BK Channels Regulate LPS-induced CCL-2 Release from Human Pulmonary Endothelial Cells.
A549 Cells
Acute Lung Injury
/ chemically induced
Alveolar Epithelial Cells
/ drug effects
Calcium
/ metabolism
Cell Line
Cell Line, Tumor
Chemokine CCL2
/ metabolism
Cytokines
/ metabolism
Endothelial Cells
/ drug effects
HEK293 Cells
Humans
Hyperoxia
/ chemically induced
Inflammation
/ chemically induced
Interleukin-6
/ metabolism
Large-Conductance Calcium-Activated Potassium Channels
/ metabolism
Lipopolysaccharides
/ pharmacology
Lung
/ drug effects
Membrane Potentials
/ drug effects
Potassium Channels, Tandem Pore Domain
/ metabolism
LPS
acute lung injury
cytokines
inflammation
large conductance potassium channels
Journal
American journal of respiratory cell and molecular biology
ISSN: 1535-4989
Titre abrégé: Am J Respir Cell Mol Biol
Pays: United States
ID NLM: 8917225
Informations de publication
Date de publication:
02 2021
02 2021
Historique:
pubmed:
21
11
2020
medline:
23
2
2021
entrez:
20
11
2020
Statut:
ppublish
Résumé
We recently established a role for the stretch-activated two-pore-domain K
Identifiants
pubmed: 33217242
doi: 10.1165/rcmb.2020-0228OC
pmc: PMC7874395
doi:
Substances chimiques
CCL2 protein, human
0
Chemokine CCL2
0
Cytokines
0
Interleukin-6
0
Large-Conductance Calcium-Activated Potassium Channels
0
Lipopolysaccharides
0
Potassium Channels, Tandem Pore Domain
0
potassium channel protein TREK-1
0
Calcium
SY7Q814VUP
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Langues
eng
Sous-ensembles de citation
IM
Pagination
224-234Subventions
Organisme : NHLBI NIH HHS
ID : K08 HL118118
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL130308
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL134346
Pays : United States
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