Role of iRhoms 1 and 2 in Endochondral Ossification.


Journal

International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791

Informations de publication

Date de publication:
19 Nov 2020
Historique:
received: 06 10 2020
revised: 10 11 2020
accepted: 12 11 2020
entrez: 24 11 2020
pubmed: 25 11 2020
medline: 5 3 2021
Statut: epublish

Résumé

Growth of the axial and appendicular skeleton depends on endochondral ossification, which is controlled by tightly regulated cell-cell interactions in the developing growth plates. Previous studies have uncovered an important role of a disintegrin and metalloprotease 17 (ADAM17) in the normal development of the mineralized zone of hypertrophic chondrocytes during endochondral ossification. ADAM17 regulates EGF-receptor signaling by cleaving EGFR-ligands such as TGFα from their membrane-anchored precursor. The activity of ADAM17 is controlled by two regulatory binding partners, the inactive Rhomboids 1 and 2 (iRhom1, 2), raising questions about their role in endochondral ossification. To address this question, we generated mice lacking iRhom2 (

Identifiants

pubmed: 33227998
pii: ijms21228732
doi: 10.3390/ijms21228732
pmc: PMC7699240
pii:
doi:

Substances chimiques

Carrier Proteins 0
Col2a1 protein, mouse 0
Collagen Type II 0
Membrane Proteins 0
Transforming Growth Factor alpha 0
iRhom1 protein, mouse 0
iRhom2 protein, mouse 0
EGFR protein, mouse EC 2.7.10.1
ErbB Receptors EC 2.7.10.1
Cre recombinase EC 2.7.7.-
Integrases EC 2.7.7.-
ADAM17 Protein EC 3.4.24.86
Adam17 protein, mouse EC 3.4.24.86

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : NIGMS NIH HHS
ID : R35 GM134907
Pays : United States
Organisme : Deutsche Forschungsgemeinschaft
ID : EXC 2145 SyNergy- ID 390857198
Organisme : NIH HHS
ID : R01 GM64750; R35 GM134907
Pays : United States

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Auteurs

Renpeng Fang (R)

Department of Orthopaedics, Xiangya Hospital, Central South University, Changsha 410008, China.
Arthritis and Tissue Degeneration Program, Hospital for Special Surgery at Weill Cornell Medicine, New York, NY 10021, USA.

Coline Haxaire (C)

Arthritis and Tissue Degeneration Program, Hospital for Special Surgery at Weill Cornell Medicine, New York, NY 10021, USA.

Miguel Otero (M)

Orthopedic Soft Tissue Research Program, Hospital for Special Surgery at Weill Cornell Medicine, New York, NY 10021, USA.

Samantha Lessard (S)

Orthopedic Soft Tissue Research Program, Hospital for Special Surgery at Weill Cornell Medicine, New York, NY 10021, USA.

Gisela Weskamp (G)

Arthritis and Tissue Degeneration Program, Hospital for Special Surgery at Weill Cornell Medicine, New York, NY 10021, USA.

David R McIlwain (DR)

Baxter Laboratory in Stem Cell Biology, Department of Pathology, Stanford University School of Medicine, Stanford, CA 94305, USA.

Tak W Mak (TW)

Campbell Family Institute for Breast Cancer Research, Ontario Cancer Institute, University Health Network, Toronto, ON M5G 2M9, Canada.

Stefan F Lichtenthaler (SF)

German Center for Neurodegenerative Diseases (DZNE), 81377 Munich, Germany.
Neuroproteomics, School of Medicine, Klinikum rechts der Isar, Technical University of Munich, 81675 Munich, Germany.
Munich Cluster for Systems Neurology (SyNergy), 81377 Munich, Germany.
Institute for Advanced Study, Technische Universität München, 85748 Garching, Germany.

Carl P Blobel (CP)

Arthritis and Tissue Degeneration Program, Hospital for Special Surgery at Weill Cornell Medicine, New York, NY 10021, USA.
Institute for Advanced Study, Technische Universität München, 85748 Garching, Germany.
Department of Medicine, Department of Biophysics, Physiology and Systems Biology, Weill Cornell Medicine, New York, NY 10021, USA.

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Classifications MeSH