Efficacy and safety of nintedanib in Asian patients with systemic sclerosis-associated interstitial lung disease: Subgroup analysis of the SENSCIS trial.


Journal

Respiratory investigation
ISSN: 2212-5353
Titre abrégé: Respir Investig
Pays: Netherlands
ID NLM: 101581124

Informations de publication

Date de publication:
Mar 2021
Historique:
received: 14 09 2020
accepted: 14 10 2020
pubmed: 24 11 2020
medline: 21 8 2021
entrez: 23 11 2020
Statut: ppublish

Résumé

In the SENSCIS trial in patients with systemic sclerosis-associated interstitial lung disease (SSc-ILD), nintedanib reduced the rate of decline in forced vital capacity (FVC) (mL/year) over 52 weeks by 44% in comparison with placebo, with manageable adverse events in most patients. We analyzed the efficacy and safety of nintedanib in patients of Asian race. Patients with SSc-ILD were randomized to receive nintedanib or placebo. The outcomes over 52 weeks were analyzed in Asian versus non-Asian patients. Of the 288 patients in each treatment group, 62 (21.5%) in the nintedanib group and 81 (28.1%) in the placebo group were Asian; 90.2% of the Asian patients were enrolled in Asian countries. In the placebo group, the rate of FVC decline over 52 weeks was consistent between Asian and non-Asian patients (-99.9 and -90.6 mL/year, respectively). The effect of nintedanib on reducing the rate of FVC decline over 52 weeks was consistent between Asian (difference, 44.3 mL/year [95% CI: -32.8, 121.4]) and non-Asian patients (difference, 39.0 mL/year [95% CI: -5.1, 83.1]) (treatment-by-time-by-subgroup interaction, p = 0.91). Diarrhea was the most frequent adverse event and was reported in similar proportions of Asian and non-Asian patients in the nintedanib group (80.6% and 74.3%, respectively) and placebo group (28.4% and 32.9%, respectively). In patients with SSc-ILD, nintedanib had a consistent benefit on slowing the progression of SSc-ILD in Asian and non-Asian patients, with a similar adverse event profile. ClinicalTrials.gov NCT02597933.

Sections du résumé

BACKGROUND AND OBJECTIVE OBJECTIVE
In the SENSCIS trial in patients with systemic sclerosis-associated interstitial lung disease (SSc-ILD), nintedanib reduced the rate of decline in forced vital capacity (FVC) (mL/year) over 52 weeks by 44% in comparison with placebo, with manageable adverse events in most patients. We analyzed the efficacy and safety of nintedanib in patients of Asian race.
METHODS METHODS
Patients with SSc-ILD were randomized to receive nintedanib or placebo. The outcomes over 52 weeks were analyzed in Asian versus non-Asian patients.
RESULTS RESULTS
Of the 288 patients in each treatment group, 62 (21.5%) in the nintedanib group and 81 (28.1%) in the placebo group were Asian; 90.2% of the Asian patients were enrolled in Asian countries. In the placebo group, the rate of FVC decline over 52 weeks was consistent between Asian and non-Asian patients (-99.9 and -90.6 mL/year, respectively). The effect of nintedanib on reducing the rate of FVC decline over 52 weeks was consistent between Asian (difference, 44.3 mL/year [95% CI: -32.8, 121.4]) and non-Asian patients (difference, 39.0 mL/year [95% CI: -5.1, 83.1]) (treatment-by-time-by-subgroup interaction, p = 0.91). Diarrhea was the most frequent adverse event and was reported in similar proportions of Asian and non-Asian patients in the nintedanib group (80.6% and 74.3%, respectively) and placebo group (28.4% and 32.9%, respectively).
CONCLUSIONS CONCLUSIONS
In patients with SSc-ILD, nintedanib had a consistent benefit on slowing the progression of SSc-ILD in Asian and non-Asian patients, with a similar adverse event profile.
TRIAL REGISTRATION BACKGROUND
ClinicalTrials.gov NCT02597933.

Identifiants

pubmed: 33223487
pii: S2212-5345(20)30156-8
doi: 10.1016/j.resinv.2020.10.005
pii:
doi:

Substances chimiques

Indoles 0
nintedanib G6HRD2P839

Banques de données

ClinicalTrials.gov
['NCT02597933']

Types de publication

Journal Article Multicenter Study Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

252-259

Informations de copyright

Copyright © 2020 The Authors. Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Conflict of Interest AA reports honoraria (≥500,000 JPY annually) from Boehringer Ingelheim and travel fees, gifts, and others (≥500,000 JPY annually) from Boehringer Ingelheim. LC reports employment/leadership position/advisory role (≥1,000,000 JPY annually) from Boehringer Ingelheim, Eicos, Mitsubishi Tanabe and Reata Pharmaceuticals; honoraria (≥500,000 JPY annually) from Boehringer Ingelheim; and research funding (≥1,000,000 JPY annually) from Boehringer Ingelheim and United Therapeutics. YK reports honoraria (≥500,000 JPY annually) from Boehringer Ingelheim. TO reports honoraria (≥500,000 JPY annually) from Boehringer Ingelheim and Shionogi; and research funding (≥1,000,000 JPY annually) from Boehringer Ingelheim. MO reports honoraria (≥500,000 JPY annually) from Boehringer Ingelheim. XM, MG and MA are employees of Boehringer Ingelheim. MK reports employment/leadership position/advisory role (≥1,000,000 JPY annually) from Boehringer Ingelheim, Chugai and Corbus; patent royalties/licensing fees (≥1,000,000 JPY annually) from MBL; honoraria (≥500,000 JPY annually) from AbbVie, Actelion, Astellas, Bayer, Boehringer Ingelheim, Chugai, Eisai, GlaxoSmithKline, Ono Pharmaceuticals, Mitsubishi Tanabe and Pfizer; research funding (≥1,000,000 JPY) annually from Boehringer Ingelheim and Ono Pharmaceuticals; and subsidies or donations (≥1,000,000 JPY annually) from AbbVie, Boehringer Ingelheim, Chugai, Eisai, Ono Pharmaceuticals and Mitsubishi Tanabe. DB, MC, RS, XZ and HZ have nothing to disclose.

Auteurs

Arata Azuma (A)

Department of Pulmonary Medicine and Oncology, Graduate School of Medicine, Nippon Medical School, Tokyo, Japan. Electronic address: azuma_arata@yahoo.co.jp.

Lorinda Chung (L)

Departments of Medicine and Dermatology, Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, CA, USA.

Digambar Behera (D)

Department of Pulmonary Medicine, Postgraduate Institute of Medical Education and Research, Chandigarh, India.

Melody Chung (M)

Department of Medicine, Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, CA, USA.

Yasuhiro Kondoh (Y)

Department of Respiratory Medicine and Allergy, Tosei General Hospital, Japan.

Takashi Ogura (T)

Department of Respiratory Medicine, Kanagawa Cardiovascular and Respiratory Centre, Yokohama, Japan.

Masaki Okamoto (M)

Department of Internal Medicine, Division of Respirology, Neurology, and Rheumatology, Kurume University School of Medicine, Japan.

Rajesh Swarnakar (R)

Department of Respiratory Medicine, Getwell Hospital and Research Institute, Nagpur, Maharashtra, India.

Xiaofeng Zeng (X)

Department of Rheumatology, Peking Union Medical College Hospital, Beijing, China.

Heijan Zou (H)

Division of Rheumatology, Huashan Hospital, Fudan University, Shanghai, China.

Xianhua Meng (X)

Boehringer Ingelheim (China) Investment Co., Ltd, Shanghai, China.

Martina Gahlemann (M)

Boehringer Ingelheim (Schweiz) GmbH, Basel, Switzerland.

Margarida Alves (M)

Boehringer Ingelheim International GmbH, Ingelheim, Germany.

Masataka Kuwana (M)

Department of Allergy and Rheumatology, Nippon Medical School Graduate School of Medicine, Tokyo, Japan.

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