PPARs and Myocardial Infarction.
angiogenesis
cardiomyocytes
cardiovascular disease
endothelial cells
myocardial infarction
peroxisome proliferator-activated receptor
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
11 Dec 2020
11 Dec 2020
Historique:
received:
01
12
2020
accepted:
10
12
2020
entrez:
16
12
2020
pubmed:
17
12
2020
medline:
16
3
2021
Statut:
epublish
Résumé
Peroxisome proliferator-activated receptors (PPARs) belong to the nuclear hormone receptor family. They are ligand-activated transcription factors and exist in three different isoforms, PPARα (NR1C1), PPARβ/δ (NR1C2), and PPARγ (NR1C3). PPARs regulate a variety of functions, including glucose and lipid homeostasis, inflammation, and development. They exhibit tissue and cell type-specific expression patterns and functions. Besides the established notion of the therapeutic potential of PPAR agonists for the treatment of glucose and lipid disorders, more recent data propose specific PPAR ligands as potential therapies for cardiovascular diseases. In this review, we focus on the knowledge of PPAR function in myocardial infarction, a severe pathological condition for which therapeutic use of PPAR modulation has been suggested.
Identifiants
pubmed: 33322384
pii: ijms21249436
doi: 10.3390/ijms21249436
pmc: PMC7764190
pii:
doi:
Substances chimiques
Peroxisome Proliferator-Activated Receptors
0
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
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