Residence at moderately high altitude and its relationship with WHO Group 1 pulmonary arterial hypertension symptom severity and clinical characteristics: the Pulmonary Hypertension Association Registry.

6-min walk distance elevation pulmonary arterial hypertension pulmonary vascular resistance vascular

Journal

Pulmonary circulation
ISSN: 2045-8932
Titre abrégé: Pulm Circ
Pays: United States
ID NLM: 101557243

Informations de publication

Date de publication:
Historique:
received: 22 07 2020
accepted: 15 09 2020
entrez: 26 11 2020
pubmed: 27 11 2020
medline: 27 11 2020
Statut: epublish

Résumé

WHO Group 1 pulmonary arterial hypertension is a progressive and potentially fatal disease. Individuals living at higher altitude are exposed to lower barometric pressure and hypobaric hypoxemia. This may result in pulmonary vasoconstriction and contribute to disease progression. We sought to examine the relationship between living at moderately high altitude and pulmonary arterial hypertension characteristics. Forty-two US centers participating in the Pulmonary Hypertension Association Registry enrolled patients who met the definition of WHO Group 1 pulmonary arterial hypertension. We utilized baseline data and patient questionnaire responses. Patients were divided into two groups: moderately high altitude residence (home ≥4000 ft) and low altitude residence (home <4000 ft) based on zip-code. Clinical characteristics, hemodynamic data, patient demographics, and patient reported quality of life metrics were compared. Controlling for potential confounders (age, sex at birth, body mass index, supplemental oxygen use, race, 100-day cigarette use, alcohol use, and pulmonary arterial hypertension medication use), subjects residing at moderately high altitude had a 6-min walk distance 32 m greater than those at low altitude, despite having a pulmonary vascular resistance that was 2.2 Wood units higher. Additionally, those residing at moderately high altitude had 3.7 times greater odds of using supplemental oxygen. Patients with pulmonary arterial hypertension who live at moderately high altitude have a higher pulmonary vascular resistance and are more likely to need supplemental oxygen. Despite these findings, moderately high altitude Pulmonary Hypertension Association Registry patients have better functional tolerance as measured by 6-min walk distance. It is possible that a "high-altitude phenotype" of pulmonary arterial hypertension may exist. These findings warrant further study.

Sections du résumé

BACKGROUND BACKGROUND
WHO Group 1 pulmonary arterial hypertension is a progressive and potentially fatal disease. Individuals living at higher altitude are exposed to lower barometric pressure and hypobaric hypoxemia. This may result in pulmonary vasoconstriction and contribute to disease progression. We sought to examine the relationship between living at moderately high altitude and pulmonary arterial hypertension characteristics.
METHODS METHODS
Forty-two US centers participating in the Pulmonary Hypertension Association Registry enrolled patients who met the definition of WHO Group 1 pulmonary arterial hypertension. We utilized baseline data and patient questionnaire responses. Patients were divided into two groups: moderately high altitude residence (home ≥4000 ft) and low altitude residence (home <4000 ft) based on zip-code. Clinical characteristics, hemodynamic data, patient demographics, and patient reported quality of life metrics were compared.
RESULTS RESULTS
Controlling for potential confounders (age, sex at birth, body mass index, supplemental oxygen use, race, 100-day cigarette use, alcohol use, and pulmonary arterial hypertension medication use), subjects residing at moderately high altitude had a 6-min walk distance 32 m greater than those at low altitude, despite having a pulmonary vascular resistance that was 2.2 Wood units higher. Additionally, those residing at moderately high altitude had 3.7 times greater odds of using supplemental oxygen.
CONCLUSION CONCLUSIONS
Patients with pulmonary arterial hypertension who live at moderately high altitude have a higher pulmonary vascular resistance and are more likely to need supplemental oxygen. Despite these findings, moderately high altitude Pulmonary Hypertension Association Registry patients have better functional tolerance as measured by 6-min walk distance. It is possible that a "high-altitude phenotype" of pulmonary arterial hypertension may exist. These findings warrant further study.

Identifiants

pubmed: 33240488
doi: 10.1177/2045894020964342
pii: 10.1177_2045894020964342
pmc: PMC7675880
doi:

Types de publication

Journal Article

Langues

eng

Pagination

2045894020964342

Commentaires et corrections

Type : ErratumIn

Informations de copyright

© The Author(s) 2020.

Déclaration de conflit d'intérêts

Conflict of interest: The author(s) declare that there is no conflict of interest.

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Auteurs

Shoaib Fakhri (S)

University of Colorado, Anschutz Medical Campus, Aurora, CO, USA.

Kelly Hannon (K)

University of Colorado, Anschutz Medical Campus, Aurora, CO, USA.

Kelly Moulden (K)

University of Colorado, Anschutz Medical Campus, Aurora, CO, USA.

Ryan Peterson (R)

University of Colorado, Anschutz Medical Campus, Aurora, CO, USA.

Peter Hountras (P)

University of Colorado, Anschutz Medical Campus, Aurora, CO, USA.

Todd Bull (T)

University of Colorado, Anschutz Medical Campus, Aurora, CO, USA.

James Maloney (J)

University of Colorado, Anschutz Medical Campus, Aurora, CO, USA.

Teresa De Marco (T)

University of California, San Francisco, CA, USA.

Dunbar Ivy (D)

University of Colorado, Anschutz Medical Campus, Aurora, CO, USA.
Children's Hospital of Colorado, Aurora, CO, USA.

Thenappan Thenappan (T)

University of Minnesota, Minneapolis, MN, USA.

Jeffrey S Sager (JS)

Cottage Health Pulmonary Hypertension Center, Santa Barbara, CA, USA.

John J Ryan (JJ)

University of Utah, Salt Lake City, UT, USA.

Sula Mazimba (S)

University of Virginia, Charlottesville, VA, USA.

Russel Hirsch (R)

Cincinnati Children's Hospital Medical Center, Cincinnati, OH, USA.

Murali Chakinala (M)

Washington University at Barnes-Jewish Hospital, St. Louis, MO, USA.

Oksana Shlobin (O)

Inova Fairfax Medical Campus, Falls Church, VA, USA.

Matthew Lammi (M)

Louisiana State University, Comprehensive Pulmonary Hypertension Center, New Orleans, LA, USA.

Dianne Zwicke (D)

Aurora St. Luke's Medical Center, Milwaukee, WI, USA.

Jeffrey Robinson (J)

The Oregon Clinic, Portland, OR, USA.

Raymond L Benza (RL)

Allegheny General Hospital, Pittsburg, PA, USA.

James Klinger (J)

Rhode Island Hospital, Providence, RI, USA.

Daniel Grinnan (D)

Virginia Commonwealth University, Richmond, VA, USA.

Stephen Mathai (S)

Johns Hopkins University, Baltimore, MD, USA.

David Badesch (D)

University of Colorado, Anschutz Medical Campus, Aurora, CO, USA.

Classifications MeSH