Human umbilical cord mesenchymal stromal cells as an adjunct therapy with therapeutic hypothermia in a piglet model of perinatal asphyxia.


Journal

Cytotherapy
ISSN: 1477-2566
Titre abrégé: Cytotherapy
Pays: England
ID NLM: 100895309

Informations de publication

Date de publication:
06 2021
Historique:
received: 08 08 2020
revised: 12 10 2020
accepted: 30 10 2020
pubmed: 3 12 2020
medline: 16 10 2021
entrez: 2 12 2020
Statut: ppublish

Résumé

With therapeutic hypothermia (HT) for neonatal encephalopathy, disability rates are reduced, but not all babies benefit. Pre-clinical rodent studies suggest mesenchymal stromal cells (MSCs) augment HT protection. The authors studied the efficacy of intravenous (IV) or intranasal (IN) human umbilical cord-derived MSCs (huMSCs) as adjunct therapy to HT in a piglet model. A total of 17 newborn piglets underwent transient cerebral hypoxia-ischemia (HI) and were then randomized to (i) HT at 33.5°C 1-13 h after HI (n = 7), (ii) HT+IV huMSCs (30 × 10 HI severity was similar between groups. Amplitude-integrated electroencephalogram (aEEG) recovery was more rapid for HT+IN huMSCs compared with HT from 25 h to 42 h and 49 h to 54 h (P ≤ 0.05). MRS phosphocreatine/inorganic phosphate was higher on day 2 in HT+IN huMSCs than HT (P = 0.035). Comparing HT+IN huMSCs with HT and HT+IV huMSCs, there were increased OLIG2 counts in hippocampus (P = 0.011 and 0.018, respectively), internal capsule (P = 0.013 and 0.037, respectively) and periventricular white matter (P = 0.15 for IN versus IV huMSCs). Reduced TUNEL-positive cells were seen in internal capsule with HT+IN huMSCs versus HT (P = 0.05). PKH-labeled huMSCs were detected in the brain 12 h after IN administration. After global HI, compared with HT alone, the authors saw beneficial effects of HT+IN huMSCs administered at 24 h and 48 h (30 × 10

Sections du résumé

BACKGROUND
With therapeutic hypothermia (HT) for neonatal encephalopathy, disability rates are reduced, but not all babies benefit. Pre-clinical rodent studies suggest mesenchymal stromal cells (MSCs) augment HT protection.
AIMS
The authors studied the efficacy of intravenous (IV) or intranasal (IN) human umbilical cord-derived MSCs (huMSCs) as adjunct therapy to HT in a piglet model.
METHODS
A total of 17 newborn piglets underwent transient cerebral hypoxia-ischemia (HI) and were then randomized to (i) HT at 33.5°C 1-13 h after HI (n = 7), (ii) HT+IV huMSCs (30 × 10
RESULTS
HI severity was similar between groups. Amplitude-integrated electroencephalogram (aEEG) recovery was more rapid for HT+IN huMSCs compared with HT from 25 h to 42 h and 49 h to 54 h (P ≤ 0.05). MRS phosphocreatine/inorganic phosphate was higher on day 2 in HT+IN huMSCs than HT (P = 0.035). Comparing HT+IN huMSCs with HT and HT+IV huMSCs, there were increased OLIG2 counts in hippocampus (P = 0.011 and 0.018, respectively), internal capsule (P = 0.013 and 0.037, respectively) and periventricular white matter (P = 0.15 for IN versus IV huMSCs). Reduced TUNEL-positive cells were seen in internal capsule with HT+IN huMSCs versus HT (P = 0.05). PKH-labeled huMSCs were detected in the brain 12 h after IN administration.
CONCLUSIONS
After global HI, compared with HT alone, the authors saw beneficial effects of HT+IN huMSCs administered at 24 h and 48 h (30 × 10

Identifiants

pubmed: 33262073
pii: S1465-3249(20)30930-0
doi: 10.1016/j.jcyt.2020.10.005
pmc: PMC8139415
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

521-535

Subventions

Organisme : Medical Research Council
ID : MR/M006743/1
Pays : United Kingdom
Organisme : Department of Health
Pays : United Kingdom

Informations de copyright

Copyright © 2020 International Society for Cell & Gene Therapy. Published by Elsevier Inc. All rights reserved.

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Auteurs

Nicola J Robertson (NJ)

Institute for Women's Health, University College London, London, UK. Electronic address: n.robertson@ucl.ac.uk.

Christopher Meehan (C)

Institute for Women's Health, University College London, London, UK.

Kathryn A Martinello (KA)

Institute for Women's Health, University College London, London, UK.

Adnan Avdic-Belltheus (A)

Institute for Women's Health, University College London, London, UK.

Tiziana Boggini (T)

Institute for Women's Health, University College London, London, UK.

Tatenda Mutshiya (T)

Institute for Women's Health, University College London, London, UK.

Ingran Lingam (I)

Institute for Women's Health, University College London, London, UK.

Qin Yang (Q)

Institute for Women's Health, University College London, London, UK.

Magdalena Sokolska (M)

University College London Hospitals NHS Foundation Trust, London, UK.

Xenia Charalambous (X)

Institute for Women's Health, University College London, London, UK.

Alan Bainbridge (A)

University College London Hospitals NHS Foundation Trust, London, UK.

Mariya Hristova (M)

Institute for Women's Health, University College London, London, UK.

Boris W Kramer (BW)

Department of Pediatrics, University of Maastricht, Maastricht, the Netherlands.

Xavier Golay (X)

Institute for Women's Health, University College London, London, UK.

Ben Weil (B)

Royal Free London NHS Foundation Trust, London, UK.

Mark W Lowdell (MW)

Institute for Women's Health, University College London, London, UK; Royal Free London NHS Foundation Trust, London, UK.

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