Activity and Adverse Events of Actinium-225-PSMA-617 in Advanced Metastatic Castration-resistant Prostate Cancer After Failure of Lutetium-177-PSMA.


Journal

European urology
ISSN: 1873-7560
Titre abrégé: Eur Urol
Pays: Switzerland
ID NLM: 7512719

Informations de publication

Date de publication:
03 2021
Historique:
received: 01 04 2020
accepted: 10 11 2020
pubmed: 10 12 2020
medline: 10 2 2022
entrez: 9 12 2020
Statut: ppublish

Résumé

Beta-emitting Lu-177-labeled prostate-specific membrane antigen (PSMA) radioligand therapy (RLT) is a new option for metastatic castration-resistant prostate cancer (mCRPC), but its antitumor effect can decrease over time. To report the safety and activity of alpha-emitting Ac-225-PSMA-617 RLT in mCRPC that has progressed after Lu-177-PSMA. Twenty-six patients were treated under a compassionate use protocol. The eligibility criteria included previous treatment with abiraterone or enzalutamide, previous taxane-based chemotherapy, progression after Lu-177-PSMA, and positive PSMA-ligand uptake. The median number of previous mCRPC regimens was 6. Ac-225-PSMA-617 was given every 8 wk until progression/intolerable side effects. Prostate-specific antigen (PSA) decline, PSA progression-free survival (PSA-PFS), clinical progression-free survival (cPFS), overall survival (OS), and toxicity were measured. Sixty-one cycles of Ac-225-PSMA-617 (median number of cycles 2; median activity 9 MBq) were administered. A PSA decline of ≥50% was achieved in 17/26 patients. The median PSA-PFS, cPFS, and OS periods were 3.5 (95% confidence interval [CI] 1.8-11.2), 4.1 (95% CI 3-14.8), and 7.7 (95% CI 4.5-12.1) mo, respectively. Liver metastases were associated with shorter PSA-PFS (median 1.9 vs 4.0 mo; p = 0.02), cPFS (median 1.8 vs 5.2 mo; p = 0.001), and OS (median 4.3 vs 10.4 mo; p = 0.01). Hematological grade 3/4 toxicities were anemia (35%), leucopenia (27%), and thrombocytopenia (19%). All patients experienced grade 1/2 xerostomia. Two and six patients stopped due to hematological toxicity and xerostomia, respectively. A limitation is the retrospective design. Ac-225-PSMA-617 showed measurable antitumor effect after Lu-177-PSMA failure in late-stage mCRPC. Grade 3/4 hematological side effects were observed in up to one-third of patients, and xerostomia led to treatment halt in a relevant number of patients. Ac-225-labeled prostate-specific membrane antigen (PSMA)-617 therapy showed substantial antitumor effect in late metastatic castration-resistant prostate cancer after Lu-177-PSMA failure. However, dry mouth is a common side effect that caused about a quarter of patients to stop therapy.

Sections du résumé

BACKGROUND
Beta-emitting Lu-177-labeled prostate-specific membrane antigen (PSMA) radioligand therapy (RLT) is a new option for metastatic castration-resistant prostate cancer (mCRPC), but its antitumor effect can decrease over time.
OBJECTIVE
To report the safety and activity of alpha-emitting Ac-225-PSMA-617 RLT in mCRPC that has progressed after Lu-177-PSMA.
DESIGN, SETTING, AND PARTICIPANTS
Twenty-six patients were treated under a compassionate use protocol. The eligibility criteria included previous treatment with abiraterone or enzalutamide, previous taxane-based chemotherapy, progression after Lu-177-PSMA, and positive PSMA-ligand uptake. The median number of previous mCRPC regimens was 6. Ac-225-PSMA-617 was given every 8 wk until progression/intolerable side effects.
OUTCOME MEASUREMENTS AND STATISTICAL ANALYSIS
Prostate-specific antigen (PSA) decline, PSA progression-free survival (PSA-PFS), clinical progression-free survival (cPFS), overall survival (OS), and toxicity were measured.
RESULTS AND LIMITATIONS
Sixty-one cycles of Ac-225-PSMA-617 (median number of cycles 2; median activity 9 MBq) were administered. A PSA decline of ≥50% was achieved in 17/26 patients. The median PSA-PFS, cPFS, and OS periods were 3.5 (95% confidence interval [CI] 1.8-11.2), 4.1 (95% CI 3-14.8), and 7.7 (95% CI 4.5-12.1) mo, respectively. Liver metastases were associated with shorter PSA-PFS (median 1.9 vs 4.0 mo; p = 0.02), cPFS (median 1.8 vs 5.2 mo; p = 0.001), and OS (median 4.3 vs 10.4 mo; p = 0.01). Hematological grade 3/4 toxicities were anemia (35%), leucopenia (27%), and thrombocytopenia (19%). All patients experienced grade 1/2 xerostomia. Two and six patients stopped due to hematological toxicity and xerostomia, respectively. A limitation is the retrospective design.
CONCLUSIONS
Ac-225-PSMA-617 showed measurable antitumor effect after Lu-177-PSMA failure in late-stage mCRPC. Grade 3/4 hematological side effects were observed in up to one-third of patients, and xerostomia led to treatment halt in a relevant number of patients.
PATIENT SUMMARY
Ac-225-labeled prostate-specific membrane antigen (PSMA)-617 therapy showed substantial antitumor effect in late metastatic castration-resistant prostate cancer after Lu-177-PSMA failure. However, dry mouth is a common side effect that caused about a quarter of patients to stop therapy.

Identifiants

pubmed: 33293081
pii: S0302-2838(20)30877-0
doi: 10.1016/j.eururo.2020.11.013
pii:
doi:

Substances chimiques

Actinium-225 0
Dipeptides 0
Heterocyclic Compounds, 1-Ring 0
PSMA-617 0
Radioisotopes 0
Radiopharmaceuticals 0
Lutetium 5H0DOZ21UJ
Lutetium-177 BRH40Y9V1Q
Prostate-Specific Antigen EC 3.4.21.77
Actinium NIK1K0956U

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

343-350

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2020 European Association of Urology. Published by Elsevier B.V. All rights reserved.

Auteurs

Benedikt Feuerecker (B)

Department of Nuclear Medicine, School of Medicine, Technical University of Munich, Munich, Germany; German Cancer Consortium (DKTK), partnersite Munich, German Cancer Research Center (DKFZ), Heidelberg, Germany. Electronic address: benedikt.feuerecker@tum.de.

Robert Tauber (R)

Department of Urology, School of Medicine, Technical University of Munich, Munich, Germany.

Karina Knorr (K)

Department of Nuclear Medicine, School of Medicine, Technical University of Munich, Munich, Germany.

Matthias Heck (M)

Department of Urology, School of Medicine, Technical University of Munich, Munich, Germany.

Ali Beheshti (A)

Department of Nuclear Medicine, School of Medicine, Technical University of Munich, Munich, Germany.

Christof Seidl (C)

Department of Nuclear Medicine, School of Medicine, Technical University of Munich, Munich, Germany.

Frank Bruchertseifer (F)

European Commission, Joint Research Centre, Directorate for Nuclear Safety and Security, Karlsruhe, Germany.

Anja Pickhard (A)

Department of Otolaryngology Head and Neck Surgery, School of Medicine, Technical University of Munich, Munich, Germany.

Andrei Gafita (A)

Department of Nuclear Medicine, School of Medicine, Technical University of Munich, Munich, Germany.

Clemens Kratochwil (C)

Department of Nuclear Medicine, University Hospital Heidelberg, Heidelberg, Germany.

Margitta Retz (M)

Department of Urology, School of Medicine, Technical University of Munich, Munich, Germany.

Jürgen E Gschwend (JE)

Department of Urology, School of Medicine, Technical University of Munich, Munich, Germany.

Wolfgang A Weber (WA)

Department of Nuclear Medicine, School of Medicine, Technical University of Munich, Munich, Germany; German Cancer Consortium (DKTK), partnersite Munich, German Cancer Research Center (DKFZ), Heidelberg, Germany.

Calogero D'Alessandria (C)

Department of Nuclear Medicine, School of Medicine, Technical University of Munich, Munich, Germany.

Alfred Morgenstern (A)

European Commission, Joint Research Centre, Directorate for Nuclear Safety and Security, Karlsruhe, Germany.

Matthias Eiber (M)

Department of Nuclear Medicine, School of Medicine, Technical University of Munich, Munich, Germany; German Cancer Consortium (DKTK), partnersite Munich, German Cancer Research Center (DKFZ), Heidelberg, Germany.

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Classifications MeSH