Linoleic Acid Status in Cell Membranes Inversely Relates to the Prevalence of Symptomatic Carotid Artery Disease.


Journal

Stroke
ISSN: 1524-4628
Titre abrégé: Stroke
Pays: United States
ID NLM: 0235266

Informations de publication

Date de publication:
01 2021
Historique:
pubmed: 5 12 2020
medline: 15 12 2021
entrez: 4 12 2020
Statut: ppublish

Résumé

The red blood cell fatty acid composition objectively reflects the long-term dietary intake of several fatty acids. In patients undergoing carotid endarterectomy, we explored whether red blood cell status of selected fatty acids related to symptomatic carotid artery disease. We included patients with symptomatic (n=22) and asymptomatic (n=23) carotid artery disease. We determined all-C18:1 trans, linoleic acid (LA, C18:2n6), alpha-linolenic acid (C18:3n3), and the omega-3 index (sum of eicosapentaenoic [C20:5n3] and docosahexaenoic [C22:6n3] acids) in both red blood cells and carotid plaque phospholipids by gas-chromatography. In a multivariate logistic regression analysis, we only observed a significant association for LA, whose red blood cell status was inversely related to symptomatic carotid artery disease (odds ratio, 0.116 [95% CI, 0.022-0.607], Cell membrane enrichment in LA, which reflects its intake, was inversely related to symptomatic carotid disease. This increases evidence supporting a favorable role of dietary LA in vascular health.

Sections du résumé

BACKGROUND AND PURPOSE
The red blood cell fatty acid composition objectively reflects the long-term dietary intake of several fatty acids. In patients undergoing carotid endarterectomy, we explored whether red blood cell status of selected fatty acids related to symptomatic carotid artery disease.
METHODS
We included patients with symptomatic (n=22) and asymptomatic (n=23) carotid artery disease. We determined all-C18:1 trans, linoleic acid (LA, C18:2n6), alpha-linolenic acid (C18:3n3), and the omega-3 index (sum of eicosapentaenoic [C20:5n3] and docosahexaenoic [C22:6n3] acids) in both red blood cells and carotid plaque phospholipids by gas-chromatography.
RESULTS
In a multivariate logistic regression analysis, we only observed a significant association for LA, whose red blood cell status was inversely related to symptomatic carotid artery disease (odds ratio, 0.116 [95% CI, 0.022-0.607],
CONCLUSIONS
Cell membrane enrichment in LA, which reflects its intake, was inversely related to symptomatic carotid disease. This increases evidence supporting a favorable role of dietary LA in vascular health.

Identifiants

pubmed: 33272124
doi: 10.1161/STROKEAHA.120.030477
doi:

Substances chimiques

Fatty Acids 0
Fatty Acids, Omega-3 0
Phospholipids 0
Linoleic Acid 9KJL21T0QJ

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

703-706

Auteurs

Iolanda Lázaro (I)

CIBER Pathophysiology of Obesity and Nutrition (CIBEROBN), Instituto de Salud Carlos III, Madrid, Spain (I.L., M.C., A.-J.A., E.O., A.S.-V.).
IMIM-Hospital del Mar Medical Research Institute, Barcelona, Spain (I.L., A.S.-V.).

Montserrat Cofán (M)

Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS), Barcelona, Spain (M.C., E.O., T.-M.F.-S., L.L.L., S.A., A.S.-V.).
CIBER Pathophysiology of Obesity and Nutrition (CIBEROBN), Instituto de Salud Carlos III, Madrid, Spain (I.L., M.C., A.-J.A., E.O., A.S.-V.).

Antonio J Amor (AJ)

Barcelonaβeta Brain Research Center, Pasqual Maragall Foundation, Barcelona, Spain (A.S.-V.).
Endocrinology and Nutrition Department (A.-J.A., E.O.), at Hospital Clínic de Barcelona, Barcelona, Spain.

Emilio Ortega (E)

Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS), Barcelona, Spain (M.C., E.O., T.-M.F.-S., L.L.L., S.A., A.S.-V.).
Barcelonaβeta Brain Research Center, Pasqual Maragall Foundation, Barcelona, Spain (A.S.-V.).
Endocrinology and Nutrition Department (A.-J.A., E.O.), at Hospital Clínic de Barcelona, Barcelona, Spain.

Tania-Marisa Freitas-Simoes (TM)

Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS), Barcelona, Spain (M.C., E.O., T.-M.F.-S., L.L.L., S.A., A.S.-V.).

Laura Llull (L)

Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS), Barcelona, Spain (M.C., E.O., T.-M.F.-S., L.L.L., S.A., A.S.-V.).
Neuroscience Department-Comprehensive Stroke Center (L.L.L., S.A.), at Hospital Clínic de Barcelona, Barcelona, Spain.

Sergio Amaro (S)

Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS), Barcelona, Spain (M.C., E.O., T.-M.F.-S., L.L.L., S.A., A.S.-V.).
Neuroscience Department-Comprehensive Stroke Center (L.L.L., S.A.), at Hospital Clínic de Barcelona, Barcelona, Spain.

Gaspar Mestres (G)

Division of Vascular Surgery-Cardiovascular Institute (G.M., X.Y., V.R.) at Hospital Clínic de Barcelona, Barcelona, Spain.

Xavier Yugueros (X)

Division of Vascular Surgery-Cardiovascular Institute (G.M., X.Y., V.R.) at Hospital Clínic de Barcelona, Barcelona, Spain.

William S Harris (WS)

Department of Internal Medicine, Sanford School of Medicine, University of South Dakota and OmegaQuant Analytics, LLC; Sioux Falls, South Dakota (W.S.H.).

Vicente Riambau (V)

Division of Vascular Surgery-Cardiovascular Institute (G.M., X.Y., V.R.) at Hospital Clínic de Barcelona, Barcelona, Spain.

Aleix Sala-Vila (A)

Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS), Barcelona, Spain (M.C., E.O., T.-M.F.-S., L.L.L., S.A., A.S.-V.).
CIBER Pathophysiology of Obesity and Nutrition (CIBEROBN), Instituto de Salud Carlos III, Madrid, Spain (I.L., M.C., A.-J.A., E.O., A.S.-V.).
Barcelonaβeta Brain Research Center, Pasqual Maragall Foundation, Barcelona, Spain (A.S.-V.).
IMIM-Hospital del Mar Medical Research Institute, Barcelona, Spain (I.L., A.S.-V.).

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