Signaling via the Interleukin-10 Receptor Attenuates Cardiac Hypertrophy in Mice During Pressure Overload, but not Isoproterenol Infusion.
cardiac hypertrophy
heart failure
inflammation
interleukin-10
signaling pathway
Journal
Frontiers in pharmacology
ISSN: 1663-9812
Titre abrégé: Front Pharmacol
Pays: Switzerland
ID NLM: 101548923
Informations de publication
Date de publication:
2020
2020
Historique:
received:
05
05
2020
accepted:
06
10
2020
entrez:
16
11
2020
pubmed:
17
11
2020
medline:
17
11
2020
Statut:
epublish
Résumé
Inflammation plays a key role during cardiac hypertrophy and the development of heart failure. Interleukin-10 (IL-10) is a major anti-inflammatory cytokine that is expressed in the heart and may play a crucial role in cardiac remodeling. Based on the evidence that IL-10 potentially reduces pathological hypertrophy, it was hypothesized that signaling via the IL-10 receptor (IL10R) in the heart produces a protective role in reducing cardiac hypertrophy. The aim of this study was to investigate the effects of the ablation of
Identifiants
pubmed: 33192505
doi: 10.3389/fphar.2020.559220
pii: 559220
pmc: PMC7662881
doi:
Types de publication
Journal Article
Langues
eng
Pagination
559220Subventions
Organisme : British Heart Foundation
ID : PG/16/77/32400
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/P015816/1
Pays : United Kingdom
Organisme : British Heart Foundation
ID : PG/17/78/33304
Pays : United Kingdom
Organisme : British Heart Foundation
ID : PG/18/40/33767
Pays : United Kingdom
Organisme : British Heart Foundation
ID : PG/11/23/28801
Pays : United Kingdom
Organisme : Medical Research Council
ID : G1002082
Pays : United Kingdom
Informations de copyright
Copyright © 2020 Stafford, Assrafally, Prehar, Zi, De Morais, Maqsood, Cartwright, Muller and Oceandy.
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