Beaujon Hospital, Paris 7 University, APHP, Clichy, France.

M Giovannini (M)

Cancer Research Center of Marseille, CRCM, Inserm, CNRS, Paoli-Calmettes Institut, Aix-Marseille University, Marseille, France; Paoli-Calmettes Institut, Marseille, France.

P Grandval (P)

Cancer Research Center of Marseille, CRCM, Inserm, CNRS, Paoli-Calmettes Institut, Aix-Marseille University, Marseille, France; La Timone Hospital, Marseille, France.

L Dahan (L)

Cancer Research Center of Marseille, CRCM, Inserm, CNRS, Paoli-Calmettes Institut, Aix-Marseille University, Marseille, France; La Timone Hospital, Marseille, France.

V Moutardier (V)

Cancer Research Center of Marseille, CRCM, Inserm, CNRS, Paoli-Calmettes Institut, Aix-Marseille University, Marseille, France; Nord Hospital, Marseille, France.

M Gilabert (M)

Cancer Research Center of Marseille, CRCM, Inserm, CNRS, Paoli-Calmettes Institut, Aix-Marseille University, Marseille, France; Paoli-Calmettes Institut, Marseille, France.

J L Van Laethem (JL)

Laboratory of Experimental Gastroenterology (Université Libre de Bruxelles), Brussels, Belgium; Department of Gastroenterology and Digestive Oncology, Delta Hospital, Center Hospitalier Interregional Edith Cavell, Brussels, Belgium.

J B Bachet (JB)

Sorbonne University, UPMC University, Department of Gastroenterology, Pitié-Salpetriére Hospital, Paris, France.

J Cros (J)

Department of Digestive Oncology, Beaujon Hospital, Paris 7 University, APHP, Clichy, France.

J Iovanna (J)

Cancer Research Center of Marseille, CRCM, Inserm, CNRS, Paoli-Calmettes Institut, Aix-Marseille University, Marseille, France. Electronic address: juan.iovanna@inserm.fr.

N J Dusetti (NJ)

Cancer Research Center of Marseille, CRCM, Inserm, CNRS, Paoli-Calmettes Institut, Aix-Marseille University, Marseille, France. Electronic address: nelson.dusetti@inserm.fr.

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