To study impact of treatment with Rosuvastatin versus Atorvastatin on 25 hydroxy Vitamin D concentrations among adult Indian men- a randomized control trial.


Journal

Indian journal of pharmacology
ISSN: 1998-3751
Titre abrégé: Indian J Pharmacol
Pays: India
ID NLM: 7902477

Informations de publication

Date de publication:
Historique:
entrez: 7 12 2020
pubmed: 8 12 2020
medline: 23 11 2021
Statut: ppublish

Résumé

Dyslipidemias are on the rise and are increasingly being treated with statins. As the metabolism of cholecalciferol and cholesterol are interrelated, reduction in cholesterol synthesis by statins is likely to affect Vitamin D status. (1) The aim is to study the effect of treatment with statins (Atorvastatin/Rosuvastatin) on 25-hydroxy-Vitamin-D (25OHD) among newly detected subjects with dyslipidemia for 6 months (2) To study the impact of 25OHD concentrations on the efficacy of statin treatment. This was a prospective, balanced randomized (1:1), open-label, parallel-group study, in apparently healthy Indian adult men (south Asian, 40-60 years). At baseline, serum lipids and 25OHD concentrations were measured. Based on the Adult Treatment Panel III guidelines, subjects were divided as per lipid concentrations into controls (who did not require statin treatment) and intervention (who required statin treatment) groups. Random allocation of subjects was done in two groups for receiving intervention for 6 months: Atorvastatin group (n = 52, received Atorvastatin) or Rosuvastatin group (n = 52, received Rosuvastatin). Lipids and 25OHD concentrations were measured at the end line. Atorvastatin group presented significant reduction (P < 0.05) in 25OHD, total cholesterol (TC) and low-density-lipoprotein-cholesterol (LDL-C) concentrations at the end line. In the Rosuvastatin group, significant drop in TC, LDL-C and high-density lipoprotein cholesterol (concentrations (P < 0.05) was observed, while 25OHD concentrations showed no significant change. Mean 25OHD concentrations were significantly correlated with a reduction in LDL-C concentrations in Atorvastatin group. Treatment with Atorvastatin resulted in a reduction in 25OHD concentrations; further, its efficacy in reducing LDL-C concentrations was related to the 25OHD concentrations.

Sections du résumé

BACKGROUND BACKGROUND
Dyslipidemias are on the rise and are increasingly being treated with statins. As the metabolism of cholecalciferol and cholesterol are interrelated, reduction in cholesterol synthesis by statins is likely to affect Vitamin D status.
OBJECTIVES OBJECTIVE
(1) The aim is to study the effect of treatment with statins (Atorvastatin/Rosuvastatin) on 25-hydroxy-Vitamin-D (25OHD) among newly detected subjects with dyslipidemia for 6 months (2) To study the impact of 25OHD concentrations on the efficacy of statin treatment.
MATERIALS AND METHODS METHODS
This was a prospective, balanced randomized (1:1), open-label, parallel-group study, in apparently healthy Indian adult men (south Asian, 40-60 years). At baseline, serum lipids and 25OHD concentrations were measured. Based on the Adult Treatment Panel III guidelines, subjects were divided as per lipid concentrations into controls (who did not require statin treatment) and intervention (who required statin treatment) groups. Random allocation of subjects was done in two groups for receiving intervention for 6 months: Atorvastatin group (n = 52, received Atorvastatin) or Rosuvastatin group (n = 52, received Rosuvastatin). Lipids and 25OHD concentrations were measured at the end line.
RESULTS RESULTS
Atorvastatin group presented significant reduction (P < 0.05) in 25OHD, total cholesterol (TC) and low-density-lipoprotein-cholesterol (LDL-C) concentrations at the end line. In the Rosuvastatin group, significant drop in TC, LDL-C and high-density lipoprotein cholesterol (concentrations (P < 0.05) was observed, while 25OHD concentrations showed no significant change. Mean 25OHD concentrations were significantly correlated with a reduction in LDL-C concentrations in Atorvastatin group.
CONCLUSIONS CONCLUSIONS
Treatment with Atorvastatin resulted in a reduction in 25OHD concentrations; further, its efficacy in reducing LDL-C concentrations was related to the 25OHD concentrations.

Identifiants

pubmed: 33283767
pii: Indian J Pharmacol_2020_52_5_365_302509
doi: 10.4103/ijp.IJP_93_18
pmc: PMC8025761
doi:

Substances chimiques

Cholesterol, HDL 0
Cholesterol, LDL 0
Hydroxymethylglutaryl-CoA Reductase Inhibitors 0
Vitamin D 1406-16-2
Rosuvastatin Calcium 83MVU38M7Q
Cholesterol 97C5T2UQ7J
Atorvastatin A0JWA85V8F
25-hydroxyvitamin D A288AR3C9H

Types de publication

Comparative Study Journal Article Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

365-371

Déclaration de conflit d'intérêts

None

Références

Hepatology. 2014 Aug;60(2):679-86
pubmed: 24700436
J Am Coll Cardiol. 2004 Aug 4;44(3):720-32
pubmed: 15358046
Lipids Health Dis. 2010 May 21;9:52
pubmed: 20487572
J Cardiovasc Pharmacol Ther. 2013 May;18(3):229-33
pubmed: 23288870
Health Aff (Millwood). 2014 Feb;33(2):273-82
pubmed: 24493771
Ann Intern Med. 2010 Mar 2;152(5):315-23
pubmed: 20194238
Pharmacogenomics J. 2006 Nov-Dec;6(6):360-74
pubmed: 16550210
J Osteoporos. 2014;2014:468397
pubmed: 25197610
Clin Chem. 2010 Nov;56(11):1696-700
pubmed: 20817794
Indian J Endocrinol Metab. 2015 Sep-Oct;19(5):546-53
pubmed: 26425462
Eur Rev Med Pharmacol Sci. 2008 Mar-Apr;12(2):83-8
pubmed: 18575157
J Clin Densitom. 2000 Spring;3(1):35-41
pubmed: 10745300
Nutrients. 2014 Feb 21;6(2):729-75
pubmed: 24566435
Atherosclerosis. 2011 Aug;217(2):308-21
pubmed: 21762916
Mol Nutr Food Res. 2011 May;55(5):691-702
pubmed: 21280209
J Lipid Res. 2009 Apr;50(4):730-9
pubmed: 19043140
Cardiovasc Drugs Ther. 2009 Aug;23(4):295-9
pubmed: 19543962
Acta Derm Venereol. 2011 Mar;91(2):115-24
pubmed: 21384086
Clin Pharmacokinet. 2003;42(13):1141-60
pubmed: 14531725
QJM. 2012 May;105(5):487-91
pubmed: 22323613
Ann Intern Med. 2010 Mar 2;152(5):307-14
pubmed: 20194237
Am J Physiol Gastrointest Liver Physiol. 2008 Apr;294(4):G839-43
pubmed: 18276831
J Clin Endocrinol Metab. 2005 Feb;90(2):1210-9
pubmed: 15546903
Cardiovasc Ther. 2011 Apr;29(2):146-52
pubmed: 20370794
Open Cardiovasc Med J. 2014 Jul 11;8:55-60
pubmed: 25110531
J Assoc Physicians India. 2009 Jan;57:40-8
pubmed: 19753759
PLoS One. 2014 Feb 19;9(2):e88877
pubmed: 24586424
Indian J Med Res. 2010 Nov;132:561-6
pubmed: 21150008
Clin Ther. 2003 Oct;25(10):2553-63
pubmed: 14667956

Auteurs

Vivek G Patwardhan (VG)

Department of Pediatric Growth and Endocrine Unit, Hirabai Cowasji Jehangir Medical Research Institute, Jehangir Hospital, Pune, Maharashtra, India.

Zulf M Mughal (ZM)

Department of Paediatric Endocrinology, Royal Manchester Children's Hospital, Manchester, UK.

Raja Padidela (R)

Department of Paediatric Endocrinology, Royal Manchester Children's Hospital, Manchester, UK.

Shashi A Chiplonkar (SA)

Department of Pediatric Growth and Endocrine Unit, Hirabai Cowasji Jehangir Medical Research Institute, Jehangir Hospital, Pune, Maharashtra, India.

Vaman V Khadilkar (VV)

Department of Pediatric Growth and Endocrine Unit, Hirabai Cowasji Jehangir Medical Research Institute, Jehangir Hospital, Pune, Maharashtra, India.

Anuradha V Khadilkar (AV)

Department of Pediatric Growth and Endocrine Unit, Hirabai Cowasji Jehangir Medical Research Institute, Jehangir Hospital, Pune, Maharashtra, India.

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Classifications MeSH