Host CD39 Deficiency Affects Radiation-Induced Tumor Growth Delay and Aggravates Radiation-Induced Normal Tissue Toxicity.
ATP
CD73
cancer
ionizing radiation
purinergic signaling
Journal
Frontiers in oncology
ISSN: 2234-943X
Titre abrégé: Front Oncol
Pays: Switzerland
ID NLM: 101568867
Informations de publication
Date de publication:
2020
2020
Historique:
received:
23
04
2020
accepted:
10
09
2020
entrez:
16
11
2020
pubmed:
17
11
2020
medline:
17
11
2020
Statut:
epublish
Résumé
The ectonucleoside triphosphate diphosphohydrolase (CD39)/5' ectonuclotidase (CD73)-dependent purinergic pathway emerges as promising cancer target. Yet, except for own previous work revealing a pathogenic role of CD73 and adenosine in radiation-induced lung fibrosis, the role of purinergic signaling for radiotherapy outcome remained elusive. Here we used C57BL/6 wild-type (WT), CD39 knockout (CD39
Identifiants
pubmed: 33194619
doi: 10.3389/fonc.2020.554883
pmc: PMC7649817
doi:
Types de publication
Journal Article
Langues
eng
Pagination
554883Informations de copyright
Copyright © 2020 Meyer, Klein, de Leve, Szymonowicz, Stuschke, Robson, Jendrossek and Wirsdörfer.
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