Higher omega-3 index is associated with more rapid heart rate recovery in healthy men and women.


Journal

Prostaglandins, leukotrienes, and essential fatty acids
ISSN: 1532-2823
Titre abrégé: Prostaglandins Leukot Essent Fatty Acids
Pays: Scotland
ID NLM: 8802730

Informations de publication

Date de publication:
12 2020
Historique:
received: 30 07 2020
revised: 07 10 2020
accepted: 07 11 2020
pubmed: 24 11 2020
medline: 3 6 2021
entrez: 23 11 2020
Statut: ppublish

Résumé

Previous studies have suggested that omega-3 polyunsaturated fatty acids (n-3 PUFA) can favorably influence cardiac autonomic tone. However, data regarding n-3 PUFA status and heart rate recovery (HRR) in healthy adults are sparse. To examine the association between n-3 PUFA status and HRR. Participants included 13,912 patients who underwent a comprehensive examination at the Cooper Clinic, Dallas TX. Fitness was determined from a maximal exercise test. HRR was calculated by subtracting the heart rate at 1, 3, and 5 min of an active recovery period from the maximal heart rate. Participants were categorized as having a low (<4%), normal (4-8%) or optimal (>8%) Omega-3 Index (O3I) (i.e., erythrocyte levels of eicosapentaenoic and docosahexaenoic acids). Multiple linear regression was used to model the association between O3I and HRR adjusting for age, maximal METs, body mass index, and smoking by sex. Higher categories of O3I were associated with greater HRR at 1 min (men: 23.7, 23.9, 24.6 beats/min; women: 23.9, 24.6, 25.9 and 3 min (men: 52.4, 52.9, 53.6 beats/min; women: 51.9, 53.4, 54.6), p trend <0.01 for all. Corresponding HRR at 5 min were (men: 60.0, 60.2, 60.7 beats/min, p trend=0.09; women: 59.4, 60.8, 61.6, p trend <0.001). The HRR gradients across O3I categories were steeper in women than men at 1, 3, and 5 min (p<0.03 for all sex x O3I category interactions with HRR). A direct relationship between HRR and O3I values was observed in both men and women, with a steeper gradient in women. These findings suggest a potential cardioprotective mechanism for n-3 PUFA.

Sections du résumé

BACKGROUND
Previous studies have suggested that omega-3 polyunsaturated fatty acids (n-3 PUFA) can favorably influence cardiac autonomic tone. However, data regarding n-3 PUFA status and heart rate recovery (HRR) in healthy adults are sparse.
PURPOSE
To examine the association between n-3 PUFA status and HRR.
METHODS
Participants included 13,912 patients who underwent a comprehensive examination at the Cooper Clinic, Dallas TX. Fitness was determined from a maximal exercise test. HRR was calculated by subtracting the heart rate at 1, 3, and 5 min of an active recovery period from the maximal heart rate. Participants were categorized as having a low (<4%), normal (4-8%) or optimal (>8%) Omega-3 Index (O3I) (i.e., erythrocyte levels of eicosapentaenoic and docosahexaenoic acids). Multiple linear regression was used to model the association between O3I and HRR adjusting for age, maximal METs, body mass index, and smoking by sex.
RESULTS
Higher categories of O3I were associated with greater HRR at 1 min (men: 23.7, 23.9, 24.6 beats/min; women: 23.9, 24.6, 25.9 and 3 min (men: 52.4, 52.9, 53.6 beats/min; women: 51.9, 53.4, 54.6), p trend <0.01 for all. Corresponding HRR at 5 min were (men: 60.0, 60.2, 60.7 beats/min, p trend=0.09; women: 59.4, 60.8, 61.6, p trend <0.001). The HRR gradients across O3I categories were steeper in women than men at 1, 3, and 5 min (p<0.03 for all sex x O3I category interactions with HRR).
CONCLUSIONS
A direct relationship between HRR and O3I values was observed in both men and women, with a steeper gradient in women. These findings suggest a potential cardioprotective mechanism for n-3 PUFA.

Identifiants

pubmed: 33227647
pii: S0952-3278(20)30164-2
doi: 10.1016/j.plefa.2020.102206
pii:
doi:

Substances chimiques

Cardiotonic Agents 0
Fatty Acids, Omega-3 0

Types de publication

Clinical Trial Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

102206

Informations de copyright

Copyright © 2020. Published by Elsevier Ltd.

Auteurs

Stephen W Farrell (SW)

The Cooper Institute, Dallas, TX USA. Electronic address: sfarrell@cooperinst.org.

Laura F DeFina (LF)

The Cooper Institute, Dallas, TX USA.

Nathan Tintle (N)

Fatty Acid Research Institute, Sioux Falls, SD; and Department of Mathematics & Statistics; Dordt University; Sioux Center; IA.

Carolyn E Barlow (CE)

The Cooper Institute, Dallas, TX USA.

David Leonard (D)

The Cooper Institute, Dallas, TX USA.

William L Haskell (WL)

Department of Medicine, Stanford University, Palo Alto, CA United States.

Jarett D Berry (JD)

Division of Cardiology, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas TX United States.

Benjamin L Willis (BL)

The Cooper Institute, Dallas, TX USA.

Andjelka Pavlovic (A)

The Cooper Institute, Dallas, TX USA.

William S Harris (WS)

Fatty Acid Research Institute, Sioux Falls, SD; and Department of Internal Medicine, Sanford School of Medicine, University of South Dakota, Sioux Falls, SD.

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Classifications MeSH